Behavioral Pharmacology of Novel Kappa Opioid Receptor Antagonists in Rats.


Journal

The international journal of neuropsychopharmacology
ISSN: 1469-5111
Titre abrégé: Int J Neuropsychopharmacol
Pays: England
ID NLM: 9815893

Informations de publication

Date de publication:
01 11 2019
Historique:
received: 01 07 2019
revised: 09 09 2019
accepted: 10 10 2019
pubmed: 16 10 2019
medline: 13 5 2020
entrez: 16 10 2019
Statut: ppublish

Résumé

New treatments for stress-related disorders including depression, anxiety, and substance use disorder are greatly needed. Kappa opioid receptors are expressed in the central nervous system, including areas implicated in analgesia and affective state. Although kappa opioid receptor agonists share the antinociceptive effects of mu opioid receptor agonists, they also tend to produce negative affective states. In contrast, selective kappa opioid receptor antagonists have antidepressant- and anxiolytic-like effects, stimulating interest in their therapeutic potential. The prototypical kappa opioid receptor antagonists (e.g., norBNI, JDTic) have an exceptionally long duration of action that complicates their use in humans, particularly in tests to establish safety. This study was designed to test dose- and time-course effects of novel kappa opioid receptor antagonists with the goal of identifying short-acting lead compounds for future medication development. We screened 2 novel, highly selective kappa opioid receptor antagonists (CYM-52220 and CYM-52288) with oral efficacy in the warm water tail flick assay in rats to determine initial dose and time course effects. For comparison, we tested existing kappa opioid receptor antagonists JDTic and LY-2456302 (also known as CERC-501 or JNJ-67953964). In the tail flick assay, the rank order of duration of action for the antagonists was LY-2456302 < CYM-52288 < CYM-52220 << JDTic. Furthermore, LY-2456302 blocked the depressive (anhedonia-producing) effects of the kappa opioid receptor agonist U50,488 in the intracranial self-stimulation paradigm, albeit at a higher dose than that needed for analgesic blockade in the tail flick assay. These results suggest that structurally diverse kappa opioid receptor antagonists can have short-acting effects and that LY-2456302 reduces anhedonia as measured in the intracranial self-stimulation test.

Sections du résumé

BACKGROUND
New treatments for stress-related disorders including depression, anxiety, and substance use disorder are greatly needed. Kappa opioid receptors are expressed in the central nervous system, including areas implicated in analgesia and affective state. Although kappa opioid receptor agonists share the antinociceptive effects of mu opioid receptor agonists, they also tend to produce negative affective states. In contrast, selective kappa opioid receptor antagonists have antidepressant- and anxiolytic-like effects, stimulating interest in their therapeutic potential. The prototypical kappa opioid receptor antagonists (e.g., norBNI, JDTic) have an exceptionally long duration of action that complicates their use in humans, particularly in tests to establish safety. This study was designed to test dose- and time-course effects of novel kappa opioid receptor antagonists with the goal of identifying short-acting lead compounds for future medication development.
METHODS
We screened 2 novel, highly selective kappa opioid receptor antagonists (CYM-52220 and CYM-52288) with oral efficacy in the warm water tail flick assay in rats to determine initial dose and time course effects. For comparison, we tested existing kappa opioid receptor antagonists JDTic and LY-2456302 (also known as CERC-501 or JNJ-67953964).
RESULTS
In the tail flick assay, the rank order of duration of action for the antagonists was LY-2456302 < CYM-52288 < CYM-52220 << JDTic. Furthermore, LY-2456302 blocked the depressive (anhedonia-producing) effects of the kappa opioid receptor agonist U50,488 in the intracranial self-stimulation paradigm, albeit at a higher dose than that needed for analgesic blockade in the tail flick assay.
CONCLUSIONS
These results suggest that structurally diverse kappa opioid receptor antagonists can have short-acting effects and that LY-2456302 reduces anhedonia as measured in the intracranial self-stimulation test.

Identifiants

pubmed: 31613314
pii: 5587707
doi: 10.1093/ijnp/pyz054
pmc: PMC7145521
doi:

Substances chimiques

7-hydroxy-N-(1-((4-(3-hydroxyphenyl)-3,4-dimethyl-1-piperidinyl)methyl)-2-methylpropyl)-1,2,3,4-tetrahydro-3-isoquinolinecarboxamide 0
Analgesics, Non-Narcotic 0
Anti-Anxiety Agents 0
Antidepressive Agents 0
Benzamides 0
Narcotic Antagonists 0
Piperidines 0
Pyrrolidines 0
Receptors, Opioid, kappa 0
Tetrahydroisoquinolines 0
3,4-Dichloro-N-methyl-N-(2-(1-pyrrolidinyl)-cyclohexyl)-benzeneacetamide, (trans)-Isomer 67198-13-4
Aticaprant DE4G8X55F5

Types de publication

Journal Article Research Support, N.I.H., Extramural

Langues

eng

Sous-ensembles de citation

IM

Pagination

735-745

Subventions

Organisme : NIMH NIH HHS
ID : R01 MH063266
Pays : United States

Informations de copyright

© The Author(s) 2019. Published by Oxford University Press on behalf of CINP.

Références

Neuropsychopharmacology. 2018 Mar;43(4):838-850
pubmed: 28589966
Brain Res. 2010 Feb 16;1314:44-55
pubmed: 19716811
Neuropsychopharmacology. 2013 Jul;38(8):1585-97
pubmed: 23446450
J Neurosci. 1999 Mar 1;19(5):1804-13
pubmed: 10024364
Neuropsychopharmacology. 2018 Mar;43(4):928
pubmed: 29422497
Biol Psychiatry. 2011 Oct 15;70(8):744-53
pubmed: 21757186
J Clin Pharmacol. 2014 Sep;54(9):968-78
pubmed: 24619932
Nat Rev Drug Discov. 2018 Dec 28;18(1):82-84
pubmed: 30591715
Neuropsychopharmacology. 2015 May;40(6):1448-55
pubmed: 25518754
Psychopharmacology (Berl). 2002 Mar;160(2):170-81
pubmed: 11875635
Mol Cell Neurosci. 1994 Apr;5(2):124-44
pubmed: 8032682
J Pharmacol Exp Ther. 2003 Apr;305(1):323-30
pubmed: 12649385
Psychopharmacology (Berl). 2004 Nov;176(2):204-13
pubmed: 15112031
Bioorg Med Chem Lett. 2014 May 1;24(9):2021-32
pubmed: 24690494
J Pharmacol Exp Ther. 2007 Dec;323(3):838-45
pubmed: 17823306
Arch Int Pharmacodyn Ther. 1992 Mar-Apr;316:30-42
pubmed: 1326932
Life Sci. 1982 Oct 18-25;31(16-17):1781-4
pubmed: 6130439
Pharmacol Ther. 2019 Sep;201:51-76
pubmed: 31051197
Psychopharmacology (Berl). 2005 Nov;183(1):118-26
pubmed: 16184376
J Neurosci. 2016 May 25;36(21):5748-62
pubmed: 27225765
J Med Chem. 2013 Mar 28;56(6):2178-95
pubmed: 23360448
J Neurosci. 2012 Dec 5;32(49):17582-96
pubmed: 23223282
Bioorg Med Chem Lett. 2009 Apr 15;19(8):2289-94
pubmed: 19282177
Neuropharmacology. 2012 Jan;62(1):167-76
pubmed: 21736885
Biol Psychiatry. 2014 Aug 1;76(3):213-22
pubmed: 24090794
Neuropsychopharmacology. 2008 Feb;33(3):643-52
pubmed: 17473837
Psychopharmacology (Berl). 2004 Apr;172(4):463-70
pubmed: 14727002
Depress Anxiety. 2016 Oct;33(10):895-906
pubmed: 27699938
J Med Chem. 2011 Dec 8;54(23):8000-12
pubmed: 21958337
J Med Chem. 2019 Feb 28;62(4):1761-1780
pubmed: 30707578
Eur J Pharmacol. 2007 Aug 13;569(1-2):84-9
pubmed: 17568579
Psychopharmacology (Berl). 2010 Jun;210(2):241-52
pubmed: 20372879
J Neurosci. 2001 Sep 15;21(18):7397-403
pubmed: 11549750
J Neurosci. 2002 Dec 15;22(24):10883-90
pubmed: 12486182
J Neurosci. 2016 Sep 21;36(38):9937-48
pubmed: 27656031
Eur J Pharmacol. 2004 Oct 6;501(1-3):111-9
pubmed: 15464069
Am J Psychiatry. 2016 May 1;173(5):499-508
pubmed: 26869247
Trends Neurosci. 1995 Jan;18(1):22-9
pubmed: 7535487
Behav Pharmacol. 2015 Oct;26(7 Spec No):654-63
pubmed: 26110224
Neuropharmacology. 2014 Feb;77:131-44
pubmed: 24071566
BMC Pharmacol. 2012 May 29;12:5
pubmed: 22642416
Psychopharmacology (Berl). 2013 Mar;226(2):381-92
pubmed: 23135605
Brain Res. 2010 Feb 16;1314:56-73
pubmed: 19782055
Cephalalgia. 2017 Jul;37(8):780-794
pubmed: 28376659
Behav Pharmacol. 2014 Sep;25(5-6):473-92
pubmed: 25083570
Nat Protoc. 2007;2(11):2987-95
pubmed: 18007634
Psychopharmacology (Berl). 2010 Jun;210(2):137-47
pubmed: 20401607
Mol Pharmacol. 2011 Nov;80(5):920-9
pubmed: 21832171
Neuropsychopharmacology. 2015 Aug;40(9):2057-8
pubmed: 26174493
Science. 1986 Aug 15;233(4765):774-6
pubmed: 3016896

Auteurs

Sarah Page (S)

Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, MA.

Maria M Mavrikaki (MM)

Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, MA.

Tania Lintz (T)

Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, MA.

Daniel Puttick (D)

Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, MA.

Edward Roberts (E)

Department of Molecular Medicine, The Scripps Research Institute, La Jolla, CA.

Hugh Rosen (H)

Department of Molecular Medicine, The Scripps Research Institute, La Jolla, CA.

F Ivy Carroll (FI)

Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, MA.

William A Carlezon (WA)

Department of Psychiatry, Harvard Medical School, McLean Hospital, Belmont, MA.

Elena H Chartoff (EH)

Research Triangle Institute, Research Triangle Park, NC.

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Classifications MeSH