Being overweight in childhood, puberty, or early adulthood: Changing asthma risk in the next generation?


Journal

The Journal of allergy and clinical immunology
ISSN: 1097-6825
Titre abrégé: J Allergy Clin Immunol
Pays: United States
ID NLM: 1275002

Informations de publication

Date de publication:
03 2020
Historique:
received: 31 01 2019
revised: 20 08 2019
accepted: 22 08 2019
pubmed: 11 9 2019
medline: 1 12 2020
entrez: 11 9 2019
Statut: ppublish

Résumé

Overweight status and asthma have increased during the last decades. Being overweight is a known risk factor for asthma, but it is not known whether it might also increase asthma risk in the next generation. We aimed to examine whether parents being overweight in childhood, adolescence, or adulthood is associated with asthma in their offspring. We included 6347 adult offspring (age, 18-52 years) investigated in the Respiratory Health in Northern Europe, Spain and Australia (RHINESSA) multigeneration study of 2044 fathers and 2549 mothers (age, 37-66 years) investigated in the European Community Respiratory Health Survey (ECRHS) study. Associations of parental overweight status at age 8 years, puberty, and age 30 years with offspring's childhood overweight status (potential mediator) and offspring's asthma with or without nasal allergies (outcomes) was analyzed by using 2-level logistic regression and 2-level multinomial logistic regression, respectively. Counterfactual-based mediation analysis was performed to establish whether observed associations were direct or indirect effects mediated through the offspring's own overweight status. We found statistically significant associations between both fathers' and mothers' childhood overweight status and offspring's childhood overweight status (odds ratio, 2.23 [95% CI, 1.45-3.42] and 2.45 [95% CI, 1.86-3.22], respectively). We also found a statistically significant effect of fathers' onset of being overweight in puberty on offspring's asthma without nasal allergies (relative risk ratio, 2.31 [95% CI, 1.23-4.33]). This effect was direct and not mediated through the offspring's own overweight status. No effect on offspring's asthma with nasal allergies was found. Our findings suggest that metabolic factors long before conception can increase asthma risk and that male puberty is a time window of particular importance for offspring's health.

Sections du résumé

BACKGROUND
Overweight status and asthma have increased during the last decades. Being overweight is a known risk factor for asthma, but it is not known whether it might also increase asthma risk in the next generation.
OBJECTIVE
We aimed to examine whether parents being overweight in childhood, adolescence, or adulthood is associated with asthma in their offspring.
METHODS
We included 6347 adult offspring (age, 18-52 years) investigated in the Respiratory Health in Northern Europe, Spain and Australia (RHINESSA) multigeneration study of 2044 fathers and 2549 mothers (age, 37-66 years) investigated in the European Community Respiratory Health Survey (ECRHS) study. Associations of parental overweight status at age 8 years, puberty, and age 30 years with offspring's childhood overweight status (potential mediator) and offspring's asthma with or without nasal allergies (outcomes) was analyzed by using 2-level logistic regression and 2-level multinomial logistic regression, respectively. Counterfactual-based mediation analysis was performed to establish whether observed associations were direct or indirect effects mediated through the offspring's own overweight status.
RESULTS
We found statistically significant associations between both fathers' and mothers' childhood overweight status and offspring's childhood overweight status (odds ratio, 2.23 [95% CI, 1.45-3.42] and 2.45 [95% CI, 1.86-3.22], respectively). We also found a statistically significant effect of fathers' onset of being overweight in puberty on offspring's asthma without nasal allergies (relative risk ratio, 2.31 [95% CI, 1.23-4.33]). This effect was direct and not mediated through the offspring's own overweight status. No effect on offspring's asthma with nasal allergies was found.
CONCLUSION
Our findings suggest that metabolic factors long before conception can increase asthma risk and that male puberty is a time window of particular importance for offspring's health.

Identifiants

pubmed: 31505189
pii: S0091-6749(19)31172-8
doi: 10.1016/j.jaci.2019.08.030
pii:
doi:

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

791-799.e4

Informations de copyright

Copyright © 2019 The Authors. Published by Elsevier Inc. All rights reserved.

Auteurs

Ane Johannessen (A)

Department of Global Public Health and Primary Care, University of Bergen, Bergen, Norway; Department of Occupational Medicine, Haukeland University Hospital, Bergen, Norway.

Marianne Lønnebotn (M)

Department of Global Public Health and Primary Care, University of Bergen, Bergen, Norway; Department of Occupational Medicine, Haukeland University Hospital, Bergen, Norway. Electronic address: marianne.lonnebotn@uib.no.

Lucia Calciano (L)

Unit of Epidemiology and Medical Statistics, Department of Diagnostics and Public Health, University of Verona, Verona, Italy.

Bryndis Benediktsdóttir (B)

Faculty of Medicine, University of Iceland, Reykjavik, Iceland; Department of Sleep, Landspitali University Hospital, Reykjavik, Iceland.

Randi Jacobsen Bertelsen (RJ)

Department of Clinical Science, University of Bergen, Bergen, Norway.

Lennart Bråbäck (L)

Occupational and Environmental Medicine, Department of Public Health and Clinical Medicine, Umeå, Sweden.

Shyamali Dharmage (S)

Allergy and Lung Health Unit, School of Population and Global Health, University of Melbourne, Melbourne, Australia.

Karl A Franklin (KA)

Department of Surgical and Perioperative Sciences, Surgery, Umeå University, Umeå, Sweden.

Thorarinn Gislason (T)

Department of Sleep, Landspitali University Hospital, Reykjavik, Iceland.

Mathias Holm (M)

Department of Occupational and Environmental Medicine, Sahlgrenska University Hospital, Gothenburg, Sweden.

Christer Janson (C)

Department of Medical Sciences, Respiratory, Allergy & Sleep Research, Uppsala University, Uppsala, Sweden.

Deborah Jarvis (D)

Faculty of Medicine, National Heart and Lung Institute, Imperial College, London, United Kingdom.

Rain Jõgi (R)

Lung Clinic, Tartu University Hospital, Tartu, Estonia.

Jeong-Lim Kim (JL)

Occupational and Environmental Medicine, Department of Public Health and Community Medicine, Sahlgrenska Academy, University of Gothenburg, Gothenburg, Sweden.

Jorunn Kirkeleit (J)

Department of Global Public Health and Primary Care, University of Bergen, Bergen, Norway.

Caroline Lodge (C)

Department of Population Health, University of Melbourne, Melbourne, Australia.

Andrei Malinovschi (A)

Department of Medical Sciences, Uppsala University, Uppsala, Sweden.

Jesus Martinez-Moratalla (J)

Servicio de Neurología del Complejo Hospitalario Universitario de Albacete (CHUA), Servicio de Salud de Castilla-La Mancha (SESCAM), Albacete, Spain.

Roy Miodini Nilsen (RM)

Department of Nursing, Bergen University College, Bergen, Norway.

Antonio Pereira-Vega (A)

Pneumology Section, Hospital Juan Ramón Jiménez, Huelva, Spain.

Francisco Gómez Real (FG)

Department of Clinical Science, University of Bergen, Bergen, Norway; Department of Obstetrics and Gynecology, Haukeland University Hospital, Bergen, Norway.

Vivi Schlünssen (V)

Department of Public Health, Aarhus University, Aarhus, Denmark.

Simone Accordini (S)

Unit of Epidemiology and Medical Statistics, Department of Diagnostics and Public Health, University of Verona, Verona, Italy.

Cecilie Svanes (C)

Department of Occupational Medicine, Haukeland University Hospital, Bergen, Norway; Centre for International Health, Department of Global Public Health and Primary Care, University of Bergen, Bergen, Norway.

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