Intracoronary pressure increase due to contrast injection for optical coherence tomography imaging.


Journal

Journal of cardiology
ISSN: 1876-4738
Titre abrégé: J Cardiol
Pays: Netherlands
ID NLM: 8804703

Informations de publication

Date de publication:
03 2020
Historique:
received: 02 05 2019
revised: 31 07 2019
accepted: 06 08 2019
pubmed: 11 9 2019
medline: 23 12 2020
entrez: 11 9 2019
Statut: ppublish

Résumé

Optical coherence tomography (OCT) requires intracoronary injection of contrast media to remove blood from the field of view during image acquisition. Contrast injection may cause a temporal increase in intracoronary pressure. The aim of this study was to compare the intracoronary pressure during contrast injection between OCT and coronary angiography. We measured intracoronary pressure by using a pressure guidewire during contrast injection for OCT and angiography in 30 coronary arteries (mean fractional flow reserve = 0.90 ± 0.03). Contrast media was injected into coronary artery through the guiding catheter by using a mechanical injector pump. Intracoronary pressure before contrast injection was similar between OCT and angiography (systolic pressure: 123 ± 18 mmHg vs. 122 ± 19 mmHg, p = 0.863). Intracoronary pressure was increased due to contrast injection in both OCT (systolic pressure: 123 ± 18 mmHg to 132 ± 18 mmHg, p < 0.001) and angiography (systolic pressure: 122 ± 19 mmHg to 128 ± 19 mmHg, p < 0.001). The increase in intracoronary pressure was slightly greater in OCT compared with angiography (absolute increase of systolic pressure: 9 ± 2 mmHg vs. 6 ± 1 mmHg, p < 0.001; and relative increase of systolic pressure: 8 ± 2% vs. 5 ± 1%, p < 0.001). Intracoronary pressure during contrast injection was not significantly different between OCT and angiography (systolic pressure: 132 ± 18 mmHg vs. 128 ± 19 mmHg, p = 0.831). Contrast injection for OCT induced significant but small increase in intracoronary pressure compared with that for angiography.

Sections du résumé

BACKGROUND
Optical coherence tomography (OCT) requires intracoronary injection of contrast media to remove blood from the field of view during image acquisition. Contrast injection may cause a temporal increase in intracoronary pressure. The aim of this study was to compare the intracoronary pressure during contrast injection between OCT and coronary angiography.
METHODS
We measured intracoronary pressure by using a pressure guidewire during contrast injection for OCT and angiography in 30 coronary arteries (mean fractional flow reserve = 0.90 ± 0.03). Contrast media was injected into coronary artery through the guiding catheter by using a mechanical injector pump.
RESULTS
Intracoronary pressure before contrast injection was similar between OCT and angiography (systolic pressure: 123 ± 18 mmHg vs. 122 ± 19 mmHg, p = 0.863). Intracoronary pressure was increased due to contrast injection in both OCT (systolic pressure: 123 ± 18 mmHg to 132 ± 18 mmHg, p < 0.001) and angiography (systolic pressure: 122 ± 19 mmHg to 128 ± 19 mmHg, p < 0.001). The increase in intracoronary pressure was slightly greater in OCT compared with angiography (absolute increase of systolic pressure: 9 ± 2 mmHg vs. 6 ± 1 mmHg, p < 0.001; and relative increase of systolic pressure: 8 ± 2% vs. 5 ± 1%, p < 0.001). Intracoronary pressure during contrast injection was not significantly different between OCT and angiography (systolic pressure: 132 ± 18 mmHg vs. 128 ± 19 mmHg, p = 0.831).
CONCLUSIONS
Contrast injection for OCT induced significant but small increase in intracoronary pressure compared with that for angiography.

Identifiants

pubmed: 31500960
pii: S0914-5087(19)30267-9
doi: 10.1016/j.jjcc.2019.08.008
pii:
doi:

Substances chimiques

Contrast Media 0

Types de publication

Clinical Trial Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

296-301

Informations de copyright

Copyright © 2019. Published by Elsevier Ltd.

Auteurs

Kunihiro Shimamura (K)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Takashi Kubo (T)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan. Electronic address: takakubo@wakayama-med.ac.jp.

Yasushi Ino (Y)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Yasutsugu Shiono (Y)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Yoshiki Matsuo (Y)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Hironori Kitabata (H)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Kosei Terada (K)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Hiroki Emori (H)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Yosuke Katayama (Y)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Amir Kh M Khalifa (AKM)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Masahiro Takahata (M)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Teruaki Wada (T)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Daisuke Higashioka (D)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Manabu Kashiwagi (M)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Akio Kuroi (A)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Atsushi Tanaka (A)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Takeshi Hozumi (T)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

Takashi Akasaka (T)

Department of Cardiovascular Medicine, Wakayama Medical University, Wakayama, Japan.

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