Interleukin 17 Receptor E (IL-17RE) and IL-17C Mediate the Recruitment of Neutrophils during Acute


Journal

Infection and immunity
ISSN: 1098-5522
Titre abrégé: Infect Immun
Pays: United States
ID NLM: 0246127

Informations de publication

Date de publication:
11 2019
Historique:
received: 26 04 2019
accepted: 26 08 2019
pubmed: 5 9 2019
medline: 14 1 2020
entrez: 5 9 2019
Statut: epublish

Résumé

Neutrophils contribute to lung injury in acute pneumococcal pneumonia. The interleukin 17 receptor E (IL-17RE) is the functional receptor for the epithelial-derived cytokine IL-17C, which is known to mediate innate immune functions. The aim of this study was to investigate the contribution of IL-17RE/IL-17C to pulmonary inflammation in a mouse model of acute

Identifiants

pubmed: 31481409
pii: IAI.00329-19
doi: 10.1128/IAI.00329-19
pmc: PMC6803337
pii:
doi:

Substances chimiques

Il17c protein, mouse 0
Interleukin-17 0
Receptors, Interleukin-17 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2019 American Society for Microbiology.

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Auteurs

Patrick Steck (P)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany.

Felix Ritzmann (F)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany.

Anja Honecker (A)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany.

Giovanna Vella (G)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany.

Christian Herr (C)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany.

Rosmarie Gaupp (R)

Institute of Medical Microbiology and Hygiene, Saarland University, Homburg, Germany.

Markus Bischoff (M)

Institute of Medical Microbiology and Hygiene, Saarland University, Homburg, Germany.

Timotheus Speer (T)

Department of Internal Medicine IV, Saarland University Medical Centre, Homburg, Germany.

Thomas Tschernig (T)

Institute of Anatomy and Cell Biology, Saarland University, Homburg, Germany.

Robert Bals (R)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany.

Christoph Beisswenger (C)

Department of Internal Medicine V-Pulmonology, Allergology and Critical Care Medicine, Saarland University, Homburg, Germany christoph.beisswenger@uks.eu.

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Classifications MeSH