Clinical Outcomes in Persons Coinfected With Human Immunodeficiency Virus and Hepatitis C Virus: Impact of Hepatitis C Virus Treatment.


Journal

Clinical infectious diseases : an official publication of the Infectious Diseases Society of America
ISSN: 1537-6591
Titre abrégé: Clin Infect Dis
Pays: United States
ID NLM: 9203213

Informations de publication

Date de publication:
06 05 2020
Historique:
received: 14 03 2019
accepted: 28 06 2019
pubmed: 11 9 2019
medline: 7 1 2021
entrez: 11 9 2019
Statut: ppublish

Résumé

A hepatitis C (HCV) cure is associated with changes in lipids and inflammatory biomarkers, but its impact on clinical endpoints among treated human immunodeficiency virus (HIV)/HCV coinfected persons is unclear. People living with HIV from EuroSIDA with a known HCV status after January 2001 were classified into strata based on time-updated HCV RNA measurements and HCV treatment, as either HCV antibody-negative; spontaneously resolved HCV; chronic, untreated HCV; cured HCV (HCV RNA-negative); or HCV treatment failures (HCV RNA-positive). Poisson regression was used to compare incidence rates between HCV groups for end-stage liver disease (ESLD; including hepatocellular carcinoma [HCC]), non-acquired immunodeficiency virus defining malignancy (NADM; excluding HCC), and cardiovascular disease (CVD). There were 16 618 persons included (median follow-up 8.3 years, interquartile range 3.1-13.7). There were 887 CVD, 902 NADM, and 436 ESLD events; crude incidence rates/1000 person-years follow-up were 6.4 (95% confidence interval [CI] 6.0-6.9) for CVD, 6.5 (95% CI 6.1-6.9) for NADM, and 3.1 (95% CI 2.8-3.4) for ESLD. After adjustment, there were no differences in incidence rates of NADM or CVD across the 5 groups. HCV-negative individuals (adjusted incidence rate ratio [aIRR] 0.22, 95% CI 0.14-0.34) and those with spontaneous clearance (aIRR 0.61, 95% CI 0.36-1.02) had reduced rates of ESLD compared to cured individuals. Persons with chronic, untreated HCV infections (aIRR 1.47, 95% CI 1.02-2.13) or treatment failure (aIRR 1.80, 95% CI 1.22-2.66) had significantly raised rates of ESLD, compared to those who were cured. Incidences of NADM or CVD were independent of HCV group, whereas those cured had substantially lower incidences of ESLD, underlining the importance of successful HCV treatment for reducing ESLD.

Sections du résumé

BACKGROUND
A hepatitis C (HCV) cure is associated with changes in lipids and inflammatory biomarkers, but its impact on clinical endpoints among treated human immunodeficiency virus (HIV)/HCV coinfected persons is unclear.
METHODS
People living with HIV from EuroSIDA with a known HCV status after January 2001 were classified into strata based on time-updated HCV RNA measurements and HCV treatment, as either HCV antibody-negative; spontaneously resolved HCV; chronic, untreated HCV; cured HCV (HCV RNA-negative); or HCV treatment failures (HCV RNA-positive). Poisson regression was used to compare incidence rates between HCV groups for end-stage liver disease (ESLD; including hepatocellular carcinoma [HCC]), non-acquired immunodeficiency virus defining malignancy (NADM; excluding HCC), and cardiovascular disease (CVD).
RESULTS
There were 16 618 persons included (median follow-up 8.3 years, interquartile range 3.1-13.7). There were 887 CVD, 902 NADM, and 436 ESLD events; crude incidence rates/1000 person-years follow-up were 6.4 (95% confidence interval [CI] 6.0-6.9) for CVD, 6.5 (95% CI 6.1-6.9) for NADM, and 3.1 (95% CI 2.8-3.4) for ESLD. After adjustment, there were no differences in incidence rates of NADM or CVD across the 5 groups. HCV-negative individuals (adjusted incidence rate ratio [aIRR] 0.22, 95% CI 0.14-0.34) and those with spontaneous clearance (aIRR 0.61, 95% CI 0.36-1.02) had reduced rates of ESLD compared to cured individuals. Persons with chronic, untreated HCV infections (aIRR 1.47, 95% CI 1.02-2.13) or treatment failure (aIRR 1.80, 95% CI 1.22-2.66) had significantly raised rates of ESLD, compared to those who were cured.
CONCLUSIONS
Incidences of NADM or CVD were independent of HCV group, whereas those cured had substantially lower incidences of ESLD, underlining the importance of successful HCV treatment for reducing ESLD.

Identifiants

pubmed: 31504296
pii: 5527213
doi: 10.1093/cid/ciz601
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

2131-2140

Investigateurs

M Losso (M)
B Schmied (B)
I Karpov (I)
N Clumeck (N)
V Hadziosmanovic (V)
J Begovac (J)
L Machala (L)
K Zilmer (K)
I Aho (I)
J-P Viard (JP)
J Rockstroh (J)
N Chkhartishvili (N)
H Sambatakou (H)
J Szlávik (J)
M Gottfredsson (M)
F Mulcahy (F)
L Tau (L)
A D'Arminio Monforte (A)
B Rozentale (B)
V Uzdaviniene (V)
T Staub (T)
P Reiss (P)
D H Reikvam (DH)
B Knysz (B)
L Caldeira (L)
R Radoi (R)
A Panteleev (A)
G Dragovic (G)
J Tomazic (J)
J M Miró (JM)
K Falconer (K)
A Scherrer (A)
B Gazzard (B)

Commentaires et corrections

Type : CommentIn

Informations de copyright

© The Author(s) 2019. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.

Auteurs

Amanda Mocroft (A)

Centre for Clinical Research, Epidemiology, Modelling and Evaluation, Institute for Global Health, University College London, United Kingdom.

Jens Lundgren (J)

Centre of Excellence for Health, Immunity and Infections, Rigshospitalet, Copenhagen, and Department of Infectious Diseases, Copenhagen.

Jan Gerstoft (J)

Department of Infectious Diseases, Rigshospitalet, Copenhagen.

Line D Rasmussen (LD)

Department of Infectious Diseases, Odense University Hospital, Denmark.

Sanjay Bhagani (S)

Department of Infectious Diseases, Royal Free Hospital, London, United Kingdom.

Inka Aho (I)

Division of Infectious Diseases, Helsinki University Hospital, Finland.

Christian Pradier (C)

Department of Public Health, Centre Hospitalier Universitaire de Nice, France.

Johannes R Bogner (JR)

Division of Infectious Diseases, Medizinische Klinik und Poliklinik IV, Ludwig Maximilians University of Munich, Germany.

Christina Mussini (C)

Clinic of Infectious Diseases, University of Modena and Reggio Emilia, Milan, Italy.

Caterina Uberti Foppa (C)

Vita-Salute San Raffaele University, San Raffaele Scientific Institue, Clinic of Infectious Diseases, Milan, Italy.

Fernando Maltez (F)

Hospital de Curry Cabral, Serviço de Doenças Infecciosas, Lisbon, Portugal.

Montse Laguno (M)

Infectious Diseases Service, Hospital Clinic-L'Institut d'Investigacions Biomèdiques August Pi i Sunyer, University of Barcelona, Spain.

Gilles Wandeler (G)

Department of Infectious Diseases, Bern University Hospital, University of Bern, Switzerland.

Karolin Falconer (K)

Infectious Diseases Department, Karolinska University Hospital, Stockholm, Sweden.

Tatyana Trofimova (T)

Novgorod Centre for Acquired Immunodeficiency Syndrome Prevention and Control, Novgorod the Great, Russia.

Elena Borodulina (E)

Samara State Medical University, Russia.

Djordje Jevtovic (D)

Belgrade University School of Medicine, Infectious & Tropical Diseases Hospital, Serbia.

Elzbieta Bakowska (E)

Wojewodzki Szpital Zakazny, Warsaw, Poland.

Kerstin Kase (K)

Centre of Infectious Diseases, West-Tallin Central Hospital, Tallin, Estonia.

Galina Kyselyova (G)

Crimean Republican Acquired Immunodeficiency Syndrome Centre, Simferopol.

Richard Haubrich (R)

Gilead Sciences Inc., Foster City, California.

Jürgen K Rockstroh (JK)

Department of Medicine, University Hospital Bonn, Germany.

Lars Peters (L)

Centre of Excellence for Health, Immunity and Infections, Rigshospitalet, Copenhagen, and Department of Infectious Diseases, Copenhagen.

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