Response to salvage chemotherapy after progression on immune checkpoint inhibitors in patients with recurrent and/or metastatic squamous cell carcinoma of the head and neck.


Journal

European journal of cancer (Oxford, England : 1990)
ISSN: 1879-0852
Titre abrégé: Eur J Cancer
Pays: England
ID NLM: 9005373

Informations de publication

Date de publication:
11 2019
Historique:
received: 18 03 2019
revised: 22 07 2019
accepted: 26 08 2019
pubmed: 2 10 2019
medline: 9 6 2020
entrez: 2 10 2019
Statut: ppublish

Résumé

Immune checkpoint inhibitors (ICI) are active in patients with recurrent/metastatic squamous cell carcinoma of the head and neck (R/M SCCHN). Recent data suggest that exposure to ICI improves response to salvage chemotherapy (SCT) in advanced non-small-cell lung cancer. We evaluated response to chemotherapy in patients who had progressed on ICI in patients with R/M SCCHN. A retrospective study was conducted at 4 French centres. Eligibility criteria were patients who progressed after treatment with ICI for R/M SCCHN and received SCT and for whom efficacy data were available between September 2014 and January 2018. Of 232 patients treated with ICI, 82 met eligibility criteria: 84% were male. ICI was given as monotherapy in 45% of patients or as combination in 55%. SCT included taxanes (56.1%), cetuximab in combination with taxanes or platinum (50%), platinum-based regimen (36.6%). The median number of treatment lines before SCT was 2 (range 1-6). The objective response rate (ORR) to SCT was 30%. Three patients (4%) presented complete response and 22 patients (27%) had partial response. Median progression-free survival was 3.6 months and median overall survival was 7.8 months. The age at SCT, initial tumour location, number of prior chemotherapy regimens, type of chemotherapy before ICI, best response to ICI, site of relapse and Eastern Cooperative Oncology Group at SCT were not associated with response to SCT on univariate analysis. In R/M SCCHN, the ORR to SCT was high (30%) suggesting that exposure to ICI may increase tumour sensitivity to chemotherapy.

Sections du résumé

BACKGROUND
Immune checkpoint inhibitors (ICI) are active in patients with recurrent/metastatic squamous cell carcinoma of the head and neck (R/M SCCHN). Recent data suggest that exposure to ICI improves response to salvage chemotherapy (SCT) in advanced non-small-cell lung cancer. We evaluated response to chemotherapy in patients who had progressed on ICI in patients with R/M SCCHN.
PATIENTS AND METHODS
A retrospective study was conducted at 4 French centres. Eligibility criteria were patients who progressed after treatment with ICI for R/M SCCHN and received SCT and for whom efficacy data were available between September 2014 and January 2018.
RESULTS
Of 232 patients treated with ICI, 82 met eligibility criteria: 84% were male. ICI was given as monotherapy in 45% of patients or as combination in 55%. SCT included taxanes (56.1%), cetuximab in combination with taxanes or platinum (50%), platinum-based regimen (36.6%). The median number of treatment lines before SCT was 2 (range 1-6). The objective response rate (ORR) to SCT was 30%. Three patients (4%) presented complete response and 22 patients (27%) had partial response. Median progression-free survival was 3.6 months and median overall survival was 7.8 months. The age at SCT, initial tumour location, number of prior chemotherapy regimens, type of chemotherapy before ICI, best response to ICI, site of relapse and Eastern Cooperative Oncology Group at SCT were not associated with response to SCT on univariate analysis.
CONCLUSION
In R/M SCCHN, the ORR to SCT was high (30%) suggesting that exposure to ICI may increase tumour sensitivity to chemotherapy.

Identifiants

pubmed: 31574417
pii: S0959-8049(19)30486-1
doi: 10.1016/j.ejca.2019.08.026
pii:
doi:

Substances chimiques

Antibodies, Monoclonal 0
Protein Kinase Inhibitors 0

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

123-129

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Khalil Saleh (K)

Department of Head and Neck Oncology, Gustave Roussy Cancer Campus, Villejuif, France. Electronic address: khalil.saleh@gustaveroussy.fr.

Amaury Daste (A)

Department of Medical Oncology, Hôpital Saint-André, Bordeaux University Hospital-CHU Bordeaux, France.

Nicolas Martin (N)

Department of Medical Oncology, Centre Antoine Lacassagne, Nice, France.

Elvire Pons-Tostivint (E)

Department of Drug Development and Innovation, Institute Curie, Paris, Saint-Cloud, France.

Anne Auperin (A)

Department of Biostatistics and Epidemiology, Gustave Roussy Cancer Campus, Villejuif, France.

Ruth Gabriela Herrera-Gomez (RG)

Department of Head and Neck Oncology, Gustave Roussy Cancer Campus, Villejuif, France.

Neus Baste-Rotllan (N)

Department of Head and Neck Oncology, Gustave Roussy Cancer Campus, Villejuif, France.

Francois Bidault (F)

Department of Radiology, Institute Gustave Roussy, Villejuif, France.

Joel Guigay (J)

Department of Medical Oncology, Centre Antoine Lacassagne, Nice, France.

Christophe Le Tourneau (C)

Department of Drug Development and Innovation, Institute Curie, Paris, Saint-Cloud, France; INSERM U900 Research Unit, Saint-Cloud, France; Paris-Saclay University, Paris, France.

Esma Saada-Bouzid (E)

Department of Medical Oncology, Centre Antoine Lacassagne, Nice, France; Université Côte d'Azur, Nice, France.

Caroline Even (C)

Department of Head and Neck Oncology, Gustave Roussy Cancer Campus, Villejuif, France.

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