AKR1D1 and CYP7B1 mutations in patients with inborn errors of bile acid metabolism: Possibly underdiagnosed diseases.


Journal

Pediatrics and neonatology
ISSN: 2212-1692
Titre abrégé: Pediatr Neonatol
Pays: Singapore
ID NLM: 101484755

Informations de publication

Date de publication:
02 2020
Historique:
received: 05 02 2019
revised: 22 05 2019
accepted: 28 06 2019
pubmed: 25 7 2019
medline: 21 8 2020
entrez: 25 7 2019
Statut: ppublish

Résumé

Inborn errors of bile acid metabolism (IEBAM) cause rare but treatable genetic disorders that can present as neonatal cholestasis or neurological diseases. Without timely primary bile acid treatment, patients may develop liver failure early in life. This study aimed to analyze the types and treatment outcomes of IEBAM in Taiwanese infants and document the allele frequency of CYP7B1 hot spot mutations in the population. Urine samples from patients with infantile intrahepatic cholestasis and suspected IEBAM were subjected to urinary bile acid analysis by gas chromatography-mass spectrometry (GC/MS). Genetic diagnoses were made using direct sequencing or next-generation sequencing. We also tested healthy control subjects for a probable hot spot point mutation of CYP7B1. Among the 75 patients with infantile intrahepatic cholestasis tested during 2000 -2016, three had ∆ Distinct types of IEBAM disease were found in the Taiwanese population. Patients with early diagnosis and early treatment had a favorable outcome. IEBAM prevalence rates may be higher than expected due to the presence of heterozygous mutations in the general population.

Sections du résumé

BACKGROUND
Inborn errors of bile acid metabolism (IEBAM) cause rare but treatable genetic disorders that can present as neonatal cholestasis or neurological diseases. Without timely primary bile acid treatment, patients may develop liver failure early in life. This study aimed to analyze the types and treatment outcomes of IEBAM in Taiwanese infants and document the allele frequency of CYP7B1 hot spot mutations in the population.
METHODS
Urine samples from patients with infantile intrahepatic cholestasis and suspected IEBAM were subjected to urinary bile acid analysis by gas chromatography-mass spectrometry (GC/MS). Genetic diagnoses were made using direct sequencing or next-generation sequencing. We also tested healthy control subjects for a probable hot spot point mutation of CYP7B1.
RESULTS
Among the 75 patients with infantile intrahepatic cholestasis tested during 2000 -2016, three had ∆
CONCLUSION
Distinct types of IEBAM disease were found in the Taiwanese population. Patients with early diagnosis and early treatment had a favorable outcome. IEBAM prevalence rates may be higher than expected due to the presence of heterozygous mutations in the general population.

Identifiants

pubmed: 31337596
pii: S1875-9572(19)30095-6
doi: 10.1016/j.pedneo.2019.06.009
pii:
doi:

Substances chimiques

Bile Acids and Salts 0
Oxidoreductases EC 1.-
Steroid Hydroxylases EC 1.14.-
Cytochrome P450 Family 7 EC 1.14.14.23
CYP7B1 protein, human EC 1.14.14.29
3-oxo-5 beta-steroid delta 4-dehydrogenase EC 1.3.99.6

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

75-83

Informations de copyright

Copyright © 2019. Published by Elsevier B.V.

Auteurs

Ju-Yin Chen (JY)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan; Department of Pediatrics, National Taiwan University Hospital Yunlin Branch, Yunlin, Taiwan.

Jia-Feng Wu (JF)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan.

Akihiko Kimura (A)

Department of Pediatrics and Child Health, Kurume University School of Medicine, Kurume, Japan.

Hiroshi Nittono (H)

Junshin Clinic Bile Acid Institute, Meguro-ku, Tokyo, Japan.

Bang-Yu Liou (BY)

Hepatitis Research Centre, National Taiwan University Hospital, Taipei, Taiwan.

Chee-Seng Lee (CS)

Department of Pediatrics, Taipei Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, Taipei, Taiwan.

Ho-Sheng Chen (HS)

Department of Pediatrics, National Taiwan University Hospital Hsin-Chu Branch, Hsin-Chu, Taiwan.

Yu-Chun Chiu (YC)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan; Department of Medical Education, National Taiwan University Hospital, Taipei, Taiwan.

Yen-Hsuan Ni (YH)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan; Hepatitis Research Centre, National Taiwan University Hospital, Taipei, Taiwan.

Steven Shinn-Forng Peng (SS)

Department of Radiology, National Taiwan University Hospital, Taipei, Taiwan.

Wang-Tso Lee (WT)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan.

I-Jung Tsai (IJ)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan.

Mei-Hwei Chang (MH)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan; Hepatitis Research Centre, National Taiwan University Hospital, Taipei, Taiwan.

Huey-Ling Chen (HL)

Department of Pediatrics, National Taiwan University Hospital, Taipei, Taiwan; Hepatitis Research Centre, National Taiwan University Hospital, Taipei, Taiwan; Department of Medical Education and Bioethics, Graduate Institute of Medical Education and Bioethics, National Taiwan University College of Medicine, Taipei, Taiwan. Electronic address: hueyling@ntu.edu.tw.

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Classifications MeSH