Natriuretic Peptide Response and Outcomes in Chronic Heart Failure With Reduced Ejection Fraction.


Journal

Journal of the American College of Cardiology
ISSN: 1558-3597
Titre abrégé: J Am Coll Cardiol
Pays: United States
ID NLM: 8301365

Informations de publication

Date de publication:
03 09 2019
Historique:
received: 26 01 2019
revised: 12 06 2019
accepted: 17 06 2019
entrez: 31 8 2019
pubmed: 31 8 2019
medline: 15 5 2020
Statut: ppublish

Résumé

The GUIDE-IT (GUIDing Evidence Based Therapy Using Biomarker Intensified Treatment in Heart Failure) trial demonstrated that a strategy to "guide" application of guideline-directed medical therapy (GDMT) by reducing amino-terminal pro-B-type natriuretic peptide (NT-proBNP) was not superior to GDMT alone. The purpose of this study was to examine the prognostic meaning of NT-proBNP changes following heart failure (HF) therapy intensification relative to the goal NT-proBNP value of 1,000 pg/ml explored in the GUIDE-IT trial. A total of 638 study participants were included who were alive and had available NT-proBNP results 90 days after randomization. Rates of subsequent cardiovascular (CV) death/HF hospitalization or all-cause mortality during follow-up and Kansas City Cardiomyopathy Questionnaire (KCCQ) overall scores were analyzed. A total of 198 (31.0%) subjects had an NT-proBNP ≤1,000 pg/ml at 90 days with no difference in achievement of NT-proBNP goal between the biomarker-guided and usual care arms. NT-proBNP ≤1,000 pg/ml by 90 days was associated with longer freedom from CV/HF hospitalization or all-cause mortality (p < 0.001 for both) and lower adjusted hazard of subsequent HF hospitalization/CV death (hazard ratio: 0.26; 95% confidence interval: 0.15 to 0.46; p < 0.001) and all-cause mortality (hazard ratio: 0.34; 95% confidence interval: 0.15 to 0.77; p = 0.009). Regardless of elevated baseline concentration, an NT-proBNP ≤1,000 pg/ml at 90 days was associated with better outcomes and significantly better KCCQ overall scores (p = 0.02). Patients with heart failure with reduced ejection fraction whose NT-proBNP levels decreased to ≤1,000 pg/ml during GDMT had better outcomes. These findings may help to understand the results of the GUIDE-IT trial. (Guiding Evidence Based Therapy Using Biomarker Intensified Treatment [GUIDE-IT]; NCT01685840).

Sections du résumé

BACKGROUND
The GUIDE-IT (GUIDing Evidence Based Therapy Using Biomarker Intensified Treatment in Heart Failure) trial demonstrated that a strategy to "guide" application of guideline-directed medical therapy (GDMT) by reducing amino-terminal pro-B-type natriuretic peptide (NT-proBNP) was not superior to GDMT alone.
OBJECTIVES
The purpose of this study was to examine the prognostic meaning of NT-proBNP changes following heart failure (HF) therapy intensification relative to the goal NT-proBNP value of 1,000 pg/ml explored in the GUIDE-IT trial.
METHODS
A total of 638 study participants were included who were alive and had available NT-proBNP results 90 days after randomization. Rates of subsequent cardiovascular (CV) death/HF hospitalization or all-cause mortality during follow-up and Kansas City Cardiomyopathy Questionnaire (KCCQ) overall scores were analyzed.
RESULTS
A total of 198 (31.0%) subjects had an NT-proBNP ≤1,000 pg/ml at 90 days with no difference in achievement of NT-proBNP goal between the biomarker-guided and usual care arms. NT-proBNP ≤1,000 pg/ml by 90 days was associated with longer freedom from CV/HF hospitalization or all-cause mortality (p < 0.001 for both) and lower adjusted hazard of subsequent HF hospitalization/CV death (hazard ratio: 0.26; 95% confidence interval: 0.15 to 0.46; p < 0.001) and all-cause mortality (hazard ratio: 0.34; 95% confidence interval: 0.15 to 0.77; p = 0.009). Regardless of elevated baseline concentration, an NT-proBNP ≤1,000 pg/ml at 90 days was associated with better outcomes and significantly better KCCQ overall scores (p = 0.02).
CONCLUSIONS
Patients with heart failure with reduced ejection fraction whose NT-proBNP levels decreased to ≤1,000 pg/ml during GDMT had better outcomes. These findings may help to understand the results of the GUIDE-IT trial. (Guiding Evidence Based Therapy Using Biomarker Intensified Treatment [GUIDE-IT]; NCT01685840).

Identifiants

pubmed: 31466618
pii: S0735-1097(19)35785-7
doi: 10.1016/j.jacc.2019.06.055
pmc: PMC6719723
mid: NIHMS1534169
pii:
doi:

Substances chimiques

Biomarkers 0
Peptide Fragments 0
pro-brain natriuretic peptide (1-76) 0
Natriuretic Peptide, Brain 114471-18-0

Banques de données

ClinicalTrials.gov
['NCT01685840']

Types de publication

Journal Article Randomized Controlled Trial Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't Research Support, U.S. Gov't, Non-P.H.S.

Langues

eng

Sous-ensembles de citation

IM

Pagination

1205-1217

Subventions

Organisme : NHLBI NIH HHS
ID : R01 HL105448
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL105451
Pays : United States
Organisme : NHLBI NIH HHS
ID : R01 HL105457
Pays : United States
Organisme : NCATS NIH HHS
ID : UL1 TR001863
Pays : United States

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2019 American College of Cardiology Foundation. Published by Elsevier Inc. All rights reserved.

Références

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pubmed: 19179406
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J Am Coll Cardiol. 2016 Dec 6;68(22):2425-2436
pubmed: 27908347

Auteurs

James L Januzzi (JL)

Cardiology Division, Massachusetts General Hospital, Baim Institute for Clinical Research, Boston, Massachusetts. Electronic address: JJanuzzi@partners.org.

Tariq Ahmad (T)

Section of Cardiovascular Medicine, Yale University Medical Center, New Haven, Connecticut.

Hillary Mulder (H)

Duke Clinical Research Institute, Durham, North Carolina.

Adrian Coles (A)

Duke Clinical Research Institute, Durham, North Carolina.

Kevin J Anstrom (KJ)

Duke Clinical Research Institute, Durham, North Carolina.

Kirkwood F Adams (KF)

University of North Carolina, Chapel Hill, North Carolina.

Justin A Ezekowitz (JA)

Canadian VIGOUR Centre, University of Alberta, Edmonton, Alberta, California.

Mona Fiuzat (M)

Duke Clinical Research Institute, Durham, North Carolina.

Nancy Houston-Miller (N)

Duke Clinical Research Institute, Durham, North Carolina.

Daniel B Mark (DB)

Duke Clinical Research Institute, Durham, North Carolina.

Ileana L Piña (IL)

Albert Einstein College of Medicine, Bronx, New York.

Gayle Passmore (G)

Duke Clinical Research Institute, Durham, North Carolina.

David J Whellan (DJ)

Thomas Jefferson University, Philadelphia, Pennsylvania.

Lawton S Cooper (LS)

Division of Cardiovascular Sciences, National Heart, Lung, and Blood Institute, Bethesda, Maryland.

Eric S Leifer (ES)

Division of Cardiovascular Sciences, National Heart, Lung, and Blood Institute, Bethesda, Maryland.

Patrice Desvigne-Nickens (P)

Division of Cardiovascular Sciences, National Heart, Lung, and Blood Institute, Bethesda, Maryland.

G Michael Felker (GM)

Duke Clinical Research Institute, Durham, North Carolina.

Christopher M O'Connor (CM)

Inova Heart and Vascular Center, Fairfax, Virginia.

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Classifications MeSH