Humanized anti-CD271 monoclonal antibody exerts an anti-tumor effect by depleting cancer stem cells.


Journal

Cancer letters
ISSN: 1872-7980
Titre abrégé: Cancer Lett
Pays: Ireland
ID NLM: 7600053

Informations de publication

Date de publication:
01 Oct 2019
Historique:
received: 09 05 2019
revised: 21 06 2019
accepted: 12 07 2019
pubmed: 22 7 2019
medline: 28 5 2020
entrez: 21 7 2019
Statut: ppublish

Résumé

CD271, known as a neurotrophin receptor, is expressed in various cancers such as hypopharyngeal cancer (HPC) and melanoma. We recently reported that CD271 is a cancer-stem-cell biomarker of HPC, and that its expression is essential for cancer-cell proliferation and is correlated with a poor prognosis in this disease. Here, to develop a therapeutic antibody to CD271, we established a humanized anti-CD271 monoclonal antibody (hCD271 mA b). hCD271 mA b bound to the cysteine-rich domain 1 (CRD1) of human CD271 with high affinity (K

Identifiants

pubmed: 31325530
pii: S0304-3835(19)30407-0
doi: 10.1016/j.canlet.2019.07.011
pii:
doi:

Substances chimiques

Antibodies, Monoclonal, Humanized 0
NGFR protein, human 0
Nerve Tissue Proteins 0
Receptors, Nerve Growth Factor 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

144-152

Informations de copyright

Copyright © 2019 Elsevier B.V. All rights reserved.

Auteurs

Shinkichi Morita (S)

Division of Cancer Stem Cell, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan; Department of Head and Neck Surgery, Miyagi Cancer Center, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Mai Mochizuki (M)

Division of Cancer Stem Cell, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Kouichi Wada (K)

Research Unit/Innovative Medical Science, Sohyaku, Innovative Research Division, Mitsubishi Tanabe Pharma Corporation, 1000, Kamoshida-cho, Aoba-ku, Yokohama, Kanagawa, Japan.

Rie Shibuya (R)

Division of Cancer Stem Cell, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Mao Nakamura (M)

Division of Molecular and Cellular Oncology, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Kazunori Yamaguchi (K)

Division of Molecular and Cellular Oncology, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Tomoko Yamazaki (T)

Department of Head and Neck Medical Oncology, Miyagi Cancer Center, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Takayuki Imai (T)

Department of Head and Neck Surgery, Miyagi Cancer Center, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Yukinori Asada (Y)

Department of Head and Neck Surgery, Miyagi Cancer Center, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Kazuto Matsuura (K)

Department of Head and Neck Surgery, Miyagi Cancer Center, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Kazuo Sugamura (K)

Division of Molecular and Cellular Oncology, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan.

Yukio Katori (Y)

Department of Otolaryngology-Head and Neck Surgery, Tohoku University Graduate School of Medicine, 2-1 Seiryo-machi, Aoba-ku, Sendai, Miyagi, Japan.

Kennichi Satoh (K)

Division of Cancer Stem Cell, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan; Division of Gastroenterology and Hepatology, Tohoku Medical and Pharmaceutical University, 4-4-1 Komatsushima, Aobaku, Sendai, Miyagi, Japan.

Keiichi Tamai (K)

Division of Cancer Stem Cell, Miyagi Cancer Center Research Institute, 47-1, Medeshima-Shiode, Natori, Miyagi, Japan. Electronic address: tamaikeiichi@med.tohoku.ac.jp.

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Classifications MeSH