Drug-induced chromatin accessibility changes associate with sensitivity to liver tumor promotion.
Animals
Chromatin
/ drug effects
Chromatin Assembly and Disassembly
/ drug effects
Computational Biology
Epigenesis, Genetic
/ drug effects
Gene Expression Profiling
/ methods
Gene Regulatory Networks
Humans
Liver Neoplasms
/ chemically induced
Male
Mice
Phenobarbital
/ adverse effects
Wnt Signaling Pathway
/ drug effects
Journal
Life science alliance
ISSN: 2575-1077
Titre abrégé: Life Sci Alliance
Pays: United States
ID NLM: 101728869
Informations de publication
Date de publication:
10 2019
10 2019
Historique:
received:
20
06
2019
revised:
26
09
2019
accepted:
26
09
2019
entrez:
17
10
2019
pubmed:
17
10
2019
medline:
1
7
2020
Statut:
epublish
Résumé
Liver cancer susceptibility varies amongst humans and between experimental animal models because of multiple genetic and epigenetic factors. The molecular characterization of such susceptibilities has the potential to enhance cancer risk assessment of xenobiotic exposures and disease prevention strategies. Here, using DNase I hypersensitivity mapping coupled with transcriptomic profiling, we investigate perturbations in
Identifiants
pubmed: 31615920
pii: 2/5/e201900461
doi: 10.26508/lsa.201900461
pmc: PMC6795216
pii:
doi:
Substances chimiques
Chromatin
0
Phenobarbital
YQE403BP4D
Types de publication
Comparative Study
Journal Article
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Cancer Research UK
ID : 10822
Pays : United Kingdom
Organisme : Cancer Research UK
ID : C4639/A10822
Pays : United Kingdom
Informations de copyright
© 2019 Vitobello et al.
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