Quantitative assessment of betainized compounds and associations with dietary and metabolic biomarkers in the randomized study of the healthy Nordic diet (SYSDIET).


Journal

The American journal of clinical nutrition
ISSN: 1938-3207
Titre abrégé: Am J Clin Nutr
Pays: United States
ID NLM: 0376027

Informations de publication

Date de publication:
01 11 2019
Historique:
received: 20 03 2019
accepted: 10 07 2019
pubmed: 11 9 2019
medline: 3 4 2020
entrez: 11 9 2019
Statut: ppublish

Résumé

Recently, a group of betainized compounds have been suggested to play a role in health effects in relation to a whole-grain-rich diet. The aims of this study were to develop a quantitative mass spectrometric method for selected betainized compounds in human plasma, and to investigate their association with nutrient intake and measures of metabolic health in participants of the SYSDIET study. The SYSDIET study was a controlled randomized intervention including individuals with metabolic syndrome, where the healthy Nordic diet (HND) group increased intakes of whole grains, canola oil, berries, and fish, whereas the control diet (CD) group consumed low-fiber cereal products, milk fat, and restricted amounts of fish and berries. A quantitative LC combined with triple quadrupole MS method for betainized compounds was developed and applied to fasting plasma samples from baseline (week 0) and the end of the intervention (week 18 or 24). Concentrations of betainized compounds were correlated with intakes of selected nutrients and fiber and measures of metabolic health. Pipecolic acid betaine (PAB) concentrations were significantly higher in the HND group than in the CD group (P = 0.00032) at the end of the intervention and correlated directly (P < 0.0001) with intakes of dietary fiber (r = 0.376) and a biomarker related to whole-grain rye intake, namely the ratio of alkylresorcinol C17:0 to C21:0 (r = 0.442). PAB was associated inversely with fasting plasma insulin consistently at the beginning and at the end of the intervention (P < 0.001, r = -0.300; P < 0.01, r = -0.250, respectively), as well as IL-1 receptor antagonist (P < 0.01, r = -0.232 at the beginning; P < 0.01, r = -0.236 at the end) and serum LDL/HDL cholesterol (P < 0.01, r = -0.239 at the beginning; P < 0.01, r = -0.241 at the end). Among adults with the metabolic syndrome, PAB plasma concentrations were associated with fasting insulin, inflammation, and lipids and were significantly increased with adoption of the HND. Further studies are needed to clarify the biological functions of betainized compounds. This trial was registered at clinicaltrials.gov as NCT00992641.

Sections du résumé

BACKGROUND
Recently, a group of betainized compounds have been suggested to play a role in health effects in relation to a whole-grain-rich diet.
OBJECTIVES
The aims of this study were to develop a quantitative mass spectrometric method for selected betainized compounds in human plasma, and to investigate their association with nutrient intake and measures of metabolic health in participants of the SYSDIET study.
METHODS
The SYSDIET study was a controlled randomized intervention including individuals with metabolic syndrome, where the healthy Nordic diet (HND) group increased intakes of whole grains, canola oil, berries, and fish, whereas the control diet (CD) group consumed low-fiber cereal products, milk fat, and restricted amounts of fish and berries. A quantitative LC combined with triple quadrupole MS method for betainized compounds was developed and applied to fasting plasma samples from baseline (week 0) and the end of the intervention (week 18 or 24). Concentrations of betainized compounds were correlated with intakes of selected nutrients and fiber and measures of metabolic health.
RESULTS
Pipecolic acid betaine (PAB) concentrations were significantly higher in the HND group than in the CD group (P = 0.00032) at the end of the intervention and correlated directly (P < 0.0001) with intakes of dietary fiber (r = 0.376) and a biomarker related to whole-grain rye intake, namely the ratio of alkylresorcinol C17:0 to C21:0 (r = 0.442). PAB was associated inversely with fasting plasma insulin consistently at the beginning and at the end of the intervention (P < 0.001, r = -0.300; P < 0.01, r = -0.250, respectively), as well as IL-1 receptor antagonist (P < 0.01, r = -0.232 at the beginning; P < 0.01, r = -0.236 at the end) and serum LDL/HDL cholesterol (P < 0.01, r = -0.239 at the beginning; P < 0.01, r = -0.241 at the end).
CONCLUSIONS
Among adults with the metabolic syndrome, PAB plasma concentrations were associated with fasting insulin, inflammation, and lipids and were significantly increased with adoption of the HND. Further studies are needed to clarify the biological functions of betainized compounds. This trial was registered at clinicaltrials.gov as NCT00992641.

Identifiants

pubmed: 31504116
pii: S0002-9165(22)01295-3
doi: 10.1093/ajcn/nqz179
doi:

Substances chimiques

Biomarkers 0
Dietary Fiber 0
Pipecolic Acids 0
Betaine 3SCV180C9W
Proline 9DLQ4CIU6V
pipecolic acid H254GW7PVV
stachydrine S1L688345C

Banques de données

ClinicalTrials.gov
['NCT00992641']

Types de publication

Journal Article Randomized Controlled Trial Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

1108-1118

Informations de copyright

Copyright © American Society for Nutrition 2019.

Auteurs

Marjo Tuomainen (M)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.

Olli Kärkkäinen (O)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.
School of Pharmacy, University of Eastern Finland, Kuopio, Finland.

Jukka Leppänen (J)

School of Pharmacy, University of Eastern Finland, Kuopio, Finland.

Seppo Auriola (S)

School of Pharmacy, University of Eastern Finland, Kuopio, Finland.
LC-MS Metabolomics Center, Biocenter Kuopio, Kuopio, Finland.

Marko Lehtonen (M)

School of Pharmacy, University of Eastern Finland, Kuopio, Finland.
LC-MS Metabolomics Center, Biocenter Kuopio, Kuopio, Finland.

Markku J Savolainen (MJ)

Medical Research Center, Department of Internal Medicine, University of Oulu and Oulu University Hospital, Oulu, Finland.

Kjeld Hermansen (K)

Department of Endocrinology and Internal Medicine, Aarhus University Hospital, Aarhus, Denmark.

Ulf Risérus (U)

Department of Public Health and Caring Sciences, Clinical Nutrition and Metabolism, Uppsala University, Uppsala, Sweden.

Björn Åkesson (B)

Biomedical Nutrition, Pure and Applied Biochemistry, Lund University, Lund, Sweden.
Department of Clinical Nutrition, Skåne University Hospital, Lund, Sweden.

Inga Thorsdottir (I)

Unit for Nutrition Research, University of Iceland and Landspitali-The National University Hospital of Iceland, Reykjavik, Iceland.

Marjukka Kolehmainen (M)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.

Matti Uusitupa (M)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.

Kaisa Poutanen (K)

VTT Technical Research Centre of Finland, Espoo, Finland.

Ursula Schwab (U)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.
Department of Medicine, Endocrinology and Clinical Nutrition, Kuopio University Hospital, Kuopio, Finland.

Kati Hanhineva (K)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.

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