Renal Function and Outcomes With Dabigatran Dual Antithrombotic Therapy in Atrial Fibrillation Patients After PCI.


Journal

JACC. Cardiovascular interventions
ISSN: 1876-7605
Titre abrégé: JACC Cardiovasc Interv
Pays: United States
ID NLM: 101467004

Informations de publication

Date de publication:
26 08 2019
Historique:
received: 28 01 2019
revised: 17 04 2019
accepted: 21 05 2019
entrez: 24 8 2019
pubmed: 24 8 2019
medline: 2 9 2020
Statut: ppublish

Résumé

The study sought to evaluate the effect of dabigatran dual therapy versus warfarin triple therapy across categories of renal function in the RE-DUAL PCI (Randomized Evaluation of Dual Antithrombotic Therapy with Dabigatran versus Triple Therapy with Warfarin in Patients with Nonvalvular Atrial Fibrillation Undergoing Percutaneous Coronary Intervention) trial. The RE-DUAL PCI (NCT02164864) trial of patients with atrial fibrillation undergoing percutaneous coronary intervention reported that dabigatran dual therapy (110 or 150 mg twice daily, plus clopidogrel or ticagrelor) reduced the primary endpoint of major bleeding events (MBE) or clinically relevant nonmajor bleeding events (CRNMBE) compared with warfarin triple therapy, with noninferiority in overall thromboembolic events. Risk of a first MBE or CRNMBE and the composite of death or thromboembolic event (DTE) or unplanned revascularization were evaluated in 2,725 patients according to baseline creatinine clearance (CrCl) categories: 30 to <50, 50 to <80, and ≥80 ml/min. Compared with warfarin, dabigatran 110 mg dual therapy reduced risk of MBE or CRNMBE across all categories of CrCl (p for interaction = 0.19). Dabigatran 150 mg dual therapy reduced risk of MBE or CRNMBE regardless of the CrCl category (p for interaction = 0.31). Risk of DTE or unplanned revascularization was similar to warfarin triple therapy for dabigatran 110 mg dual therapy across all CrCl categories. Dabigatran 150 mg dual therapy versus warfarin triple therapy had similar risk for DTE or unplanned revascularization in patients with CrCl 30 to <80 ml/min and lower risk at CrCl ≥80 ml/min (p for interaction = 0.02). In the RE-DUAL PCI trial, dabigatran dual therapy reduced bleeding events versus warfarin triple therapy irrespective of renal function, with overall similar risks of thromboembolic events but lower risks with dabigatran 150 mg in patients with normal CrCl.

Sections du résumé

OBJECTIVES
The study sought to evaluate the effect of dabigatran dual therapy versus warfarin triple therapy across categories of renal function in the RE-DUAL PCI (Randomized Evaluation of Dual Antithrombotic Therapy with Dabigatran versus Triple Therapy with Warfarin in Patients with Nonvalvular Atrial Fibrillation Undergoing Percutaneous Coronary Intervention) trial.
BACKGROUND
The RE-DUAL PCI (NCT02164864) trial of patients with atrial fibrillation undergoing percutaneous coronary intervention reported that dabigatran dual therapy (110 or 150 mg twice daily, plus clopidogrel or ticagrelor) reduced the primary endpoint of major bleeding events (MBE) or clinically relevant nonmajor bleeding events (CRNMBE) compared with warfarin triple therapy, with noninferiority in overall thromboembolic events.
METHODS
Risk of a first MBE or CRNMBE and the composite of death or thromboembolic event (DTE) or unplanned revascularization were evaluated in 2,725 patients according to baseline creatinine clearance (CrCl) categories: 30 to <50, 50 to <80, and ≥80 ml/min.
RESULTS
Compared with warfarin, dabigatran 110 mg dual therapy reduced risk of MBE or CRNMBE across all categories of CrCl (p for interaction = 0.19). Dabigatran 150 mg dual therapy reduced risk of MBE or CRNMBE regardless of the CrCl category (p for interaction = 0.31). Risk of DTE or unplanned revascularization was similar to warfarin triple therapy for dabigatran 110 mg dual therapy across all CrCl categories. Dabigatran 150 mg dual therapy versus warfarin triple therapy had similar risk for DTE or unplanned revascularization in patients with CrCl 30 to <80 ml/min and lower risk at CrCl ≥80 ml/min (p for interaction = 0.02).
CONCLUSIONS
In the RE-DUAL PCI trial, dabigatran dual therapy reduced bleeding events versus warfarin triple therapy irrespective of renal function, with overall similar risks of thromboembolic events but lower risks with dabigatran 150 mg in patients with normal CrCl.

Identifiants

pubmed: 31439336
pii: S1936-8798(19)31254-3
doi: 10.1016/j.jcin.2019.05.050
pii:
doi:

Substances chimiques

Antithrombins 0
Fibrinolytic Agents 0
Platelet Aggregation Inhibitors 0
Warfarin 5Q7ZVV76EI
Dabigatran I0VM4M70GC

Banques de données

ClinicalTrials.gov
['NCT02164864']

Types de publication

Comparative Study Journal Article Multicenter Study Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1553-1561

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019. Published by Elsevier Inc.

Auteurs

Stefan H Hohnloser (SH)

Department of Cardiology, Johann Wolfgang Goethe University, Frankfurt am Main, Germany. Electronic address: hohnloser@em.uni-frankfurt.de.

Philippe Gabriel Steg (PG)

Département Hospitalo-Universitaire, French Alliance for Cardiovascular Trials (FACT), Hôpital Bichat, Paris, France; Université Paris Diderot, Paris, France; INSERM U_1148, Paris, France; Hôpital Bichat, Assistance Publique-Hôpitaux de Paris, Paris, France.

Jonas Oldgren (J)

Uppsala Clinical Research Center and Department of Medical Sciences, Uppsala, Sweden.

Georg Nickenig (G)

Medizinische Klinik und Poliklinik II, University of Bonn, Bonn, Germany.

Robert G Kiss (RG)

Medical Centre, Hungarian Defence Forces, Budapest, Hungary.

Zeki Ongen (Z)

Department of Cardiology, Istanbul University Cerrahpasa, Istanbul, Turkey.

Jose L Navarro Estrada (JL)

Department of Cardiology, Hospital Italiano de Buenos Aires, Buenos Aires, Argentina.

Ton Oude Ophuis (T)

Department of Cardiology, Canisius-Wilhelmina Hospital, Nijmegen, the Netherlands.

Gregory Y H Lip (GYH)

Liverpool Centre for Cardiovascular Science, University of Liverpool and Liverpool Heart & Chest Hospital, Liverpool, United Kingdom; Aalborg Thrombosis Research Unit, Department of Clinical Medicine, Aalborg University, Aalborg, Denmark.

Matias Nordaby (M)

Boehringer Ingelheim International GmbH, Ingelheim, Germany.

Eva Kleine (E)

Boehringer Ingelheim International GmbH, Ingelheim, Germany.

Jurrien M Ten Berg (JM)

Department of Cardiology, St. Antonius Ziekenhuis, Nieuwegein, the Netherlands.

Deepak L Bhatt (DL)

Brigham and Women's Hospital Heart and Vascular Center, and Harvard Medical School, Boston, Massachusetts.

Christopher P Cannon (CP)

Brigham and Women's Hospital Heart and Vascular Center, and Harvard Medical School, Boston, Massachusetts.

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