A multicenter, open-label, single-arm study of anamorelin (ONO-7643) in advanced gastrointestinal cancer patients with cancer cachexia.


Journal

Cancer
ISSN: 1097-0142
Titre abrégé: Cancer
Pays: United States
ID NLM: 0374236

Informations de publication

Date de publication:
01 Dec 2019
Historique:
received: 18 12 2018
revised: 06 06 2019
accepted: 27 06 2019
pubmed: 16 8 2019
medline: 4 6 2020
entrez: 16 8 2019
Statut: ppublish

Résumé

Cancer cachexia is characterized by weight loss and is associated with increased morbidity and mortality in patients with cancer. Anamorelin (ONO-7643; ANAM) is a novel and selective ghrelin receptor agonist that improves appetite, lean body mass (LBM), body weight, and anorexia. This multicenter, open-label, single-arm study investigated the efficacy and safety of 100 mg anamorelin in 50 Japanese patients with advanced and unresectable gastrointestinal (colorectal, gastric, or pancreatic) cancer. ANAM was administered once daily over 12 weeks. The primary endpoint was the proportion of patients that maintained or gained LBM over the course of the study. Secondary endpoints included changes in LBM, body weight, quality of life (QoL), and nutritional status biomarkers. The proportion of patients who responded to treatment was 63.3% (95% CI, 48.3%-76.6%), with a least square mean ± SE change in LBM and body weight from baseline of 1.89 ± 0.36 kg and 1.41 ± 0.61 kg, respectively. Appetite-related questions on the QoL questionnaire showed that ANAM improved appetite. Adverse events occurred in 79.6% of patients, and the most common treatment-related adverse events were increased γ-glutamyl transpeptidase (8.2%), diabetes mellitus (6.1%), hyperglycemia (6.1%), and prolonged QRS complex (6.1%). ANAM improved anorexia and patients' nutritional status, resulting in rapid increases in LBM and body weight in patients with advanced gastrointestinal cancer who had cancer cachexia. ANAM treatment was well tolerated over 12 weeks. ANAM is a potential clinically beneficial pharmacotherapeutic option for patients with advanced gastrointestinal cancer who have cancer cachexia.

Sections du résumé

BACKGROUND BACKGROUND
Cancer cachexia is characterized by weight loss and is associated with increased morbidity and mortality in patients with cancer. Anamorelin (ONO-7643; ANAM) is a novel and selective ghrelin receptor agonist that improves appetite, lean body mass (LBM), body weight, and anorexia.
METHODS METHODS
This multicenter, open-label, single-arm study investigated the efficacy and safety of 100 mg anamorelin in 50 Japanese patients with advanced and unresectable gastrointestinal (colorectal, gastric, or pancreatic) cancer. ANAM was administered once daily over 12 weeks. The primary endpoint was the proportion of patients that maintained or gained LBM over the course of the study. Secondary endpoints included changes in LBM, body weight, quality of life (QoL), and nutritional status biomarkers.
RESULTS RESULTS
The proportion of patients who responded to treatment was 63.3% (95% CI, 48.3%-76.6%), with a least square mean ± SE change in LBM and body weight from baseline of 1.89 ± 0.36 kg and 1.41 ± 0.61 kg, respectively. Appetite-related questions on the QoL questionnaire showed that ANAM improved appetite. Adverse events occurred in 79.6% of patients, and the most common treatment-related adverse events were increased γ-glutamyl transpeptidase (8.2%), diabetes mellitus (6.1%), hyperglycemia (6.1%), and prolonged QRS complex (6.1%).
CONCLUSIONS CONCLUSIONS
ANAM improved anorexia and patients' nutritional status, resulting in rapid increases in LBM and body weight in patients with advanced gastrointestinal cancer who had cancer cachexia. ANAM treatment was well tolerated over 12 weeks. ANAM is a potential clinically beneficial pharmacotherapeutic option for patients with advanced gastrointestinal cancer who have cancer cachexia.

Identifiants

pubmed: 31415709
doi: 10.1002/cncr.32406
pmc: PMC6900019
doi:

Substances chimiques

Hydrazines 0
Oligopeptides 0
anamorelin DD5RBA1NKF

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

4294-4302

Subventions

Organisme : Ono Pharmaceutical Co., Ltd.
Organisme : Ono Pharmaceutical Company Ltd

Informations de copyright

© 2019 The Authors. Cancer published by Wiley Periodicals, Inc. on behalf of American Cancer Society.

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Auteurs

Satoshi Hamauchi (S)

Department of Gastrointestinal Oncology, Shizuoka Cancer Center, Shizuoka, Japan.

Junji Furuse (J)

Department of Medical Oncology, Faculty of Medicine, Kyorin University, Tokyo, Japan.

Toshimi Takano (T)

Department of Medical Oncology, Toranomon Hospital, Tokyo, Japan.

Yoshinori Munemoto (Y)

Department of Surgery, Fukui-ken Saiseikai Hospital, Fukui, Japan.

Ken Furuya (K)

Department of Gastroenterology and Hepatology, Japan Community Health Care Organization, Hokkaido Hospital, Hokkaido, Japan.

Hideo Baba (H)

Department of Gastroenterological Surgery, Graduate School of Medical Sciences, Kumamoto University, Kumamoto, Japan.

Manabu Takeuchi (M)

Department of Gastroenterology, Nagaoka Red Cross Hospital, Niigata, Japan.

Yasuhiro Choda (Y)

Department of Surgery, Hiroshima Citizens Hospital, Hiroshima, Japan.

Takashi Higashiguchi (T)

Department of Surgery and Palliative Medicine, Fujita Health University School of Medicine, Aichi, Japan.

Tateaki Naito (T)

Division of Thoracic Oncology, Shizuoka Cancer Center, Shizuoka, Japan.

Kei Muro (K)

Department of Clinical Oncology, Aichi Cancer Center Hospital, Nagoya, Japan.

Koichi Takayama (K)

Department of Pulmonary Medicine, Kyoto Prefectural University of Medicine, Kyoto, Japan.

Shusuke Oyama (S)

Data Science, Ono Pharmaceutical Company Ltd, Osaka, Japan.

Toru Takiguchi (T)

Clinical Development Planning, Ono Pharmaceutical Company Ltd, Osaka, Japan.

Naoyuki Komura (N)

Clinical Development Planning, Ono Pharmaceutical Company Ltd, Osaka, Japan.

Kazuo Tamura (K)

General Medical Research Center, Fukuoka University, Fukuoka, Japan.

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