Folate levels in pregnancy and offspring food allergy and eczema.


Journal

Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology
ISSN: 1399-3038
Titre abrégé: Pediatr Allergy Immunol
Pays: England
ID NLM: 9106718

Informations de publication

Date de publication:
01 2020
Historique:
received: 01 04 2019
revised: 09 09 2019
accepted: 16 09 2019
pubmed: 1 10 2019
medline: 15 12 2020
entrez: 1 10 2019
Statut: ppublish

Résumé

High folate status in pregnancy has been implicated in the increased prevalence of allergic disease, but there are no published data relating directly measured folate status in pregnancy to challenge-proven food allergy among offspring. The study aim was to examine the association between red blood cell (RBC) folate status in trimester three of pregnancy and allergic disease among offspring. Red blood cell folate levels were measured at 28-32 weeks' gestation in a prospective birth cohort (n = 1074). Food allergy outcomes were assessed in 1-year-old infants by skin prick testing and subsequent food challenge. Eczema was assessed by questionnaire and clinical review. High trimester three RBC folate was defined as greater than (>) 1360 nmol/L. Binomial regression was used to examine associations between trimester three RBC folate and allergic outcomes, adjusting for potential confounders. Red blood cell folate levels were measured in 88% (894/1064) of pregnant women. The mean concentration was 1695.6 nmol/L (standard deviation 415.4) with 82% (731/894) >1360 nmol/L. There was no evidence of either linear or non-linear relationships between trimester three RBC folate and allergic outcomes, nor evidence of associations between high RBC folate and food allergy (adjusted risk ratio (aRR) 2.89, 95% CI 0.90-9.35), food sensitization (aRR 1.72, 95% CI 0.85-3.49), or eczema (aRR 0.97, 95% CI 0.67-1.38). The majority of pregnant women in this study had high RBC folate levels. There was no evidence of associations between trimester three RBC folate and food allergy, food sensitization, or eczema among the offspring, although larger studies are required.

Sections du résumé

BACKGROUND
High folate status in pregnancy has been implicated in the increased prevalence of allergic disease, but there are no published data relating directly measured folate status in pregnancy to challenge-proven food allergy among offspring. The study aim was to examine the association between red blood cell (RBC) folate status in trimester three of pregnancy and allergic disease among offspring.
METHODS
Red blood cell folate levels were measured at 28-32 weeks' gestation in a prospective birth cohort (n = 1074). Food allergy outcomes were assessed in 1-year-old infants by skin prick testing and subsequent food challenge. Eczema was assessed by questionnaire and clinical review. High trimester three RBC folate was defined as greater than (>) 1360 nmol/L. Binomial regression was used to examine associations between trimester three RBC folate and allergic outcomes, adjusting for potential confounders.
RESULTS
Red blood cell folate levels were measured in 88% (894/1064) of pregnant women. The mean concentration was 1695.6 nmol/L (standard deviation 415.4) with 82% (731/894) >1360 nmol/L. There was no evidence of either linear or non-linear relationships between trimester three RBC folate and allergic outcomes, nor evidence of associations between high RBC folate and food allergy (adjusted risk ratio (aRR) 2.89, 95% CI 0.90-9.35), food sensitization (aRR 1.72, 95% CI 0.85-3.49), or eczema (aRR 0.97, 95% CI 0.67-1.38).
CONCLUSION
The majority of pregnant women in this study had high RBC folate levels. There was no evidence of associations between trimester three RBC folate and food allergy, food sensitization, or eczema among the offspring, although larger studies are required.

Identifiants

pubmed: 31566807
doi: 10.1111/pai.13128
doi:

Substances chimiques

Folic Acid 935E97BOY8

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

38-46

Informations de copyright

© 2019 EAACI and John Wiley and Sons A/S. Published by John Wiley and Sons Ltd.

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Auteurs

John Molloy (J)

School of Medicine, Deakin University, Waurn Ponds, Victoria, Australia.
Child Health Research Unit, Barwon Health, Geelong, Victoria, Australia.
Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.

Fiona Collier (F)

School of Medicine, Deakin University, Waurn Ponds, Victoria, Australia.
Child Health Research Unit, Barwon Health, Geelong, Victoria, Australia.

Richard Saffery (R)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.

Katrina J Allen (KJ)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.
Department of Allergy and Immunology, Royal Children's Hospital, Parkville, Victoria, Australia.

Jennifer J Koplin (JJ)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.
Centre for Epidemiology and Biostatistics, The University of Melbourne, Carlton, Victoria, Australia.

Anne Louise Ponsonby (A)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.

Mimi L K Tang (MLK)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.
Department of Allergy and Immunology, Royal Children's Hospital, Parkville, Victoria, Australia.

Alister C Ward (AC)

School of Medicine, Deakin University, Waurn Ponds, Victoria, Australia.

David Martino (D)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.

David Burgner (D)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.
Department of Paediatrics, Monash University, Clayton, Victoria, Australia.

John B Carlin (JB)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.
Centre for Epidemiology and Biostatistics, The University of Melbourne, Carlton, Victoria, Australia.

Sarath Ranganathan (S)

Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.
Department of Respiratory Medicine, Royal Children's Hospital, Parkville, Victoria, Australia.

Christos Symeonedies (C)

Child Health Research Unit, Barwon Health, Geelong, Victoria, Australia.
Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Department of Paediatrics, University of Melbourne, Parkville, Victoria, Australia.

Terence Dwyer (T)

The George Institute for Global Health, University of Oxford, Oxford, UK.

Peter Vuillermin (P)

School of Medicine, Deakin University, Waurn Ponds, Victoria, Australia.
Child Health Research Unit, Barwon Health, Geelong, Victoria, Australia.
Murdoch Childrens Research Institute, Parkville, Victoria, Australia.
Centre for Food and Allergy Research, Parkville, Victoria, Australia.

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