Calcium/Calmodulin-Dependent Kinase (CaMKII) Inhibition Protects Against Purkinje Cell Damage Following CA/CPR in Mice.
Calcium/calmodulin-dependent protein kinase
Cerebellum
Excitotoxicity
Ischemia
Neuroprotection
Journal
Molecular neurobiology
ISSN: 1559-1182
Titre abrégé: Mol Neurobiol
Pays: United States
ID NLM: 8900963
Informations de publication
Date de publication:
Jan 2020
Jan 2020
Historique:
received:
29
08
2019
accepted:
29
08
2019
pubmed:
15
9
2019
medline:
20
11
2020
entrez:
15
9
2019
Statut:
ppublish
Résumé
Ischemic brain damage is triggered by glutamate excitotoxicity resulting in neuronal cell death. Previous research has demonstrated that N-methly-D-aspartate (NMDA) receptor activation triggers downstream calcium-dependent signaling pathways, specifically Ca
Identifiants
pubmed: 31520314
doi: 10.1007/s12035-019-01765-9
pii: 10.1007/s12035-019-01765-9
pmc: PMC6980452
mid: NIHMS1539857
doi:
Substances chimiques
Calmodulin
0
Protective Agents
0
Calcium-Calmodulin-Dependent Protein Kinase Type 2
EC 2.7.11.17
Calcium
SY7Q814VUP
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
150-158Subventions
Organisme : NINDS NIH HHS
ID : K01 NS086969
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS080851
Pays : United States
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