Multiplex gene expression profile in inflamed mucosa of patients with Crohn's disease ileal localization: A pilot study.

Colon Crohn’s disease Ileum Inflammation Messenger ribonucleic acid Microbiota Th1/Th17

Journal

World journal of clinical cases
ISSN: 2307-8960
Titre abrégé: World J Clin Cases
Pays: United States
ID NLM: 101618806

Informations de publication

Date de publication:
06 Sep 2019
Historique:
received: 20 03 2019
revised: 12 07 2019
accepted: 27 07 2019
entrez: 28 9 2019
pubmed: 29 9 2019
medline: 29 9 2019
Statut: ppublish

Résumé

Crohn's disease (CD) is a complex disorder resulting from the interaction of genetic, environmental, and microbial factors. The pathogenic process may potentially affect any segment of the gastrointestinal tract, but a selective location in the terminal ileum was reported in 50% of patients. To characterize clinical sub-phenotypes (colonic and/or ileal) within the same disease, in order to identify new therapeutic targets. 14 consecutive patients undergoing surgery for ileal CD were recruited for this study. Peripheral blood samples from each patient were collected and the main polymorphisms of the gene We found a significant increase of Th17 (IL17A and IL17F, IL 23R and CCR6) and Th1 (IFN-γ) gene expression in inflamed mucosa compared to non-inflamed sites of 14 CD patients. We observed that the expression of ileal genes related to Th1 and Th17 activity is strongly activated as well as the expression of genes involved in microbiota regulation.

Sections du résumé

BACKGROUND BACKGROUND
Crohn's disease (CD) is a complex disorder resulting from the interaction of genetic, environmental, and microbial factors. The pathogenic process may potentially affect any segment of the gastrointestinal tract, but a selective location in the terminal ileum was reported in 50% of patients.
AIM OBJECTIVE
To characterize clinical sub-phenotypes (colonic and/or ileal) within the same disease, in order to identify new therapeutic targets.
METHODS METHODS
14 consecutive patients undergoing surgery for ileal CD were recruited for this study. Peripheral blood samples from each patient were collected and the main polymorphisms of the gene
RESULTS RESULTS
We found a significant increase of Th17 (IL17A and IL17F, IL 23R and CCR6) and Th1 (IFN-γ) gene expression in inflamed mucosa compared to non-inflamed sites of 14 CD patients.
CONCLUSION CONCLUSIONS
We observed that the expression of ileal genes related to Th1 and Th17 activity is strongly activated as well as the expression of genes involved in microbiota regulation.

Identifiants

pubmed: 31559282
doi: 10.12998/wjcc.v7.i17.2463
pmc: PMC6745337
doi:

Types de publication

Journal Article

Langues

eng

Pagination

2463-2476

Déclaration de conflit d'intérêts

Conflict-of-interest statement: The authors declare that they have no competing interests.

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Auteurs

Francesco Giudici (F)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Letizia Lombardelli (L)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Edda Russo (E)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy. edda.russo@unifi.it.

Tiziana Cavalli (T)

Dipartimento Chirurgico Ortopedico, Ospedale Carlo Poma di Mantova, Firenze 50134, Italy.

Daniela Zambonin (D)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Federica Logiodice (F)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Ornela Kullolli (O)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Lamberto Giusti (L)

Department of Experimental and Clinical Biomedical Sciences "Mario Serio", University of Firenze, Firenze 50134, Italy.

Tatiana Bargellini (T)

Surgical Unit, Department of Surgery and Translational Medicine, University of Firenze, Firenze 50134, Italy.

Marilena Fazi (M)

Surgical Unit, Department of Surgery and Translational Medicine, University of Firenze, Firenze 50134, Italy.

Livia Biancone (L)

Department of Internal Medicine, University of Roma Tor Vergata, Roma 00133, Italy.

Stefano Scaringi (S)

Surgical Unit, Department of Surgery and Translational Medicine, University of Firenze, Firenze 50134, Italy.

Ann Maria Clemente (AM)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Eloisa Perissi (E)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Giovanni Delfino (G)

Department of Experimental and Clinical Biomedical Sciences "Mario Serio", University of Firenze, Firenze 50134, Italy.

Maria G Torcia (MG)

Marie- Pierre Piccinni, Department of Experimental and Clinical Medicine, University of Firenze, Firenze 50134, Italy.

Ferdinando Ficari (F)

Surgical Unit, Department of Surgery and Translational Medicine, University of Firenze, Firenze 50134, Italy.

Francesco Tonelli (F)

Surgical Unit, Department of Surgery and Translational Medicine, University of Firenze, Firenze 50134, Italy.

Cecilia Malentacchi (C)

Department of Experimental and Clinical Biomedical Sciences "Mario Serio", University of Firenze, Firenze 50134, Italy.

Classifications MeSH