What remains after transient global amnesia (TGA)? An ultra-high field 7 T magnetic resonance imaging study of the hippocampus.


Journal

European journal of neurology
ISSN: 1468-1331
Titre abrégé: Eur J Neurol
Pays: England
ID NLM: 9506311

Informations de publication

Date de publication:
02 2020
Historique:
received: 29 04 2019
revised: 10 08 2019
accepted: 27 09 2019
pubmed: 2 10 2019
medline: 25 6 2021
entrez: 2 10 2019
Statut: ppublish

Résumé

The aim was to study whether ultra-high field 7 T magnetic resonance imaging (MRI) can demonstrate chronic focal defects in the hippocampus corresponding to the former acute diffusion-weighted imaging (DWI) lesions and to assess chronic T2-hyperintense hippocampal lesion load in transient global amnesia (TGA) patients. Follow-up of 7 T MRI of the hippocampus was performed in 13 patients with documented hippocampal DWI lesions (detected via 3 T MRI) after acute TGA. The location of the DWI lesions was transformed to 7 T T2 images after data co-registration. Additionally, the T2-hyperintense lesion load was estimated in each patient and compared with that of 13 healthy controls. Magnetic resonance imaging (7 T) was performed after a median of 4 months. No structural abnormality at the site of the previous TGA lesion was observed in any case. None of the controls showed DWI lesions. There was no significant difference between patients and controls concerning the number (P = 0.67) or volume (P = 0.45) of T2-hyperintense hippocampal lesions. Diffusion-weighted imaging lesions in patients with TGA do not provoke any visible sequelae and do not result in hippocampal cavities. The occurrence of incidental hippocampal T2 lesions after TGA is not more frequent than in controls.

Sections du résumé

BACKGROUND AND PURPOSE
The aim was to study whether ultra-high field 7 T magnetic resonance imaging (MRI) can demonstrate chronic focal defects in the hippocampus corresponding to the former acute diffusion-weighted imaging (DWI) lesions and to assess chronic T2-hyperintense hippocampal lesion load in transient global amnesia (TGA) patients.
METHODS
Follow-up of 7 T MRI of the hippocampus was performed in 13 patients with documented hippocampal DWI lesions (detected via 3 T MRI) after acute TGA. The location of the DWI lesions was transformed to 7 T T2 images after data co-registration. Additionally, the T2-hyperintense lesion load was estimated in each patient and compared with that of 13 healthy controls.
RESULTS
Magnetic resonance imaging (7 T) was performed after a median of 4 months. No structural abnormality at the site of the previous TGA lesion was observed in any case. None of the controls showed DWI lesions. There was no significant difference between patients and controls concerning the number (P = 0.67) or volume (P = 0.45) of T2-hyperintense hippocampal lesions.
CONCLUSIONS
Diffusion-weighted imaging lesions in patients with TGA do not provoke any visible sequelae and do not result in hippocampal cavities. The occurrence of incidental hippocampal T2 lesions after TGA is not more frequent than in controls.

Identifiants

pubmed: 31573112
doi: 10.1111/ene.14099
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

406-409

Informations de copyright

© 2019 European Academy of Neurology.

Références

Nakada T, Kwee IL, Fujii Y, Knight RT. High-field, T2 reversed MRI of the hippocampus in transient global amnesia. Neurology 2005; 64: 1170-1174.
Trattnig S, Springer E, Bogner W, et al. Key clinical benefits of neuroimaging at 7 T. NeuroImage 2018; 168: 477-489.
Jenkinson M, Smith S. A global optimisation method for robust affine registration of brain images. Med Image Anal. 2001; 5: 143-156.
Greve DN, Fischl B. Accurate and robust brain image alignment using boundary-based registration. NeuroImage 2009; 48: 63-72.
Bartsch T, Alfke K, Stingele R, et al. Selective affection of hippocampal CA-1 neurons in patients with transient global amnesia without long-term sequelae. Brain 2006; 129(Pt 11): 2874-2884.
Uttner I, Weber S, Freund W, et al. Hippocampal cavities are not associated with cognitive impairment in transient global amnesia. Eur J Neurol. 2011; 18: 882-887.
Wang X, Zhang R, Wei W, et al. Long-term sequelae of hippocampal lesions in patients with transient global amnesia: a multiparametric MRI study. J Magn Reson Imaging. 2018; 47: 1350-1358.
Maller JJ, Reglade-Meslin C, Chan P, et al. Hippocampal sulcal cavities: prevalence, risk factors and relationship to memory impairment. Brain Res. 2011; 1368: 222-230.
van Veluw SJ, Wisse LE, Kuijf HJ, et al. Hippocampal T2 hyperintensities on 7 Tesla MRI. Neuroimage Clin. 2013; 3: 196-201.

Auteurs

D Paech (D)

Department of Radiology, German Cancer Research Center (dkfz), Heidelberg, Germany.

T A Kuder (TA)

Medical Physics in Radiology, German Cancer Research Center (dkfz), Heidelberg, Germany.

C Roßmanith (C)

Department of Neurology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

M Griebe (M)

Department of Neurology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

P Eisele (P)

Department of Neurology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

M Platten (M)

Department of Neurology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

M E Ladd (ME)

Medical Physics in Radiology, German Cancer Research Center (dkfz), Heidelberg, Germany.
Faculty of Physics and Astronomy and Faculty of Medicine, Heidelberg University, Heidelberg, Germany.

H-P Schlemmer (HP)

Department of Radiology, German Cancer Research Center (dkfz), Heidelberg, Germany.

A Gass (A)

Department of Neurology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

K Szabo (K)

Department of Neurology, Medical Faculty Mannheim, Heidelberg University, Mannheim, Germany.

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