The role of Müller cells in tractional macular disorders: an optical coherence tomography study and physical model of mechanical force transmission.


Journal

The British journal of ophthalmology
ISSN: 1468-2079
Titre abrégé: Br J Ophthalmol
Pays: England
ID NLM: 0421041

Informations de publication

Date de publication:
04 2020
Historique:
received: 12 03 2019
revised: 12 06 2019
accepted: 06 07 2019
pubmed: 22 7 2019
medline: 31 10 2020
entrez: 22 7 2019
Statut: ppublish

Résumé

To explore the role of foveal and parafoveal Müller cells in the morphology and pathophysiology of tractional macular disorders with a mathematical model of mechanical force transmission. In this retrospective observational study, spectral-domain optical coherence tomography images of tractional lamellar macular holes and patients with myopic foveoschisis were reviewed and analysed with a mathematical model of force transmission. Parafoveal In tractional lamellar macular holes, there was a significant reduction of the angle θ towards the foveal centre (p<0.001). By contrast, there were no significant differences in θ in myopic foveoschisis (p=0.570). R2 segments were more vertical in myopic foveoschisis. There was a significant association between lower θ angles at 200 µm temporal and nasal to the fovea and lower BCVA (p<0.001 and p=0.005, respectively). The stiffness of parafoveal Müller cells was predicted to be function of the angle θ , and it grew very rapidly as the θ decreased. Parafoveal Müller cells in the Henle fibre layer may guarantee structural stability of the parafovea by increasing retinal compliance and resistance to mechanical stress. Small values of the angle θ were related to worse BCVA possibly due to damage to Müller cell processes and photoreceptor's axons.

Sections du résumé

BACKGROUND
To explore the role of foveal and parafoveal Müller cells in the morphology and pathophysiology of tractional macular disorders with a mathematical model of mechanical force transmission.
METHODS
In this retrospective observational study, spectral-domain optical coherence tomography images of tractional lamellar macular holes and patients with myopic foveoschisis were reviewed and analysed with a mathematical model of force transmission. Parafoveal
RESULTS
In tractional lamellar macular holes, there was a significant reduction of the angle θ towards the foveal centre (p<0.001). By contrast, there were no significant differences in θ in myopic foveoschisis (p=0.570). R2 segments were more vertical in myopic foveoschisis. There was a significant association between lower θ angles at 200 µm temporal and nasal to the fovea and lower BCVA (p<0.001 and p=0.005, respectively). The stiffness of parafoveal Müller cells was predicted to be function of the angle θ , and it grew very rapidly as the θ decreased.
CONCLUSION
Parafoveal Müller cells in the Henle fibre layer may guarantee structural stability of the parafovea by increasing retinal compliance and resistance to mechanical stress. Small values of the angle θ were related to worse BCVA possibly due to damage to Müller cell processes and photoreceptor's axons.

Identifiants

pubmed: 31326893
pii: bjophthalmol-2019-314245
doi: 10.1136/bjophthalmol-2019-314245
doi:

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

466-472

Informations de copyright

© Author(s) (or their employer(s)) 2020. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

Auteurs

Andrea Govetto (A)

Department of Ophthalmology, Fatebenefratelli-Oftalmico Hospital, ASST-Fatebenefratelli-Sacco, Milan, Italy a.govetto@gmail.com.

Jean-Pierre Hubschman (JP)

Retina Division, Stein Eye Institute, UCLA, Los Angeles, California, USA.

David Sarraf (D)

Retinal Disorders and Ophthalmic Genetics Division, Stein Eye Institute, UCLA, Los Angeles, California, USA.
Department of Ophthalmology, Greater Los Angeles VA Healthcare Center, Los Angeles, California, USA.

Marta S Figueroa (MS)

Department of Ophthalmology, Ramon y Cajal University Hospital, Madrid, Spain.

Ferdinando Bottoni (F)

Eye Clinic, Department of Biomedical and Clinical Science 'Luigi Sacco', Sacco Hospital, University of Milan, Milan, Italy.

Roberto dell'Omo (R)

Department of Medicine and Health Sciences, University of Molise, Campobasso, Italy.

Christine A Curcio (CA)

Department of Ophthalmology, University of Alabama at Birmingham College of Arts and Sciences, Birmingham, Alabama, USA.

Patrizio Seidenari (P)

Department of Ophthalmology, Fatebenefratelli-Oftalmico Hospital, ASST-Fatebenefratelli-Sacco, Milan, Italy.

Giulia Delledonne (G)

Eye Clinic, Department of Biomedical and Clinical Science 'Luigi Sacco', Sacco Hospital, University of Milan, Milan, Italy.

Robert Gunzenhauser (R)

Retina Division, Stein Eye Institute, UCLA, Los Angeles, California, USA.

Mariantonia Ferrara (M)

Eye Unit, Humanitas University, Humanitas-Gavazzeni Hospital, Bergamo, Italy.

Adrian Au (A)

Retinal Disorders and Ophthalmic Genetics Division, Stein Eye Institute, UCLA, Los Angeles, California, USA.

Gianni Virgili (G)

Department of Ophthalmology, Careggi University Hospital, University of Florence, Florence, Italy.

Antonio Scialdone (A)

Department of Ophthalmology, Fatebenefratelli-Oftalmico Hospital, ASST-Fatebenefratelli-Sacco, Milan, Italy.

Rodolfo Repetto (R)

Department of Civil, Chemical and Environmental Engineering, University of Genoa, Genoa, Italy.

Mario R Romano (MR)

Eye Unit, Humanitas University, Humanitas-Gavazzeni Hospital, Bergamo, Italy.

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