Older and younger patients treated with immune checkpoint inhibitors have similar outcomes in real-life setting.


Journal

European journal of cancer (Oxford, England : 1990)
ISSN: 1879-0852
Titre abrégé: Eur J Cancer
Pays: England
ID NLM: 9005373

Informations de publication

Date de publication:
11 2019
Historique:
received: 20 04 2019
revised: 10 08 2019
accepted: 26 08 2019
pubmed: 8 10 2019
medline: 9 6 2020
entrez: 8 10 2019
Statut: ppublish

Résumé

Age-related immune dysfunction might impair the efficacy of immune checkpoint inhibitors (ICIs) in older patients. We aimed to evaluate the impact of age on clinical outcomes and tolerance of ICIs in a real-life setting. All patients receiving a single-agent ICI (cytotoxic T-lymphocyte-associated protein 4 [CTLA-4] or programmed death(ligand)1 [PD(L)-1] inhibitors) for the standard treatment of a locally advanced or metastatic cancer were included in this retrospective multicentric series. The primary end-point was overall survival (OS). Progression-free survival (PFS) and immune-related adverse events (irAEs) were secondary end-points. The impact of age was assessed using the threshold of 70 years. A total of 410 patients were included, for 435 lines of treatment, including 150 lines (34%) given to patients aged 70 years or older. The primary tumour types were lung cancer (n = 304, 74%), melanoma (n = 79, 19%) and urologic cancer (n = 27, 7%). Most of the administered treatments were PD(L)-1 inhibitors (n = 356, 82%). Median follow-up reached 46 months in the CTLA-4 cohort, and 20 months in the PD(L)-1 cohort. In both treatment cohorts, age did not impact OS (respectively, HR = 0.82, 95% CI 0.5-1.4; log-rank P = 0.49 and HR = 0.9, 95% CI 0.7-1.1; log-rank P = 0.27) or PFS (HR = 0.7, 95% CI 0.4-1.1; log-rank P = 0.13 and HR = 0.9, 95% CI 0.7-1.1; log-rank P = 0.19). Grade 3-4 irAEs rates were not statistically different between older and younger patients (11% vs 12%, P = 0.87). In a large real-world series of patients treated by ICI monotherapy, the long-term clinical outcomes were not statistically different between older or younger patients, with no increased immune-related toxicity.

Sections du résumé

BACKGROUND
Age-related immune dysfunction might impair the efficacy of immune checkpoint inhibitors (ICIs) in older patients. We aimed to evaluate the impact of age on clinical outcomes and tolerance of ICIs in a real-life setting.
METHODS
All patients receiving a single-agent ICI (cytotoxic T-lymphocyte-associated protein 4 [CTLA-4] or programmed death(ligand)1 [PD(L)-1] inhibitors) for the standard treatment of a locally advanced or metastatic cancer were included in this retrospective multicentric series. The primary end-point was overall survival (OS). Progression-free survival (PFS) and immune-related adverse events (irAEs) were secondary end-points. The impact of age was assessed using the threshold of 70 years.
RESULTS
A total of 410 patients were included, for 435 lines of treatment, including 150 lines (34%) given to patients aged 70 years or older. The primary tumour types were lung cancer (n = 304, 74%), melanoma (n = 79, 19%) and urologic cancer (n = 27, 7%). Most of the administered treatments were PD(L)-1 inhibitors (n = 356, 82%). Median follow-up reached 46 months in the CTLA-4 cohort, and 20 months in the PD(L)-1 cohort. In both treatment cohorts, age did not impact OS (respectively, HR = 0.82, 95% CI 0.5-1.4; log-rank P = 0.49 and HR = 0.9, 95% CI 0.7-1.1; log-rank P = 0.27) or PFS (HR = 0.7, 95% CI 0.4-1.1; log-rank P = 0.13 and HR = 0.9, 95% CI 0.7-1.1; log-rank P = 0.19). Grade 3-4 irAEs rates were not statistically different between older and younger patients (11% vs 12%, P = 0.87).
CONCLUSION
In a large real-world series of patients treated by ICI monotherapy, the long-term clinical outcomes were not statistically different between older or younger patients, with no increased immune-related toxicity.

Identifiants

pubmed: 31590080
pii: S0959-8049(19)30487-3
doi: 10.1016/j.ejca.2019.08.027
pii:
doi:

Substances chimiques

Antibodies, Monoclonal 0
Antineoplastic Agents, Immunological 0
B7-H1 Antigen 0
CD274 protein, human 0
CTLA-4 Antigen 0
CTLA4 protein, human 0
Protein Kinase Inhibitors 0

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

192-201

Informations de copyright

Copyright © 2019 Elsevier Ltd. All rights reserved.

Auteurs

Pauline Corbaux (P)

Hospices Civils de Lyon, Oncology Department, Pierre-Bénite, France; Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France.

Denis Maillet (D)

Hospices Civils de Lyon, Oncology Department, Pierre-Bénite, France; Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France.

Amélie Boespflug (A)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; Hospices Civils de Lyon, Cancer Research Center of Lyon, Dermatology Department, Pierre-Bénite, France.

Myriam Locatelli-Sanchez (M)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; Hospices Civils de Lyon, Cancer Research Center of Lyon, Department of Respiratory Medicine, Pierre-Bénite, France.

Marie Perier-Muzet (M)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; Hospices Civils de Lyon, Cancer Research Center of Lyon, Dermatology Department, Pierre-Bénite, France.

Michaël Duruisseaux (M)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; Hospices Civils de Lyon, Department of Respiratory Medicine, Groupement Hospitalier Est, Hôpital Louis-Pradel, Lyon, France.

Lize Kiakouama-Maleka (L)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; Hospices Civils de Lyon, Department of Respiratory Medicine, Croix-Rousse Hospital, Lyon, France.

Stéphane Dalle (S)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; Hospices Civils de Lyon, Cancer Research Center of Lyon, Dermatology Department, Pierre-Bénite, France.

Claire Falandry (C)

Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; Hospices Civils de Lyon, Geriatrics Unit, Pierre-Bénite, France; CarMen Biomedical Research Laboratory (Cardiovascular Diseases, Metabolism, Diabetology and Nutrition) INSERM UMR 1060, Université de Lyon, Oullins, France.

Julien Péron (J)

Hospices Civils de Lyon, Oncology Department, Pierre-Bénite, France; Université de Lyon, F-69000, Lyon, France; Université Lyon 1, F-69100, Villeurbanne, France; ImmuCare (Immunology Cancer Research) Institut de Cancérologie des Hospices Civils de Lyon, Lyon, France; CNRS, UMR 5558, Laboratoire de Biométrie et Biologie Evolutive, Equipe Biostatistique-Santé, F-69100, Villeurbanne, France. Electronic address: julien.peron@chu-lyon.fr.

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