FGF23, Hypophosphatemia, and Emerging Treatments.

BUROSUMAB FGF23 KLOTHO XLH X‐LINKED HYPOPHOSPHATEMIA

Journal

JBMR plus
ISSN: 2473-4039
Titre abrégé: JBMR Plus
Pays: England
ID NLM: 101707013

Informations de publication

Date de publication:
Aug 2019
Historique:
received: 29 12 2018
revised: 13 02 2019
accepted: 26 02 2019
entrez: 6 9 2019
pubmed: 6 9 2019
medline: 6 9 2019
Statut: epublish

Résumé

FGF23 is an important hormonal regulator of phosphate homeostasis. Together with its co-receptor Klotho, it modulates phosphate reabsorption and both 1α-hydroxylation and 24-hydroxylation in the renal proximal tubules. The most common FGF23-mediated hypophosphatemia is X-linked hypophosphatemia (XLH), caused by mutations in the

Identifiants

pubmed: 31485552
doi: 10.1002/jbm4.10190
pii: JBM410190
pmc: PMC6715782
doi:

Types de publication

Journal Article Review

Langues

eng

Pagination

e10190

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Auteurs

Erik A Imel (EA)

Division of Endocrinology Indiana University School of Medicine, Indianapolis, IN USA.

Andrew Biggin (A)

The University of Sydney Children's Hospital Westmead Clinical School, University of Sydney Sydney Australia.
Department of Endocrinology The Children's Hospital at Westmead Westmead Australia.

Aaron Schindeler (A)

The University of Sydney Children's Hospital Westmead Clinical School, University of Sydney Sydney Australia.
Orthopaedic Research Unit, The Children's Hospital at Westmead Westmead Australia.

Craig F Munns (CF)

The University of Sydney Children's Hospital Westmead Clinical School, University of Sydney Sydney Australia.
Department of Endocrinology The Children's Hospital at Westmead Westmead Australia.

Classifications MeSH