Irisin vs. Treadmill Exercise in Post Myocardial Infarction Cardiac Rehabilitation in Rats.


Journal

Archives of medical research
ISSN: 1873-5487
Titre abrégé: Arch Med Res
Pays: United States
ID NLM: 9312706

Informations de publication

Date de publication:
02 2019
Historique:
received: 06 02 2019
revised: 03 05 2019
accepted: 22 05 2019
entrez: 28 7 2019
pubmed: 28 7 2019
medline: 27 2 2020
Statut: ppublish

Résumé

Irisin is an exercise-induced myokine that could play a role in post-myocardial infarction (MI) cardiac rehabilitation. We investigated the ability of dihydromyricetin to mimic the effects of exercise on raising serum irisin and on enhancing cardiac function and remodeling following MI in rats. MI was induced in albino rats by subcutaneous injection of isoproterenol (85 mg/kg) for 2 consecutive days at an interval of 24 h. One week post-MI, rats either underwent physical exercise by running on a motor-driven treadmill at 25 m/min, 30 min/d, 5 d/week or received orally dihydromyricetin 100 mg/kg/d, for 8 weeks. Exercise and dihydromyricetin raised serum irisin 1.8 and 1.9 folds as compared to sedentary rats (p <0.001) with no difference between both regimens (p = 0.992). There was an improvement of cardiac remodeling where β-myosin heavy chain level was not different in exercise and dihydromyricetin groups from normal group (p = 0.695, p = 0.470). The heart rate variability domains increased back to normal. However, exercise was superior to dihydromyricetin in improving cardiac contractility by increasing carotid blood flow, stroke volume and cardiac output to be insignificant from normal rats (p = 0.899, p = 0.850, p = 0.912). Meanwhile, treatment with dihydromyricetin showed reduction by 29% of carotid blood flow, 24% of stroke volume and 25% of cardiac output compared to normal rats (p <0.001). DHM could mimic the effect of exercise in stimulating irisin secretion but it is not as effective as exercise in improving myocardial contractility.

Sections du résumé

BACKGROUND
Irisin is an exercise-induced myokine that could play a role in post-myocardial infarction (MI) cardiac rehabilitation.
AIM OF THE STUDY
We investigated the ability of dihydromyricetin to mimic the effects of exercise on raising serum irisin and on enhancing cardiac function and remodeling following MI in rats.
METHODS
MI was induced in albino rats by subcutaneous injection of isoproterenol (85 mg/kg) for 2 consecutive days at an interval of 24 h. One week post-MI, rats either underwent physical exercise by running on a motor-driven treadmill at 25 m/min, 30 min/d, 5 d/week or received orally dihydromyricetin 100 mg/kg/d, for 8 weeks.
RESULTS
Exercise and dihydromyricetin raised serum irisin 1.8 and 1.9 folds as compared to sedentary rats (p <0.001) with no difference between both regimens (p = 0.992). There was an improvement of cardiac remodeling where β-myosin heavy chain level was not different in exercise and dihydromyricetin groups from normal group (p = 0.695, p = 0.470). The heart rate variability domains increased back to normal. However, exercise was superior to dihydromyricetin in improving cardiac contractility by increasing carotid blood flow, stroke volume and cardiac output to be insignificant from normal rats (p = 0.899, p = 0.850, p = 0.912). Meanwhile, treatment with dihydromyricetin showed reduction by 29% of carotid blood flow, 24% of stroke volume and 25% of cardiac output compared to normal rats (p <0.001).
CONCLUSIONS
DHM could mimic the effect of exercise in stimulating irisin secretion but it is not as effective as exercise in improving myocardial contractility.

Identifiants

pubmed: 31349953
pii: S0188-4409(19)30127-4
doi: 10.1016/j.arcmed.2019.05.009
pii:
doi:

Substances chimiques

FNDC5 protein, rat 0
Fibronectins 0
Flavonols 0
dihydromyricetin KD8QND6427

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

44-54

Informations de copyright

Copyright © 2019 IMSS. Published by Elsevier Inc. All rights reserved.

Auteurs

Passainte S Hassaan (PS)

Department of Medical Physiology, Faculty of Medicine, Alexandria University, Alexandria, Egypt.

Seham Zakaria Nassar (SZ)

Department of Medical Physiology, Faculty of Medicine, Alexandria University, Alexandria, Egypt. Electronic address: drsehamn2001@yahoo.com.

Yasmine Issa (Y)

Department of Medical Biochemistry, Faculty of Medicine, Alexandria University, Alexandria, Egypt.

Noha Zahran (N)

Department of Histology and cellular biology, Faculty of Medicine, Alexandria University, Alexandria, Egypt.

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Classifications MeSH