The Ottawa score performs poorly in cancer patients with incidental pulmonary embolism.


Journal

Thrombosis research
ISSN: 1879-2472
Titre abrégé: Thromb Res
Pays: United States
ID NLM: 0326377

Informations de publication

Date de publication:
Sep 2019
Historique:
received: 24 05 2019
revised: 09 07 2019
accepted: 09 07 2019
pubmed: 29 7 2019
medline: 14 2 2020
entrez: 29 7 2019
Statut: ppublish

Résumé

The Ottawa score was previously developed to predict recurrent venous thromboembolism (VTE) in cancer patients with VTE. The performance of this score in patients with incidental VTE is currently unclear. To evaluate the performance of the Ottawa risk score in cancer patients with incidental pulmonary embolism included in an international, prospective, observational cohort study. The score was used to classify patients as high (≥1), intermediate (0), or low risk (≤-1). The discriminative performance of the score was estimated by calculating the cumulative incidence of recurrent VTE for all groups, the time-dependent c-statistic, and the sub-distribution hazard ratio (SHR), using a competing risk approach. Of the 691 patients for which the Ottawa score could be calculated, 25 (3.6%) had recurrent VTE during 6-month follow-up and 38 (5.5%) during 12-month follow-up. The c-statistics of the continuous score at 6 and 12 months were 0.45 (95% CI, 0.36-0.54) and 0.51 (95% CI, 0.46-0.59), respectively. The 6-month cumulative incidences of recurrent VTE for those at low, intermediate, and high risk were 3.9% (95% CI, 1.5-8.4), 3.6% (95% CI, 1.9-6.2), and 3.6% (95% CI, 1.8-6.5), respectively. A sensitivity analysis restricted to the on-treatment period yielded similar results. None of the Ottawa risk score items were significantly associated with recurrent VTE. In cancer patients with incidental pulmonary embolism, the Ottawa risk score has a poor predictive value for recurrent VTE, which does not support the use of the score in this patient population.

Sections du résumé

BACKGROUND BACKGROUND
The Ottawa score was previously developed to predict recurrent venous thromboembolism (VTE) in cancer patients with VTE. The performance of this score in patients with incidental VTE is currently unclear.
AIM OBJECTIVE
To evaluate the performance of the Ottawa risk score in cancer patients with incidental pulmonary embolism included in an international, prospective, observational cohort study.
METHODS METHODS
The score was used to classify patients as high (≥1), intermediate (0), or low risk (≤-1). The discriminative performance of the score was estimated by calculating the cumulative incidence of recurrent VTE for all groups, the time-dependent c-statistic, and the sub-distribution hazard ratio (SHR), using a competing risk approach.
RESULTS RESULTS
Of the 691 patients for which the Ottawa score could be calculated, 25 (3.6%) had recurrent VTE during 6-month follow-up and 38 (5.5%) during 12-month follow-up. The c-statistics of the continuous score at 6 and 12 months were 0.45 (95% CI, 0.36-0.54) and 0.51 (95% CI, 0.46-0.59), respectively. The 6-month cumulative incidences of recurrent VTE for those at low, intermediate, and high risk were 3.9% (95% CI, 1.5-8.4), 3.6% (95% CI, 1.9-6.2), and 3.6% (95% CI, 1.8-6.5), respectively. A sensitivity analysis restricted to the on-treatment period yielded similar results. None of the Ottawa risk score items were significantly associated with recurrent VTE.
CONCLUSION CONCLUSIONS
In cancer patients with incidental pulmonary embolism, the Ottawa risk score has a poor predictive value for recurrent VTE, which does not support the use of the score in this patient population.

Identifiants

pubmed: 31352267
pii: S0049-3848(19)30288-9
doi: 10.1016/j.thromres.2019.07.005
pii:
doi:

Substances chimiques

Anticoagulants 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

59-63

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Frits I Mulder (FI)

Amsterdam UMC, University of Amsterdam, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands; Tergooi Hospitals, Hilversum, the Netherlands. Electronic address: f.i.mulder@amsterdamumc.nl.

Noémie Kraaijpoel (N)

Amsterdam UMC, University of Amsterdam, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands.

Marcello Di Nisio (M)

Department of Medicine and Ageing Sciences, University G. D'Annunzio, Chieti-Pescara, Italy.

Guy Meyer (G)

Hôpital Européen Georges-Pompidou, Paris, France; Université Paris Descartes, Paris, France; French Clinical Research Infrastructure Network, Saint-Étienne, France.

Isabelle Mahé (I)

French Clinical Research Infrastructure Network, Saint-Étienne, France; Institut National de la Santé et de la Recherche Médicale, Unité Mixte de Recherche S1140, Faculté de Pharmacie, Paris, France; Université Paris Diderot, France; Assistance Publique-Hôpitaux de Paris, Hôpital Louis Mourier, Colombes, France.

Andrés Muñoz (A)

Hospital General Universitario Gregorio Marañon, Madrid, Spain.

Laurent Bertoletti (L)

French Clinical Research Infrastructure Network, Saint-Étienne, France; Service de Médecine Vasculaire et Thérapeutique, CHU de St-Etienne, Saint-Etienne, France; Institut National de la Santé et de la Recherche Médicale, Unité Mixte de Recherche 1059, Equipe Dysfonction Vasculaire et Hémostase, Université Jean-Monnet, Saint-Étienne, France; Institut National de la Santé et de la Recherche Médicale, CIC-1408, Centre Hospitalier Universitaire Saint-Etienne, Saint-Étienne, France.

Mercedes Biosca (M)

Hospital Vall d'Hebron, Barcelona, Spain.

Anthony Maraveyas (A)

The Queen's Centre for Oncology and Haematology, Hull and York Medical School, Castle Hill, United Kingdom.

Harry R Büller (HR)

Amsterdam UMC, University of Amsterdam, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands.

Nick van Es (N)

Amsterdam UMC, University of Amsterdam, Amsterdam Cardiovascular Sciences, Amsterdam, the Netherlands.

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