Assessing cognitive impairment in SLE: examining relationships between resting glucose metabolism and anti-NMDAR antibodies with navigational performance.

DNRAb FDG-PET spatial navigation task (snt) systemic lupus erythematosus (sle)

Journal

Lupus science & medicine
ISSN: 2053-8790
Titre abrégé: Lupus Sci Med
Pays: England
ID NLM: 101633705

Informations de publication

Date de publication:
2019
Historique:
received: 12 02 2019
revised: 17 05 2019
accepted: 10 06 2019
entrez: 16 8 2019
pubmed: 16 8 2019
medline: 16 8 2019
Statut: epublish

Résumé

Resting Fluorine-18 fluorodeoxyglucose positron emission tomography (FDG-PET) brain imaging and neuropsychological testing were used to investigate the usefulness of a spatial navigation task (SNT) as a performance benchmark for cognitive impairment related to anti-N-methyl D-aspartate (anti-NMDA) receptor antibodies (DNRAb) in SLE. Neuropsychological assessments, including a desktop 3-D virtual SNT, were performed on 19 SLE participants and 9 healthy control (HC) subjects. SLE participants had stable disease activity and medication doses and no history of neuropsychiatric illness or current use of mind-altering medications. Resting FDG-PET scans were obtained on all SLE participants and compared with a historical set from 25 age-matched and sex-matched HCs. Serum DNRAb titres were measured by ELISA. 11/19 (58%) of SLE participants failed to complete the SNT (SNT-) compared with 2/9 (22%) of HCs. Compared with 7/9 (78%) in HCs, only 2/9 (22%; p=0.037) of SLE participants with high serum DNRAb titres completed the SNT, in contrast to 6/10 (60%; p=0.810) in SLE participants with low DNRAb titres. Voxel-wise comparison of FDG-PET scans between the 8 SLE participants successfully completing the SNT task (SNT+) and the 11 SNT- SLE participants revealed increased metabolism in the SNT+ participants (p<0.001) in the left anterior putamen/caudate, right anterior putamen, left prefrontal cortex (BA 9), right prefrontal cortex (BA 9/10) and left lateral and medial frontal cortex (BA 8). Compared with HCs, the SNT+ group demonstrated increased metabolism in all regions (p<0.02) except for the right prefrontal cortex (BA 9), whereas the SNT- group demonstrated either significantly decreased or similar metabolism in these seven regions. SNT performance is associated with serum DNRAb titres and resting glucose metabolism in the anterior putamen/caudate and frontal cortex, suggesting compensatory neural recruitment in SNT-associated regions is necessary for successful completion of the task. The SNT therefore has potential for use as a marker for SLE-mediated cognitive impairment.

Identifiants

pubmed: 31413849
doi: 10.1136/lupus-2019-000327
pii: lupus-2019-000327
pmc: PMC6667777
doi:

Types de publication

Journal Article

Langues

eng

Pagination

e000327

Déclaration de conflit d'intérêts

Competing interests: None declared.

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Auteurs

Elisabeth Ploran (E)

Department of Psychology, Hofstra University, Hempstead, New York, USA.

Chris Tang (C)

Center for Neurosciences, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Meggan Mackay (M)

The Center for Autoimmune, Musculoskeletal, and Hematopoietic Diseases, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Michael Small (M)

Center for Neurosciences, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Erik Anderson (E)

The Center for Autoimmune, Musculoskeletal, and Hematopoietic Diseases, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Justin Storbeck (J)

Department of Psychology, Queens College, Flushing, New York, USA.

Brittany Bascetta (B)

Department of Psychology, Queens College, Flushing, New York, USA.

Simran Kang (S)

Department of Psychology, Queens College, Flushing, New York, USA.

Cynthia Aranow (C)

The Center for Autoimmune, Musculoskeletal, and Hematopoietic Diseases, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Carl Sartori (C)

The Center for Autoimmune, Musculoskeletal, and Hematopoietic Diseases, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Philip Watson (P)

Department of Psychiatry, Zucker Hillside Hospital, Glen Oaks, New York, USA.

Bruce Volpe (B)

Center for Biomedical Science, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Betty Diamond (B)

The Center for Autoimmune, Musculoskeletal, and Hematopoietic Diseases, Feinstein Institute for Medical Research, Manhasset, New York, USA.

David Eidelberg (D)

Center for Neurosciences, Feinstein Institute for Medical Research, Manhasset, New York, USA.

Classifications MeSH