Capecitabine in advanced hepatocellular carcinoma: A multicenter experience.


Journal

Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
ISSN: 1878-3562
Titre abrégé: Dig Liver Dis
Pays: Netherlands
ID NLM: 100958385

Informations de publication

Date de publication:
12 2019
Historique:
received: 13 01 2019
revised: 04 04 2019
accepted: 19 06 2019
pubmed: 20 7 2019
medline: 22 5 2020
entrez: 20 7 2019
Statut: ppublish

Résumé

Recent data suggest a potential activity and a good tolerability of capecitabine in advanced hepatocellular carcinoma (HCC). To evaluate capecitabine activity and safety in a wide cohort of advanced HCC patients. Retrospective analysis of 143 capecitabine-treated patients (January 2010 to December 2017) in three centers of the Veneto Oncology Network. Capecitabine was administered in second and third line, but also in first line instead of sorafenib in Child-Pugh B patients (70%), compromised clinical conditions (14%) or contraindications to antiangiogenetics (16%). Median overall survival (OS) and time to progression (TTP) were 6.9 and 2.8 months, respectively. There were no differences in OS and TTP between the 32 patients treated with non-metronomic scheme (2000 mg/day for 14 days) and the 111 patients treated with metronomic scheme (1000 mg/day) after correction for prognostic factors at baseline with a propensity score analysis. Capecitabine was more active in patients intolerant to sorafenib than in those progressing during treatment (p = 0.024). At least one adverse event (mainly hematological) was experienced by 73% of patients but discontinuation was necessary only in 11 (8%). Capecitabine can be considered an active and safe option in advanced HCC, especially for patients unfit for other treatments.

Sections du résumé

BACKGROUND
Recent data suggest a potential activity and a good tolerability of capecitabine in advanced hepatocellular carcinoma (HCC).
AIMS
To evaluate capecitabine activity and safety in a wide cohort of advanced HCC patients.
METHODS
Retrospective analysis of 143 capecitabine-treated patients (January 2010 to December 2017) in three centers of the Veneto Oncology Network.
RESULTS
Capecitabine was administered in second and third line, but also in first line instead of sorafenib in Child-Pugh B patients (70%), compromised clinical conditions (14%) or contraindications to antiangiogenetics (16%). Median overall survival (OS) and time to progression (TTP) were 6.9 and 2.8 months, respectively. There were no differences in OS and TTP between the 32 patients treated with non-metronomic scheme (2000 mg/day for 14 days) and the 111 patients treated with metronomic scheme (1000 mg/day) after correction for prognostic factors at baseline with a propensity score analysis. Capecitabine was more active in patients intolerant to sorafenib than in those progressing during treatment (p = 0.024). At least one adverse event (mainly hematological) was experienced by 73% of patients but discontinuation was necessary only in 11 (8%).
CONCLUSIONS
Capecitabine can be considered an active and safe option in advanced HCC, especially for patients unfit for other treatments.

Identifiants

pubmed: 31320302
pii: S1590-8658(19)30672-3
doi: 10.1016/j.dld.2019.06.015
pii:
doi:

Substances chimiques

Antimetabolites, Antineoplastic 0
Capecitabine 6804DJ8Z9U

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

1713-1719

Informations de copyright

Copyright © 2019. Published by Elsevier Ltd.

Auteurs

Filippo Pelizzaro (F)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy.

Ambra Sammarco (A)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy.

Vincenzo Dadduzio (V)

Department of Clinical and Experimental Oncology, Unit of Medical Oncology 1, Istituto Oncologico Veneto - IRCCS, Via Gattamelata 64, 35128 Padova, PD, Italy.

Davide Pastorelli (D)

Department of Oncology, S. Maria del Prato Hospital, Via Bagnols sur Ceze 3, 32032 Feltre, BL, Italy.

Petros Giovanis (P)

Department of Oncology, S. Maria del Prato Hospital, Via Bagnols sur Ceze 3, 32032 Feltre, BL, Italy.

Caterina Soldà (C)

Medical Oncology Azienda ULSS 3 Serenissima, Ospedale dell'Angelo, Via Paccagnella 11, 30174 Mestre, VE, Italy.

Mario Domenico Rizzato (MD)

Department of Clinical and Experimental Oncology, Unit of Medical Oncology 1, Istituto Oncologico Veneto - IRCCS, Via Gattamelata 64, 35128 Padova, PD, Italy.

Giuseppe Lombardi (G)

Department of Clinical and Experimental Oncology, Unit of Medical Oncology 1, Istituto Oncologico Veneto - IRCCS, Via Gattamelata 64, 35128 Padova, PD, Italy.

Sara Lonardi (S)

Department of Clinical and Experimental Oncology, Unit of Medical Oncology 1, Istituto Oncologico Veneto - IRCCS, Via Gattamelata 64, 35128 Padova, PD, Italy.

Giulia Peserico (G)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy.

Angela Imondi (A)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy.

Anna Sartori (A)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy.

Gemma Maddalo (G)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy.

Fabio Farinati (F)

Department of Surgical, Oncological and Gastroenterological Sciences, Unit of Gastroenterology, University of Padova, Via Giustiniani 2, 35128 Padova, PD, Italy. Electronic address: fabio.farinati@unipd.it.

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Classifications MeSH