Predicting response to benralizumab in chronic obstructive pulmonary disease: analyses of GALATHEA and TERRANOVA studies.


Journal

The Lancet. Respiratory medicine
ISSN: 2213-2619
Titre abrégé: Lancet Respir Med
Pays: England
ID NLM: 101605555

Informations de publication

Date de publication:
02 2020
Historique:
received: 31 07 2019
revised: 12 09 2019
accepted: 12 09 2019
pubmed: 3 10 2019
medline: 25 8 2020
entrez: 3 10 2019
Statut: ppublish

Résumé

Benralizumab did not significantly reduce exacerbations compared with placebo in the phase 3 GALATHEA and TERRANOVA trials of benralizumab for patients with chronic obstructive pulmonary disease (COPD). We aimed to identify clinical and physiological characteristics of patients with COPD that could help to identify people who are likely to have the greatest treatment effect with benralizumab. We analysed individual study and pooled results from GALATHEA and TERRANOVA. At study enrolment, patients from GALATHEA and TERRANOVA were aged 40-85 years, had moderate to very severe airflow limitation, had elevated blood eosinophil counts, and at least two exacerbations or one severe exacerbation in the previous year despite dual inhaled therapy (inhaled corticosteroids plus long-acting β For 2665 patients with elevated blood eosinophil counts, treatment effect with benralizumab every 8 weeks at 100 mg, but not at 30 mg, occurred for patients with a history of more frequent exacerbations, poorer baseline lung function, or greater baseline lung function improvement with short-acting bronchodilators. Patients with baseline blood eosinophil counts of 220 cells per μL or greater with: three or more exacerbations in the previous year receiving benralizumab every 8 weeks versus placebo, had rate ratios (RRs) of 0·69 (95% CI 0·56-0·83) for 100 mg and 0·86 (0·71-1·04) for 30 mg; postbronchodilator FEV Elevated blood eosinophil counts combined with clinical characteristics identified a subpopulation of patients with COPD who had reductions in exacerbations with benralizumab treatment. These hypothesis-generating analyses identified the potential efficacy of benralizumab 100 mg for this subpopulation. These findings require prospective evaluation in clinical trials. AstraZeneca.

Sections du résumé

BACKGROUND
Benralizumab did not significantly reduce exacerbations compared with placebo in the phase 3 GALATHEA and TERRANOVA trials of benralizumab for patients with chronic obstructive pulmonary disease (COPD). We aimed to identify clinical and physiological characteristics of patients with COPD that could help to identify people who are likely to have the greatest treatment effect with benralizumab.
METHODS
We analysed individual study and pooled results from GALATHEA and TERRANOVA. At study enrolment, patients from GALATHEA and TERRANOVA were aged 40-85 years, had moderate to very severe airflow limitation, had elevated blood eosinophil counts, and at least two exacerbations or one severe exacerbation in the previous year despite dual inhaled therapy (inhaled corticosteroids plus long-acting β
FINDINGS
For 2665 patients with elevated blood eosinophil counts, treatment effect with benralizumab every 8 weeks at 100 mg, but not at 30 mg, occurred for patients with a history of more frequent exacerbations, poorer baseline lung function, or greater baseline lung function improvement with short-acting bronchodilators. Patients with baseline blood eosinophil counts of 220 cells per μL or greater with: three or more exacerbations in the previous year receiving benralizumab every 8 weeks versus placebo, had rate ratios (RRs) of 0·69 (95% CI 0·56-0·83) for 100 mg and 0·86 (0·71-1·04) for 30 mg; postbronchodilator FEV
INTERPRETATION
Elevated blood eosinophil counts combined with clinical characteristics identified a subpopulation of patients with COPD who had reductions in exacerbations with benralizumab treatment. These hypothesis-generating analyses identified the potential efficacy of benralizumab 100 mg for this subpopulation. These findings require prospective evaluation in clinical trials.
FUNDING
AstraZeneca.

Identifiants

pubmed: 31575508
pii: S2213-2600(19)30338-8
doi: 10.1016/S2213-2600(19)30338-8
pii:
doi:

Substances chimiques

Anti-Asthmatic Agents 0
Antibodies, Monoclonal, Humanized 0
Biomarkers 0
benralizumab 71492GE1FX

Banques de données

ClinicalTrials.gov
['NCT02138916', 'NCT02155660']

Types de publication

Evaluation Study Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

158-170

Subventions

Organisme : Department of Health
Pays : United Kingdom

Commentaires et corrections

Type : CommentIn

Informations de copyright

Copyright © 2020 Elsevier Ltd. All rights reserved.

Auteurs

Gerard J Criner (GJ)

Lewis Katz School of Medicine, Temple University, Philadelphia, PA, USA. Electronic address: gerard.criner@tuhs.temple.edu.

Bartolome R Celli (BR)

Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.

Dave Singh (D)

University of Manchester, Manchester University NHS Hospital Trust, Manchester, UK.

Alvar Agusti (A)

Respiratory Institute, Hospital Clinic, University of Barcelona, Institut d'Investigacions Biomèdiques August Pi i Sunyer, Centro de Investigación Biomédica en Red Enfermedades Respiratorias, Barcelona, Spain.

Alberto Papi (A)

Department of Medical Sciences, University of Ferrara, Ferrara, Italy.

Maria Jison (M)

AstraZeneca, Gaithersburg, MD, USA.

Natalya Makulova (N)

AstraZeneca, Gaithersburg, MD, USA.

Vivian H Shih (VH)

AstraZeneca, Gaithersburg, MD, USA.

Laura Brooks (L)

AstraZeneca, Gaithersburg, MD, USA.

Peter Barker (P)

AstraZeneca, Gaithersburg, MD, USA.

Ubaldo J Martin (UJ)

AstraZeneca, Gaithersburg, MD, USA.

Paul Newbold (P)

AstraZeneca, Gaithersburg, MD, USA.

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Classifications MeSH