Immunohistochemical analysis of tumor budding as predictor of lymph node metastasis from superficial esophageal squamous cell carcinoma.


Journal

Esophagus : official journal of the Japan Esophageal Society
ISSN: 1612-9067
Titre abrégé: Esophagus
Pays: Japan
ID NLM: 101206627

Informations de publication

Date de publication:
04 2020
Historique:
received: 18 06 2019
accepted: 01 10 2019
pubmed: 9 10 2019
medline: 15 7 2021
entrez: 10 10 2019
Statut: ppublish

Résumé

Tumor budding is known predictors of lymph node metastasis from esophageal squamous cell carcinoma. However, it is not easy to detect such small cell clusters on hematoxylin-eosin (HE) staining. Therefore, we evaluated tumor budding using immunohistochemistry (IHC) for epithelial cell markers. We analyzed tumor budding in 50 cases of superficial esophageal squamous cell carcinoma. We evaluated the impact of clinicopathological factors and tumor budding to predict lymph node metastasis. A total of 565 tumor sections were assessed using HE staining and IHC for cytokeratin 5/6. Based on receiver operating characteristic curves, the cut-off values for high-grade tumor budding evaluated using HE staining or IHC were 2 and 11, respectively. High-grade tumor budding evaluated using HE staining (P = 0.007) and IHC (P ≤ 0.001) were significantly correlated with lymph node metastasis. For tumors with pT1a-MM to pT1b-SM1, high-grade tumor budding evaluated using IHC was correlated with lymph node metastasis (P = 0.050). Tumor budding was significantly associated with lymph node metastasis. The optimal cut-off values of tumor budding on HE staining and tumor budding on IHC were 2 and 11, respectively. Even though both tumor budding on HE staining and tumor budding on IHC were significantly associated with lymph node metastasis, tumor budding on IHC tend to be more associated with lymph node metastasis.

Sections du résumé

BACKGROUND
Tumor budding is known predictors of lymph node metastasis from esophageal squamous cell carcinoma. However, it is not easy to detect such small cell clusters on hematoxylin-eosin (HE) staining. Therefore, we evaluated tumor budding using immunohistochemistry (IHC) for epithelial cell markers.
METHOD
We analyzed tumor budding in 50 cases of superficial esophageal squamous cell carcinoma. We evaluated the impact of clinicopathological factors and tumor budding to predict lymph node metastasis. A total of 565 tumor sections were assessed using HE staining and IHC for cytokeratin 5/6.
RESULTS
Based on receiver operating characteristic curves, the cut-off values for high-grade tumor budding evaluated using HE staining or IHC were 2 and 11, respectively. High-grade tumor budding evaluated using HE staining (P = 0.007) and IHC (P ≤ 0.001) were significantly correlated with lymph node metastasis. For tumors with pT1a-MM to pT1b-SM1, high-grade tumor budding evaluated using IHC was correlated with lymph node metastasis (P = 0.050).
CONCLUSIONS
Tumor budding was significantly associated with lymph node metastasis. The optimal cut-off values of tumor budding on HE staining and tumor budding on IHC were 2 and 11, respectively. Even though both tumor budding on HE staining and tumor budding on IHC were significantly associated with lymph node metastasis, tumor budding on IHC tend to be more associated with lymph node metastasis.

Identifiants

pubmed: 31595396
doi: 10.1007/s10388-019-00698-5
pii: 10.1007/s10388-019-00698-5
doi:

Substances chimiques

Keratins 68238-35-7

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

168-174

Références

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pubmed: 28548122
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pubmed: 23208313
Anticancer Res. 2013 Nov;33(11):5023-9
pubmed: 24222146
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pubmed: 28349281

Auteurs

Kazuhiro Fuchinoue (K)

Department of Gastroenterology, School of Medicine, Toho University, Tokyo, Japan.
Department of Clinical Oncology, Graduate School of Medicine, Toho University, Tokyo, Japan.

Tetsuo Nemoto (T)

Department of Pathology, Showa University Northern Yokohama Hospital, Yokohama, Japan.

Hideaki Shimada (H)

Department of Clinical Oncology, Graduate School of Medicine, Toho University, Tokyo, Japan. hideaki.shimada@med.toho-u.ac.jp.
Department of Gastroenterological Surgery, School of Medicine, Toho University, Tokyo, Japan. hideaki.shimada@med.toho-u.ac.jp.
Department of Surgery, Toho University Graduate School of Medicine, Tokyo, Japan. hideaki.shimada@med.toho-u.ac.jp.

Naobumi Tochigi (N)

Department of Surgical Pathology, School of Medicine, Toho University, Tokyo, Japan.

Yoshinori Igarashi (Y)

Department of Gastroenterology, School of Medicine, Toho University, Tokyo, Japan.

Satoshi Yajima (S)

Department of Gastroenterological Surgery, School of Medicine, Toho University, Tokyo, Japan.

Takashi Suzuki (T)

Department of Gastroenterological Surgery, School of Medicine, Toho University, Tokyo, Japan.

Yoko Oshima (Y)

Department of Gastroenterological Surgery, School of Medicine, Toho University, Tokyo, Japan.

Kazutoshi Shibuya (K)

Department of Surgical Pathology, School of Medicine, Toho University, Tokyo, Japan.

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