miR-19b-3p promotes human pancreatic cancer Capan-2 cells proliferation by targeting phosphatase and tension homolog.

Pancreatic cancer miR-19b-3p phosphatase and tension homolog (PTEN) proliferation

Journal

Annals of translational medicine
ISSN: 2305-5839
Titre abrégé: Ann Transl Med
Pays: China
ID NLM: 101617978

Informations de publication

Date de publication:
Jun 2019
Historique:
entrez: 19 7 2019
pubmed: 19 7 2019
medline: 19 7 2019
Statut: ppublish

Résumé

Pancreatic cancer is a common cancer with a poor prognosis and an increasing morbidity. miR-19b-3p has been implicated in some cancers, however, its role in pancreatic cancer is unclear. Human pancreatic cancer cell line Capan-2 cells were transfected with miR-19b-3p mimic and inhibitor. Cell proliferation was measured by 5-Ethynyl-2'-deoxyuridine (EdU) staining assays. Cell cycle of Capan-2 cells was examined by flow cytometry. The expression of phosphatase and tension homolog (PTEN) was determined by real-time quantitative polymerase chain reaction (PCR) and western blotting analysis. Functional rescue experiments were performed through PTEN overexpression and miR-19b-3p mimic by using EdU staining assays. miR-19b-3p mimic significantly increased miR-19b-3p while miR-19b-3p inhibitor decreased that. EdU staining showed that miR-19b-3p overexpression promoted Capan-2 cells proliferation while miR-19b-3p inhibition decreased that. Flow cytometry analysis of cell cycle indicated that miR-19b-3p overexpression increased the percentage of Capan-2 cells in S phase while miR-19b-3p inhibition decreased that. Our study demonstrates that miR-19b-3p promotes Capan-2 cells proliferation by targeting

Sections du résumé

BACKGROUND BACKGROUND
Pancreatic cancer is a common cancer with a poor prognosis and an increasing morbidity. miR-19b-3p has been implicated in some cancers, however, its role in pancreatic cancer is unclear.
METHODS METHODS
Human pancreatic cancer cell line Capan-2 cells were transfected with miR-19b-3p mimic and inhibitor. Cell proliferation was measured by 5-Ethynyl-2'-deoxyuridine (EdU) staining assays. Cell cycle of Capan-2 cells was examined by flow cytometry. The expression of phosphatase and tension homolog (PTEN) was determined by real-time quantitative polymerase chain reaction (PCR) and western blotting analysis. Functional rescue experiments were performed through PTEN overexpression and miR-19b-3p mimic by using EdU staining assays.
RESULTS RESULTS
miR-19b-3p mimic significantly increased miR-19b-3p while miR-19b-3p inhibitor decreased that. EdU staining showed that miR-19b-3p overexpression promoted Capan-2 cells proliferation while miR-19b-3p inhibition decreased that. Flow cytometry analysis of cell cycle indicated that miR-19b-3p overexpression increased the percentage of Capan-2 cells in S phase while miR-19b-3p inhibition decreased that.
CONCLUSIONS CONCLUSIONS
Our study demonstrates that miR-19b-3p promotes Capan-2 cells proliferation by targeting

Identifiants

pubmed: 31317006
doi: 10.21037/atm.2019.04.61
pii: atm-07-11-236
pmc: PMC6603353
doi:

Types de publication

Journal Article

Langues

eng

Pagination

236

Déclaration de conflit d'intérêts

Conflicts of Interest: The authors have no conflicts of interest to declare.

Références

Mol Cell Biochem. 2009 Nov;331(1-2):161-71
pubmed: 19437103
Nat Genet. 2011 Jun 05;43(7):673-8
pubmed: 21642990
FEBS Lett. 2011 Aug 19;585(16):2592-8
pubmed: 21781967
Blood. 2011 Nov 17;118(20):5487-97
pubmed: 21972292
Cancer Discov. 2011 Jul;1(2):158-69
pubmed: 21984975
Med Oncol. 2013 Dec;30(4):700
pubmed: 24026657
Adv Biol Regul. 2014 Sep;56:66-80
pubmed: 25088603
Lab Invest. 2015 Sep;95(9):1056-70
pubmed: 26098000
J Cancer. 2015 Oct 16;6(12):1230-5
pubmed: 26535064
Cell Rep. 2016 Feb 9;14(5):979-990
pubmed: 26804906
Lancet. 2016 Jul 2;388(10039):73-85
pubmed: 26830752
J Cell Mol Med. 2016 Jun;20(6):1191-7
pubmed: 27061862
Cancer Res. 2016 Jul 15;76(14):4149-59
pubmed: 27216198
Front Genet. 2016 Aug 31;7:143
pubmed: 27630665
Acta Biochim Biophys Sin (Shanghai). 2016 Dec;48(12):1085-1093
pubmed: 27797718
J Exp Clin Cancer Res. 2016 Dec 5;35(1):188
pubmed: 27919278
Eur J Gastroenterol Hepatol. 2017 Mar;29(3):e13-e18
pubmed: 28009716
JAMA. 2017 Jan 24;317(4):388-406
pubmed: 28118455
Am J Cancer Res. 2017 Jan 01;7(1):139-147
pubmed: 28123855
Cell Mol Life Sci. 2017 Aug;74(15):2783-2794
pubmed: 28289760
Nat Commun. 2017 May 12;8:15223
pubmed: 28497778
Cancer Epidemiol Biomarkers Prev. 2017 Aug;26(8):1225-1232
pubmed: 28619830
Cell. 2018 May 17;173(5):1191-1203.e12
pubmed: 29706542
J Sport Health Sci. 2017 Mar;6(1):125-126
pubmed: 30356527
J Sport Health Sci. 2017 Sep;6(3):292-294
pubmed: 30356613
J Sport Health Sci. 2018 Oct;7(4):433-441
pubmed: 30450252
Nat Commun. 2019 Jan 18;10(1):307
pubmed: 30659180
J Cell Physiol. 2019 Feb 14;:null
pubmed: 30767206

Auteurs

Meiyi Song (M)

Division of Gastroenterology and Hepatology, Digestive Disease Institute, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, China.

Mengxue Sun (M)

Division of Gastroenterology and Hepatology, Digestive Disease Institute, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, China.

Lu Xia (L)

Division of Gastroenterology and Hepatology, Digestive Disease Institute, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, China.

Wei Chen (W)

Emergency Department, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, China.

Changqing Yang (C)

Division of Gastroenterology and Hepatology, Digestive Disease Institute, Shanghai Tongji Hospital, Tongji University School of Medicine, Shanghai 200065, China.

Classifications MeSH