Extensive serum biomarker analysis in patients with macrophage activation syndrome associated with systemic lupus erythematosus.


Journal

Clinical immunology (Orlando, Fla.)
ISSN: 1521-7035
Titre abrégé: Clin Immunol
Pays: United States
ID NLM: 100883537

Informations de publication

Date de publication:
11 2019
Historique:
received: 18 06 2019
revised: 27 08 2019
accepted: 30 08 2019
pubmed: 3 9 2019
medline: 20 5 2020
entrez: 3 9 2019
Statut: ppublish

Résumé

The present study employed an antibody array that simultaneously detects 174 cytokines to identify cytokines involved in the development of macrophage activation syndrome (MAS) associated with systemic lupus erythematosus (SLE) with a view to elucidating potential predictive markers. Eight SLE patients, including four with MAS, were analyzed. Levels of 31 cytokines were significantly elevated in the MAS phase compared with those in the active phase of SLE. Among these cytokines, the MAS/active phase ratios of CXCL9 and soluble tumor necrosis factor receptor II (sTNFR-II) were highest. Elevated serum CXCL9 and sTNFR-II levels during the MAS phase were confirmed by ELISA and were strongly correlated with other inflammatory markers, reflecting the disease activity of MAS associated with SLE. These results highlight the clinical significance of serum CXCL-9 and sTNFR-II levels, and indicate they may be useful biomarkers for the diagnosis of MAS associated with SLE.

Identifiants

pubmed: 31476438
pii: S1521-6616(19)30322-5
doi: 10.1016/j.clim.2019.108255
pii:
doi:

Substances chimiques

Biomarkers 0
CXCL9 protein, human 0
Chemokine CXCL9 0
Receptors, Tumor Necrosis Factor, Type II 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

108255

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Masaaki Usami (M)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.

Masaki Shimizu (M)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan. Electronic address: shimizum@staff.kanazawa-u.ac.jp.

Mao Mizuta (M)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.

Natsumi Inoue (N)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.

Hitoshi Irabu (H)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.

Naoto Sakumura (N)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.

Yasuo Nakagishi (Y)

Department of Pediatric Rheumatology, Hyogo Prefectural Kobe Children's Hospital, Kobe, Japan.

Akihiro Yachie (A)

Department of Pediatrics, School of Medicine, Institute of Medical, Pharmaceutical and Health Sciences, Kanazawa University, Kanazawa, Japan.

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Classifications MeSH