Expression of IL-34 correlates with macrophage infiltration and prognosis of diffuse large B-cell lymphoma.

CSF1R DLBCL IL‐34 Macrophages M‐CSF

Journal

Clinical & translational immunology
ISSN: 2050-0068
Titre abrégé: Clin Transl Immunology
Pays: Australia
ID NLM: 101638268

Informations de publication

Date de publication:
2019
Historique:
received: 16 04 2019
revised: 16 07 2019
accepted: 16 07 2019
entrez: 17 8 2019
pubmed: 17 8 2019
medline: 17 8 2019
Statut: epublish

Résumé

Infiltration of macrophages through the tyrosine kinase receptor CSF1R is a poor prognosis factor in various solid tumors. Indeed, these tumors produce CSF1R ligand, macrophage colony-stimulating factor (M-CSF) or interleukin-34 (IL-34). However, the significance of these cytokines, particularly, the newly discovered IL-34 in haematological malignancies, is not fully understood. We therefore analysed the role of IL-34 in diffuse large B-cell lymphoma (DLBCL), the most common subtype of malignant lymphoma. We analysed formalin-fixed paraffin-embedded lymphoma tissues of 135 DLBCL patients for the expression of IL-34 and the number of macrophages, and the survival of these patients. The expression of IL-34 in DLBCL cell lines and the activity of IL-34 to induce the migration of monocytic cells were also characterised. Several lymphoma tissues showed a clear IL-34 signal, and such signal was detectable in 36% of patients. DLBCL cell lines also expressed IL-34. Interestingly, the percentage of IL-34 Our results raise the possibility that IL-34 in lymphoma tissues of DLBCL patients recruits monocytes, leading to the higher number of macrophages in the tissues and poor prognosis of patients. IL-34 may be an additional therapeutic target of DLBCL.

Identifiants

pubmed: 31417675
doi: 10.1002/cti2.1074
pii: CTI21074
pmc: PMC6691654
doi:

Types de publication

Journal Article

Langues

eng

Pagination

e1074

Déclaration de conflit d'intérêts

The authors declare no conflict of interest.

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Auteurs

Osamu Noyori (O)

International Research Center for Medical Sciences Joint Research Center for Human Retrovirus Infection Kumamoto University Kumamoto Japan.

Yoshihiro Komohara (Y)

Department of Cell Pathology Graduate School of Medical Sciences Kumamoto University Kumamoto Japan.

Hesham Nasser (H)

International Research Center for Medical Sciences Joint Research Center for Human Retrovirus Infection Kumamoto University Kumamoto Japan.

Masateru Hiyoshi (M)

International Research Center for Medical Sciences Joint Research Center for Human Retrovirus Infection Kumamoto University Kumamoto Japan.
Present address: Department of Safety Research on Blood and Biologics National Institute of Infectious Diseases Tokyo Japan.

Chaoya Ma (C)

Department of Cell Pathology Graduate School of Medical Sciences Kumamoto University Kumamoto Japan.

Cheng Pan (C)

Department of Cell Pathology Graduate School of Medical Sciences Kumamoto University Kumamoto Japan.

Joaquim Carreras (J)

Department of Pathology School of Medicine Tokai University Kanagawa Japan.

Naoya Nakamura (N)

Department of Pathology School of Medicine Tokai University Kanagawa Japan.

Ai Sato (A)

Department of Hematology and Oncology School of Medicine Tokai University Kanagawa Japan.

Kiyoshi Ando (K)

Department of Hematology and Oncology School of Medicine Tokai University Kanagawa Japan.

Yutaka Okuno (Y)

Department of Hematology, Rheumatology, and Infectious Diseases Graduate School of Medical Sciences Kumamoto University Kumamoto Japan.

Kisato Nosaka (K)

Department of Hematology, Rheumatology, and Infectious Diseases Graduate School of Medical Sciences Kumamoto University Kumamoto Japan.

Masao Matsuoka (M)

Department of Hematology, Rheumatology, and Infectious Diseases Graduate School of Medical Sciences Kumamoto University Kumamoto Japan.

Shinya Suzu (S)

International Research Center for Medical Sciences Joint Research Center for Human Retrovirus Infection Kumamoto University Kumamoto Japan.

Classifications MeSH