Lack of Renal Tubular Glucocorticoid Receptor Decreases the Thiazide-Sensitive Na
Na+-K+-Cl– cotransporter
blood pressure
blood pressure dipping
glucocorticoid receptor
sodium and hydrogen exchanger 3
sodium transport
thiazide-sensitive Na+/Cl– cotransporter
Journal
Frontiers in physiology
ISSN: 1664-042X
Titre abrégé: Front Physiol
Pays: Switzerland
ID NLM: 101549006
Informations de publication
Date de publication:
2019
2019
Historique:
received:
18
03
2019
accepted:
18
07
2019
entrez:
3
9
2019
pubmed:
3
9
2019
medline:
3
9
2019
Statut:
epublish
Résumé
Chronic glucocorticoid infusion impairs NCC activity and induces a non-dipping profile in mice, suggesting that glucocorticoids are essential for daily blood pressure variations. In this paper, we studied mice lacking the renal tubular glucocorticoid receptor (GR) in adulthood (GR knockouts,
Identifiants
pubmed: 31474871
doi: 10.3389/fphys.2019.00989
pmc: PMC6702950
doi:
Types de publication
Journal Article
Langues
eng
Pagination
989Références
J Am Soc Nephrol. 1999 May;10(5):923-34
pubmed: 10232677
Nat Genet. 1999 Sep;23(1):99-103
pubmed: 10471508
Science. 2000 Sep 29;289(5488):2344-7
pubmed: 11009419
Kidney Int. 2001 Jan;59(1):197-209
pubmed: 11135072
J Biol Chem. 2001 Jul 27;276(30):28484-92
pubmed: 11350956
Nucleic Acids Res. 2002 Dec 1;30(23):e134
pubmed: 12466566
Development. 1992 Nov;116(3):611-23
pubmed: 1337742
J Biol Chem. 2004 Apr 30;279(18):18415-24
pubmed: 14973125
J Am Soc Nephrol. 2005 Apr;16(4):878-91
pubmed: 15743993
Nat Rev Neurosci. 2005 Jun;6(6):463-75
pubmed: 15891777
Pharmacol Rev. 2006 Dec;58(4):782-97
pubmed: 17132855
Development. 1990 Oct;110(2):643-51
pubmed: 1723950
J Am Soc Nephrol. 2008 Jan;19(1):92-101
pubmed: 18032797
Nat Med. 2008 Sep;14(9):979-84
pubmed: 18724376
Kidney Int. 2008 Dec;74(11):1403-9
pubmed: 18800028
Endocrinology. 2009 May;150(5):2202-10
pubmed: 19106216
Mol Cell Endocrinol. 2009 Apr 10;302(1):18-25
pubmed: 19146914
Hypertension. 2009 Oct;54(4):890-6
pubmed: 19635986
Biochem Biophys Res Commun. 2010 Apr 2;394(2):266-71
pubmed: 20188070
Genes Dev. 2010 Apr 15;24(8):741-7
pubmed: 20395363
Am J Physiol Renal Physiol. 2010 Dec;299(6):F1473-85
pubmed: 20861076
J Am Soc Nephrol. 2010 Nov;21(11):1942-51
pubmed: 20947633
Eur J Cell Biol. 2011 May;90(5):432-9
pubmed: 21354648
Physiology (Bethesda). 2011 Apr;26(2):115-23
pubmed: 21487030
Nat Med. 2011 May;17(5):573-80
pubmed: 21499270
Am J Physiol. 1990 Oct;259(4 Pt 2):F672-8
pubmed: 2221105
Endocrinology. 2012 May;153(5):2277-86
pubmed: 22434077
Kidney Int. 2013 May;83(5):811-24
pubmed: 23447069
J Am Soc Nephrol. 2014 Mar;25(3):511-22
pubmed: 24231659
J Physiol. 2014 Apr 15;592(8):1731-44
pubmed: 24535442
Neuroendocrinology. 2014;100(1):17-25
pubmed: 24903002
J Am Soc Nephrol. 2016 Aug;27(8):2436-45
pubmed: 26712527
Pflugers Arch. 2016 May;468(5):895-908
pubmed: 26762397
Hypertension. 2016 May;67(5):1029-37
pubmed: 26953322
J Am Soc Nephrol. 2016 Oct;27(10):2997-3004
pubmed: 27056296
Curr Hypertens Rep. 2017 Nov 14;19(12):100
pubmed: 29138984
Kidney Int. 2018 Apr;93(4):893-902
pubmed: 29310825
Clin Sci (Lond). 2018 Jan 19;132(2):173-183
pubmed: 29352074
Front Physiol. 2018 Jul 09;9:848
pubmed: 30038578
Genes Dev. 1995 Jul 1;9(13):1608-21
pubmed: 7628695
Am J Physiol. 1993 Apr;264(4 Pt 1):C875-84
pubmed: 8476020
Kidney Int. 1996 Jan;49(1):40-7
pubmed: 8770947
Proc Natl Acad Sci U S A. 1998 Aug 4;95(16):9424-9
pubmed: 9689096