Percutaneous obliteration of the right ventricle to avoid coronary damage by sinusoids in patients with pulmonary atresia intact ventricular septum during staged single ventricle palliation.


Journal

Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
ISSN: 1522-726X
Titre abrégé: Catheter Cardiovasc Interv
Pays: United States
ID NLM: 100884139

Informations de publication

Date de publication:
01 Nov 2019
Historique:
received: 01 02 2019
revised: 08 08 2019
accepted: 12 08 2019
pubmed: 23 8 2019
medline: 26 8 2020
entrez: 22 8 2019
Statut: ppublish

Résumé

Suprasystemic pressure waves can damage the coronary arteries resulting in myocardial ischemia and excess early mortality. We aimed to reduce the coronary pressure wave through the sinusoids by abolishing RV volume with percutaneous devices. Four patients with PA-IVS and coronary sinusoids from the hypertensive rudimentary RV were evaluated at a median age 26.6 months (range: 2.7-51.7). Right ventricle coronary dependent flow to the left ventricular myocardium was excluded. All four patients had dual perfusion with competitive flow from the RV through the sinusoids to the coronary arteries. Devices used were: Amplatzer vascular plug II of 10-16 mm; 27 coils (diameter 5-15 mm) in the oldest patient. Right ventricular angiography after cavity obliteration showed no more significant coronary perfusion through the sinusoids. There were no complications or deaths. Only minor and transient changes in the levels of troponin were observed. Coronary angiography at pre-Fontan evaluation showed no progress of coronary abnormalities in two patients. In selected patients with functionally single left ventricle, obliteration of the hypertensive RV cavity by percutaneous devices is safe and abolishes the systolic pressure wave in coronary sinusoids. When performed early, this may halt coronary damage and avoid excess mortality.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
Suprasystemic pressure waves can damage the coronary arteries resulting in myocardial ischemia and excess early mortality. We aimed to reduce the coronary pressure wave through the sinusoids by abolishing RV volume with percutaneous devices.
METHODS AND RESULTS RESULTS
Four patients with PA-IVS and coronary sinusoids from the hypertensive rudimentary RV were evaluated at a median age 26.6 months (range: 2.7-51.7). Right ventricle coronary dependent flow to the left ventricular myocardium was excluded. All four patients had dual perfusion with competitive flow from the RV through the sinusoids to the coronary arteries. Devices used were: Amplatzer vascular plug II of 10-16 mm; 27 coils (diameter 5-15 mm) in the oldest patient. Right ventricular angiography after cavity obliteration showed no more significant coronary perfusion through the sinusoids. There were no complications or deaths. Only minor and transient changes in the levels of troponin were observed. Coronary angiography at pre-Fontan evaluation showed no progress of coronary abnormalities in two patients.
CONCLUSION CONCLUSIONS
In selected patients with functionally single left ventricle, obliteration of the hypertensive RV cavity by percutaneous devices is safe and abolishes the systolic pressure wave in coronary sinusoids. When performed early, this may halt coronary damage and avoid excess mortality.

Identifiants

pubmed: 31433549
doi: 10.1002/ccd.28457
doi:

Types de publication

Journal Article Video-Audio Media

Langues

eng

Sous-ensembles de citation

IM

Pagination

722-726

Subventions

Organisme : Eddy Merckx Research Foundation

Informations de copyright

© 2019 Wiley Periodicals, Inc.

Références

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Auteurs

Jelena Hubrechts (J)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.

Bjorn Cools (B)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.

Stephen C Brown (SC)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.
Department of Pediatric and Congenital Cardiology, University of the Free State, Bloemfontein, Free State, South Africa.

Benedicte Eyskens (B)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.

Ruth Heying (R)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.

Derize Boshoff (D)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.

Marc Gewillig (M)

Department of Pediatric and Congenital Cardiology, University Hospitals Leuven, Leuven, Belgium.

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