Should Pathologically Noninvasive Lung Adenocarcinoma Larger Than 3 cm Be Classified as T1a?


Journal

The Annals of thoracic surgery
ISSN: 1552-6259
Titre abrégé: Ann Thorac Surg
Pays: Netherlands
ID NLM: 15030100R

Informations de publication

Date de publication:
12 2019
Historique:
received: 08 12 2018
revised: 08 06 2019
accepted: 12 06 2019
pubmed: 11 8 2019
medline: 19 3 2020
entrez: 11 8 2019
Statut: ppublish

Résumé

Adenocarcinoma in situ (AIS) is defined as noninvasive adenocarcinoma with a total tumor size including lepidic growth ≤3 cm; tumors >3 cm are classified as T1a. However, a tumor >3 cm that meets the histological criteria of AIS may have a prognosis equivalent to that of AIS. Here we examine the frequency and prognosis of noninvasive adenocarcinoma with a tumor >3 cm and the validity of the current definition of AIS tumor diameter. The study was composed of patients with lung adenocarcinoma completely resected from January 2000 to December 2013 who met AIS histological criteria. Eligible patients were divided into 2 groups-the AIS group (tumor ≤3 cm) and the large AIS group (tumor >3 cm)-and the clinicopathologic characteristics and prognosis were retrospectively compared between the 2 groups. A total of 277 patients were analyzed and large AIS was found in 7.9% (22) of the patients. The 5-year overall survival was 98.3% in the AIS group and 95.5% in the large AIS group. No patient had recurrence. The consolidation size from computed tomography and pathologic alveolar collapse size were both significantly larger in the large AIS group. No significant differences between groups were found in the consolidation-tumor ratio or in the ratio of the pathologic alveolar collapse size to the pathological total tumor size. The prognosis of patients with pathologically noninvasive adenocarcinoma >3 cm was comparable to that of AIS. There is a possibility that tumor diameter need not be a consideration for AIS classification.

Sections du résumé

BACKGROUND
Adenocarcinoma in situ (AIS) is defined as noninvasive adenocarcinoma with a total tumor size including lepidic growth ≤3 cm; tumors >3 cm are classified as T1a. However, a tumor >3 cm that meets the histological criteria of AIS may have a prognosis equivalent to that of AIS. Here we examine the frequency and prognosis of noninvasive adenocarcinoma with a tumor >3 cm and the validity of the current definition of AIS tumor diameter.
METHODS
The study was composed of patients with lung adenocarcinoma completely resected from January 2000 to December 2013 who met AIS histological criteria. Eligible patients were divided into 2 groups-the AIS group (tumor ≤3 cm) and the large AIS group (tumor >3 cm)-and the clinicopathologic characteristics and prognosis were retrospectively compared between the 2 groups.
RESULTS
A total of 277 patients were analyzed and large AIS was found in 7.9% (22) of the patients. The 5-year overall survival was 98.3% in the AIS group and 95.5% in the large AIS group. No patient had recurrence. The consolidation size from computed tomography and pathologic alveolar collapse size were both significantly larger in the large AIS group. No significant differences between groups were found in the consolidation-tumor ratio or in the ratio of the pathologic alveolar collapse size to the pathological total tumor size. The prognosis of patients with pathologically noninvasive adenocarcinoma >3 cm was comparable to that of AIS.
CONCLUSIONS
There is a possibility that tumor diameter need not be a consideration for AIS classification.

Identifiants

pubmed: 31400323
pii: S0003-4975(19)31103-8
doi: 10.1016/j.athoracsur.2019.06.055
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1678-1684

Informations de copyright

Copyright © 2019 The Society of Thoracic Surgeons. Published by Elsevier Inc. All rights reserved.

Auteurs

Kenji Inafuku (K)

Department of Thoracic Surgery, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan. Electronic address: ma03007@yahoo.co.jp.

Tomoyuki Yokose (T)

Department of Pathology, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Hiroyuki Ito (H)

Department of Thoracic Surgery, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Hiroto Narimatsu (H)

Cancer Prevention and Control Division, Kanagawa Cancer Center Research Institute, Yokohama City, Kanagawa, Japan.

Joji Samejima (J)

Department of Thoracic Surgery, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Takuya Nagashima (T)

Department of Thoracic Surgery, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Haruhiko Nakayama (H)

Department of Thoracic Surgery, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Masaki Suzuki (M)

Department of Pathology, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Kouzo Yamada (K)

Department of Thoracic Oncology, Kanagawa Cancer Center, Yokohama City, Kanagawa, Japan.

Munetaka Masuda (M)

Department of Surgery, Yokohama City University, Yokohama City, Kanagawa, Japan.

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