Effects of bioenergetics, temperature and cadmium on liver mitochondria reactive oxygen species production and consumption.
Animals
Cadmium
/ toxicity
Cell Respiration
/ drug effects
Electron Transport
Energy Metabolism
Female
Hydrogen Peroxide
/ metabolism
Mitochondria
/ drug effects
Mitochondria, Liver
/ drug effects
NAD
/ metabolism
Oncorhynchus mykiss
/ physiology
Oxygen Consumption
/ drug effects
Reactive Oxygen Species
/ metabolism
Temperature
Water Pollutants, Chemical
/ toxicity
Cadmium
Metabolism
Mitochondria
Reactive oxygen species
Temperature
Journal
Aquatic toxicology (Amsterdam, Netherlands)
ISSN: 1879-1514
Titre abrégé: Aquat Toxicol
Pays: Netherlands
ID NLM: 8500246
Informations de publication
Date de publication:
Sep 2019
Sep 2019
Historique:
received:
06
06
2019
revised:
22
07
2019
accepted:
24
07
2019
pubmed:
5
8
2019
medline:
7
11
2019
entrez:
5
8
2019
Statut:
ppublish
Résumé
A by-product of mitochondrial substrate oxidation and electron transfer to generate cellular energy (ATP) is reactive oxygen species (ROS). Superoxide anion radical and hydrogen peroxide (H
Identifiants
pubmed: 31377504
pii: S0166-445X(19)30469-2
doi: 10.1016/j.aquatox.2019.105264
pii:
doi:
Substances chimiques
Reactive Oxygen Species
0
Water Pollutants, Chemical
0
Cadmium
00BH33GNGH
NAD
0U46U6E8UK
Hydrogen Peroxide
BBX060AN9V
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
105264Informations de copyright
Copyright © 2019 Elsevier B.V. All rights reserved.