Plasma Levels of Soluble PD-L1 Correlate With Tumor Regression in Patients With Lung and Gastric Cancer Treated With Immune Checkpoint Inhibitors.


Journal

Anticancer research
ISSN: 1791-7530
Titre abrégé: Anticancer Res
Pays: Greece
ID NLM: 8102988

Informations de publication

Date de publication:
Sep 2019
Historique:
received: 15 07 2019
revised: 28 07 2019
accepted: 30 07 2019
entrez: 15 9 2019
pubmed: 15 9 2019
medline: 27 9 2019
Statut: ppublish

Résumé

Cancer immune therapy by immune checkpoint inhibitors (ICIs) is a promising therapeutic strategy for various cancer types. Among ICIs, anti-programmed cell death protein-1 (PD1) and anti-programmed death-ligand 1 (PD-L1) antibodies have shown a remarkable clinical benefit. The present study aimed to address the functional and clinical significance of serum levels of soluble PD-L1 (sPD-L1) in patients. A total of 21 patients, 11 with NSCLC, nine with gastric cancer and one with bladder cancer, who underwent anti-PD-1 therapy were evaluated for sPD-L1 concentration by ELISA analyses at diagnosis and after treatment. Pretreatment levels of sPD-L1 in patients who received ICIs were not remarkably correlated with the overall survival of these patients (r=0.3394, p=0.1323). Reduction of plasma sPD-L1 level was significantly correlated with tumor regression in patients administered four cycles of treatment (p<0.05). sPD-L1 might be derived and secreted from tumors and might be useful to identify primary responders to ICIs at a relatively early treatment timepoint.

Sections du résumé

BACKGROUND/AIM OBJECTIVE
Cancer immune therapy by immune checkpoint inhibitors (ICIs) is a promising therapeutic strategy for various cancer types. Among ICIs, anti-programmed cell death protein-1 (PD1) and anti-programmed death-ligand 1 (PD-L1) antibodies have shown a remarkable clinical benefit. The present study aimed to address the functional and clinical significance of serum levels of soluble PD-L1 (sPD-L1) in patients.
MATERIALS AND METHODS METHODS
A total of 21 patients, 11 with NSCLC, nine with gastric cancer and one with bladder cancer, who underwent anti-PD-1 therapy were evaluated for sPD-L1 concentration by ELISA analyses at diagnosis and after treatment.
RESULTS RESULTS
Pretreatment levels of sPD-L1 in patients who received ICIs were not remarkably correlated with the overall survival of these patients (r=0.3394, p=0.1323). Reduction of plasma sPD-L1 level was significantly correlated with tumor regression in patients administered four cycles of treatment (p<0.05).
CONCLUSION CONCLUSIONS
sPD-L1 might be derived and secreted from tumors and might be useful to identify primary responders to ICIs at a relatively early treatment timepoint.

Identifiants

pubmed: 31519633
pii: 39/9/5195
doi: 10.21873/anticanres.13716
doi:

Substances chimiques

Antineoplastic Agents, Immunological 0
B7-H1 Antigen 0
Biomarkers, Tumor 0
CD274 protein, human 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5195-5201

Informations de copyright

Copyright© 2019, International Institute of Anticancer Research (Dr. George J. Delinasios), All rights reserved.

Auteurs

Kiyohiro Ando (K)

Department of Clinical Diagnostic Oncology, Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.

Kazuyuki Hamada (K)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Makoto Watanabe (M)

Department of Clinical Diagnostic Oncology, Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.

Ryotaro Ohkuma (R)

Department of Clinical Diagnostic Oncology, Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Midori Shida (M)

Department of Clinical Diagnostic Oncology, Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.

Rie Onoue (R)

Department of Clinical Diagnostic Oncology, Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.

Yutaro Kubota (Y)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Hiroto Matsui (H)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Tomoyuki Ishiguro (T)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Yuya Hirasawa (Y)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Hirotsugu Ariizumi (H)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Junji Tsurutani (J)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.
Advanced Cancer Translational Research Institute, Showa University, Tokyo, Japan.

Kiyoshi Yoshimura (K)

Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Takuya Tsunoda (T)

Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

Shinichi Kobayashi (S)

Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.

Satoshi Wada (S)

Department of Clinical Diagnostic Oncology, Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan st-wada@med.showa-u.ac.jp.
Clinical Research Institute for Clinical Pharmacology and Therapeutics, Showa University, Tokyo, Japan.
Department of Medicine, Division of Medical Oncology, School of Medicine, Showa University, Tokyo, Japan.

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Classifications MeSH