Brainstem Diffuse Axonal Injury and Consciousness.

Ascending reticular activating system Consciousness Diffuse axonal injury Magnetic resonance imaging Traumatic brain injury

Journal

Journal of clinical imaging science
ISSN: 2156-7514
Titre abrégé: J Clin Imaging Sci
Pays: United States
ID NLM: 101564708

Informations de publication

Date de publication:
2019
Historique:
received: 29 03 2019
accepted: 04 06 2019
entrez: 12 9 2019
pubmed: 12 9 2019
medline: 12 9 2019
Statut: epublish

Résumé

Severe traumatic brain injuries (TBI), commonly due to motor vehicle accidents may cause death and long-term disability especially when the acceleration-deceleration force on the brain is massive. This may cause shearing of the axonal connections within the cerebral cortex and brainstem in a process referred to as diffuse axonal injury (DAI). Extensive DAI has been postulated to be a poor prognostic indicator for neurological recovery. In our institution, several patients with Grade 3 DAI were observed to recover and achieve neurological outcomes greater than expected given the presence of brainstem injury. MRI studies from 100 patients admitted to a large tertiary trauma center for TBI were retrospectively analyzed by two fellowship-trained neuroradiologists. The size of DAI lesions, location of injury within the brainstem, and the number of discrete DAI lesions were measured and recorded. Glasgow Coma Scale (GCS) on arrival and at discharge was noted, as well as the presence of other neurological injuries. Of 20 patients initially noted to have DAI with lesions of the brainstem, eight of them were discharged with Glasgow Coma Scale (GCS) of 14-15. The 12 patients discharged with reduced consciousness (average GC 7.1) demonstrated a greater number of larger lesions, with a predilection for the dorsal pons. These results suggest that large, numerous pontine lesions may indicate worse neurological outcomes in patients with these findings.

Sections du résumé

BACKGROUND BACKGROUND
Severe traumatic brain injuries (TBI), commonly due to motor vehicle accidents may cause death and long-term disability especially when the acceleration-deceleration force on the brain is massive. This may cause shearing of the axonal connections within the cerebral cortex and brainstem in a process referred to as diffuse axonal injury (DAI). Extensive DAI has been postulated to be a poor prognostic indicator for neurological recovery. In our institution, several patients with Grade 3 DAI were observed to recover and achieve neurological outcomes greater than expected given the presence of brainstem injury.
METHODS METHODS
MRI studies from 100 patients admitted to a large tertiary trauma center for TBI were retrospectively analyzed by two fellowship-trained neuroradiologists. The size of DAI lesions, location of injury within the brainstem, and the number of discrete DAI lesions were measured and recorded. Glasgow Coma Scale (GCS) on arrival and at discharge was noted, as well as the presence of other neurological injuries.
RESULTS RESULTS
Of 20 patients initially noted to have DAI with lesions of the brainstem, eight of them were discharged with Glasgow Coma Scale (GCS) of 14-15. The 12 patients discharged with reduced consciousness (average GC 7.1) demonstrated a greater number of larger lesions, with a predilection for the dorsal pons.
CONCLUSION CONCLUSIONS
These results suggest that large, numerous pontine lesions may indicate worse neurological outcomes in patients with these findings.

Identifiants

pubmed: 31508267
doi: 10.25259/JCIS-11-2019
pii: JCIS-9-32
pmc: PMC6712553
doi:

Types de publication

Journal Article

Langues

eng

Pagination

32

Déclaration de conflit d'intérêts

There are no conflicts of interest.

Références

AJNR Am J Neuroradiol. 2001 Jan;22(1):143-51
pubmed: 11158900
Brain. 2003 Jul;126(Pt 7):1524-36
pubmed: 12805123
J Neurotrauma. 2005 Sep;22(9):947-54
pubmed: 16156710
Crit Care. 2007;11(5):230
pubmed: 17980050
J Korean Neurosurg Soc. 2009 Sep;46(3):205-9
pubmed: 19844619
J Neurosurg. 2010 Sep;113(3):556-63
pubmed: 19852541
J Trauma. 2011 Oct;71(4):838-46
pubmed: 21460740
AJNR Am J Neuroradiol. 2012 Nov;33(10):1925-31
pubmed: 22576887
Front Hum Neurosci. 2013 Jul 25;7:416
pubmed: 23898258
Scand J Trauma Resusc Emerg Med. 2015 Jun 05;23:42
pubmed: 26043908
Neural Regen Res. 2015 Nov;10(11):1879-81
pubmed: 26807130
J Neurotrauma. 2017 Jan 15;34(2):341-352
pubmed: 27356857
Histopathology. 1989 Jul;15(1):49-59
pubmed: 2767623
Neurocrit Care. 2017 Oct;27(2):199-207
pubmed: 28477152
Crit Care Med. 2017 Dec;45(12):e1280-e1288
pubmed: 29028764
J Trauma Acute Care Surg. 2018 Mar;84(3):473-482
pubmed: 29140952
Brain Inj. 2018;32(4):395-402
pubmed: 29381396
Asian J Neurosurg. 2018 Apr-Jun;13(2):348-356
pubmed: 29682033
Brain Inj. 2018;32(10):1208-1217
pubmed: 30024781
Front Neurol. 2019 Apr 26;10:446
pubmed: 31105646

Auteurs

Sukhwinder Sandhu (S)

Department of Neuroradiology, Mayo Clinic, University of Florida Health, Jacksonville, Florida, USA.

Erik Soule (E)

Departments of Interventional Radiology, University of Florida Health, Jacksonville, Florida, USA.

Peter Fiester (P)

Neuroradiology, University of Florida Health, Jacksonville, Florida, USA.

Patrick Natter (P)

Neuroradiology, University of Florida Health, Jacksonville, Florida, USA.

Daryoush Tavanaiepour (D)

Neurosurgery, University of Florida Health, Jacksonville, Florida, USA.

Gazanfar Rahmathulla (G)

Neurosurgery, University of Florida Health, Jacksonville, Florida, USA.

Dinesh Rao (D)

Neuroradiology, University of Florida Health, Jacksonville, Florida, USA.

Classifications MeSH