Pancreatic calcifications associate with diverse aetiological risk factors in patients with chronic pancreatitis: A multicentre study of 1500 cases.


Journal

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
ISSN: 1424-3911
Titre abrégé: Pancreatology
Pays: Switzerland
ID NLM: 100966936

Informations de publication

Date de publication:
Oct 2019
Historique:
received: 03 06 2019
revised: 08 08 2019
accepted: 19 08 2019
pubmed: 30 8 2019
medline: 16 5 2020
entrez: 30 8 2019
Statut: ppublish

Résumé

Pancreatic calcifications is a common finding in patients with chronic pancreatitis (CP), but the underlying pathophysiology is incompletely understood. Past studies for risk factors of calcifications have generally been focused on single parameters or limited by small sample sizes. The aim of this study was to explore several patient and disease characteristics and their associations with pancreatic calcifications in a large cohort of CP patients with diverse aetiological risk factors. This was a multicentre, cross-sectional study including 1509 patients with CP. Patient and disease characteristics were compared for patients with calcifications (n = 912) vs. without calcifications (n = 597). Multivariable logistic regression was performed to assess the parameters independently associated with calcifications. The mean age of patients was 53.9 ± 14.5 years and 1006 (67%) were men. The prevalence of calcifications was 60.4% in the overall patient cohort, but highly variable between patients with different aetiological risk factors (range: 2-69%). On multivariate analysis, alcoholic aetiology (OR 1.76 [95% CI, 1.39-2.24]; p < 0.001) and smoking aetiology (OR 1.77 [95% CI, 1.39-2.26], p < 0.001) were positively associated with the presence of calcifications, while an autoimmune aetiology was negatively associated with calcifications (OR 0.15 [95% CI, 0.08-0.27], p < 0.001). Patients with pancreatic calcifications were more likely to have undergone pancreatic duct stenting (OR 1.59 [95%CI, 1.16-2.19], p = 0.004). The presence of pancreatic calcifications is associated with diverse aetiological risk factors in patients with CP. This observation attest to the understanding of CP as a complex disease and may have implications for disease classification.

Sections du résumé

BACKGROUND BACKGROUND
Pancreatic calcifications is a common finding in patients with chronic pancreatitis (CP), but the underlying pathophysiology is incompletely understood. Past studies for risk factors of calcifications have generally been focused on single parameters or limited by small sample sizes. The aim of this study was to explore several patient and disease characteristics and their associations with pancreatic calcifications in a large cohort of CP patients with diverse aetiological risk factors.
METHODS METHODS
This was a multicentre, cross-sectional study including 1509 patients with CP. Patient and disease characteristics were compared for patients with calcifications (n = 912) vs. without calcifications (n = 597). Multivariable logistic regression was performed to assess the parameters independently associated with calcifications.
RESULTS RESULTS
The mean age of patients was 53.9 ± 14.5 years and 1006 (67%) were men. The prevalence of calcifications was 60.4% in the overall patient cohort, but highly variable between patients with different aetiological risk factors (range: 2-69%). On multivariate analysis, alcoholic aetiology (OR 1.76 [95% CI, 1.39-2.24]; p < 0.001) and smoking aetiology (OR 1.77 [95% CI, 1.39-2.26], p < 0.001) were positively associated with the presence of calcifications, while an autoimmune aetiology was negatively associated with calcifications (OR 0.15 [95% CI, 0.08-0.27], p < 0.001). Patients with pancreatic calcifications were more likely to have undergone pancreatic duct stenting (OR 1.59 [95%CI, 1.16-2.19], p = 0.004).
CONCLUSION CONCLUSIONS
The presence of pancreatic calcifications is associated with diverse aetiological risk factors in patients with CP. This observation attest to the understanding of CP as a complex disease and may have implications for disease classification.

Identifiants

pubmed: 31462382
pii: S1424-3903(19)30690-8
doi: 10.1016/j.pan.2019.08.009
pii:
doi:

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

922-928

Informations de copyright

Copyright © 2019 IAP and EPC. Published by Elsevier B.V. All rights reserved.

Auteurs

Søren S Olesen (SS)

Centre for Pancreatic Diseases, Department of Gastroenterology and Hepatology, Aalborg University Hospital, Aalborg, Denmark; Clinical Institute, Aalborg University, Aalborg, Denmark. Electronic address: soso@rn.dk.

Maria Valeryevna Lisitskaya (MV)

Centre for Pancreatic Diseases, Department of Gastroenterology and Hepatology, Aalborg University Hospital, Aalborg, Denmark; Department of Radiology, Aalborg University Hospital, Aalborg, Denmark.

Asbjørn M Drewes (AM)

Centre for Pancreatic Diseases, Department of Gastroenterology and Hepatology, Aalborg University Hospital, Aalborg, Denmark; Clinical Institute, Aalborg University, Aalborg, Denmark.

Srdan Novovic (S)

Department of Gastroenterology, Hvidovre University Hospital, Copenhagen, Denmark.

Camilla Nøjgaard (C)

Department of Gastroenterology, Hvidovre University Hospital, Copenhagen, Denmark.

Evangelos Kalaitzakis (E)

Copenhagen University Hospital/Herlev, University of Copenhagen, Copenhagen, Denmark.

Nanna M Jensen (NM)

Abdominalcenter K, Bispebjerg Hospital, Copenhagen, Denmark.

Trond Engjom (T)

Department of Clinical Medicine, University of Bergen, Bergen, Norway; Haukeland University Hospital, Department of Medicine, Bergen, Norway.

Friedemann Erchinger (F)

Department of Clinical Medicine, University of Bergen, Bergen, Norway.

Anne Waage (A)

Department of Surgery, Oslo University Hospital, Oslo, Norway.

Truls Hauge (T)

Department of Gastroenterology, Oslo University Hospital, Oslo, Norway; Institute of Clinical Medicine, Faculty of Medicine, University of Oslo, Oslo, Norway.

Stephan L Haas (SL)

Centre for Digestive Diseases, Karolinska University Hospital, Stockholm, Sweden.

Miroslav Vujasinovic (M)

Centre for Digestive Diseases, Karolinska University Hospital, Stockholm, Sweden.

Björn Lindkvist (B)

Department of Medicine, Sahlgrenska University Hospital, Gothenburg, Sweden.

Kristina Zviniene (K)

Department of Radiology, Lithuanian University of Health Sciences, Kaunas, Lithuania.

Aldis Pukitis (A)

Centre of Gastroenterology, Hepatology and Nutrition, Pauls Stradins Clinical University Hospital, Riga, Latvia.

Imanta Ozola-Zālīte (I)

Centre of Gastroenterology, Hepatology and Nutrition, Pauls Stradins Clinical University Hospital, Riga, Latvia.

Alexey Okhlobystin (A)

Chair of Internal Diseases Propedeutics, I.M. Sechenov First Moscow State Medical University (Sechenov University), Moscow, Russia.

Mikael Parhiala (M)

Department of Gastroenterology and Alimentary Tract Surgery, Tampere University Hospital, Tampere, Finland; Cancer Center, Faculty of Medicine and Heath Technology, Tampere University, Tampere, Finland.

Johanna Laukkarinen (J)

Department of Gastroenterology and Alimentary Tract Surgery, Tampere University Hospital, Tampere, Finland; Cancer Center, Faculty of Medicine and Heath Technology, Tampere University, Tampere, Finland.

Jens B Frøkjær (JB)

Clinical Institute, Aalborg University, Aalborg, Denmark; Department of Radiology, Aalborg University Hospital, Aalborg, Denmark.

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