Dietary salt promotes cognitive impairment through tau phosphorylation.


Journal

Nature
ISSN: 1476-4687
Titre abrégé: Nature
Pays: England
ID NLM: 0410462

Informations de publication

Date de publication:
10 2019
Historique:
received: 31 05 2018
accepted: 24 09 2019
pubmed: 28 10 2019
medline: 28 3 2020
entrez: 25 10 2019
Statut: ppublish

Résumé

Dietary habits and vascular risk factors promote both Alzheimer's disease and cognitive impairment caused by vascular factors

Identifiants

pubmed: 31645758
doi: 10.1038/s41586-019-1688-z
pii: 10.1038/s41586-019-1688-z
pmc: PMC7380655
mid: NIHMS1605533
doi:

Substances chimiques

Sodium Chloride, Dietary 0
tau Proteins 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

686-690

Subventions

Organisme : NINDS NIH HHS
ID : R01 NS095441
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS109588
Pays : United States
Organisme : NINDS NIH HHS
ID : R37 NS089323
Pays : United States
Organisme : NIH HHS
ID : 1R01-NS095441
Pays : United States

Commentaires et corrections

Type : CommentIn
Type : ErratumIn

Références

Scarmeas, N., Anastasiou, C. A. & Yannakoulia, M. Nutrition and prevention of cognitive impairment. Lancet Neurol. 17, 1006–1015 (2018).
pubmed: 30244829 doi: 10.1016/S1474-4422(18)30338-7
van de Rest, O., Berendsen, A. A., Haveman-Nies, A. & de Groot, L. C. Dietary patterns, cognitive decline, and dementia: a systematic review. Adv. Nutr. 6, 154–168 (2015).
pubmed: 25770254 pmcid: 4352174 doi: 10.3945/an.114.007617
Kendig, M. D. & Morris, M. J. Reviewing the effects of dietary salt on cognition: mechanisms and future directions. Asia Pac. J. Clin. Nutr. 28, 6–14 (2019).
pubmed: 30896408
De Strooper, B. & Karran, E. The cellular phase of Alzheimer’s disease. Cell 164, 603–615 (2016).
pubmed: 26871627 doi: 10.1016/j.cell.2015.12.056
Nation, D. A. et al. Pulse pressure in relation to tau-mediated neurodegeneration, cerebral amyloidosis, and progression to dementia in very old adults. JAMA Neurol. 72, 546–553 (2015).
pubmed: 25822631 pmcid: 4428938 doi: 10.1001/jamaneurol.2014.4477
Kim, H. J. et al. Assessment of extent and role of tau in subcortical vascular cognitive impairment using 18F-AV1451 positron emission tomography imaging. JAMA Neurol. 75, 999–1007 (2018).
pubmed: 29799981 pmcid: 6142932 doi: 10.1001/jamaneurol.2018.0975
Faraco, G. et al. Dietary salt promotes neurovascular and cognitive dysfunction through a gut-initiated TH17 response. Nat. Neurosci. 21, 240–249 (2018).
pubmed: 29335605 pmcid: 6207376 doi: 10.1038/s41593-017-0059-z
Fiocco, A. J. et al. Sodium intake and physical activity impact cognitive maintenance in older adults: the NuAge Study. Neurobiol. Aging 33, 829.e821–829.e28, (2012).
doi: 10.1016/j.neurobiolaging.2011.07.004
Gardener, H., Rundek, T., Wright, C. B., Elkind, M. S. & Sacco, R. L. Dietary sodium and risk of stroke in the Northern Manhattan study. Stroke 43, 1200–1205 (2012).
pubmed: 22499576 pmcid: 3347890 doi: 10.1161/STROKEAHA.111.641043
Blumenthal, J. A. et al. Lifestyle and neurocognition in older adults with cognitive impairments: a randomized trial. Neurology 92, e212–e223 (2019).
pubmed: 30568005 pmcid: 6340382 doi: 10.1212/WNL.0000000000006784
Heye, A. K. et al. Blood pressure and sodium: association with MRI markers in cerebral small vessel disease. J. Cereb. Blood Flow Metab. 36, 264–274 (2016).
pubmed: 25899292 pmcid: 4758556 doi: 10.1038/jcbfm.2015.64
Iadecola, C. The pathobiology of vascular dementia. Neuron 80, 844–866 (2013).
pubmed: 24267647 doi: 10.1016/j.neuron.2013.10.008
Sweeney, M. D. et al. Vascular dysfunction—the disregarded partner of Alzheimer’s disease. Alzheimers Dement. 15, 158–167 (2019).
pubmed: 30642436 pmcid: 6338083 doi: 10.1016/j.jalz.2018.07.222
Shi, Y. et al. Cerebral blood flow in small vessel disease: a systematic review and meta-analysis. J. Cereb. Blood Flow Metab. 36, 1653–1667 (2016).
pubmed: 27496552 pmcid: 5076792 doi: 10.1177/0271678X16662891
Marshall, R. S. et al. Recovery of brain function during induced cerebral hypoperfusion. Brain 124, 1208–1217 (2001).
pubmed: 11353736 doi: 10.1093/brain/124.6.1208
Wang, Y. & Mandelkow, E. Tau in physiology and pathology. Nat. Rev. Neurosci. 17, 5–21 (2016).
pubmed: 26631930 doi: 10.1038/nrn.2015.1
Zhao, Y. et al. Sodium intake regulates glucose homeostasis through the PPARδ/adiponectin-mediated SGLT2 pathway. Cell Metab. 23, 699–711 (2016).
pubmed: 27053360 doi: 10.1016/j.cmet.2016.02.019
Min, S. W. et al. Critical role of acetylation in tau-mediated neurodegeneration and cognitive deficits. Nat. Med. 21, 1154–1162 (2015).
pubmed: 26390242 pmcid: 4598295 doi: 10.1038/nm.3951
Iadecola, C. et al. SOD1 rescues cerebral endothelial dysfunction in mice overexpressing amyloid precursor protein. Nat. Neurosci. 2, 157–161 (1999).
pubmed: 10195200 doi: 10.1038/5715
Faraco, G. et al. Perivascular macrophages mediate the neurovascular and cognitive dysfunction associated with hypertension. J. Clin. Invest. 126, 4674–4689 (2016).
pubmed: 27841763 pmcid: 5127678 doi: 10.1172/JCI86950
Arendt, T., Stieler, J. T. & Holzer, M. Tau and tauopathies. Brain Res. Bull. 126, 238–292 (2016).
pubmed: 27615390 doi: 10.1016/j.brainresbull.2016.08.018
Lee, M. S. et al. Neurotoxicity induces cleavage of p35 to p25 by calpain. Nature 405, 360–364 (2000).
pubmed: 10830966 doi: 10.1038/35012636
Patrick, G. N. et al. Conversion of p35 to p25 deregulates Cdk5 activity and promotes neurodegeneration. Nature 402, 615–622 (1999).
pubmed: 10604467 doi: 10.1038/45159
Austin, S. A. & Katusic, Z. S. Loss of endothelial nitric oxide synthase promotes p25 generation and tau phosphorylation in a murine model of Alzheimer’s disease. Circ. Res. 119, 1128–1134 (2016).
pubmed: 27601478 pmcid: 5085902 doi: 10.1161/CIRCRESAHA.116.309686
Bibb, J. A. et al. Phosphorylation of DARPP-32 by Cdk5 modulates dopamine signalling in neurons. Nature 402, 669–671 (1999).
pubmed: 10604473 doi: 10.1038/45251
Shukla, V. et al. A truncated peptide from p35, a Cdk5 activator, prevents Alzheimer’s disease phenotypes in model mice. FASEB J. 27, 174–186 (2013).
pubmed: 23038754 pmcid: 3528323 doi: 10.1096/fj.12-217497
Kimura, T. et al. Isomerase Pin1 stimulates dephosphorylation of tau protein at cyclin-dependent kinase (Cdk5)-dependent Alzheimer phosphorylation sites. J. Biol. Chem. 288, 7968–7977 (2013).
pubmed: 23362255 pmcid: 3597833 doi: 10.1074/jbc.M112.433326
Ono, Y., Saido, T. C. & Sorimachi, H. Calpain research for drug discovery: challenges and potential. Nat. Rev. Drug Discov. 15, 854–876 (2016).
pubmed: 27833121 doi: 10.1038/nrd.2016.212
Etwebi, Z., Landesberg, G., Preston, K., Eguchi, S. & Scalia, R. Mechanistic role of the calcium-dependent protease calpain in the endothelial dysfunction induced by MPO (myeloperoxidase). Hypertension 71, 761–770 (2018).
pubmed: 29507101 doi: 10.1161/HYPERTENSIONAHA.117.10305
Qu, J. et al. S-Nitrosylation activates Cdk5 and contributes to synaptic spine loss induced by β-amyloid peptide. Proc. Natl Acad. Sci. USA 108, 14330–14335 (2011).
pubmed: 21844361 pmcid: 3161554 doi: 10.1073/pnas.1105172108
Iadecola, C. The neurovascular unit coming of age: a journey through neurovascular coupling in health and disease. Neuron 96, 17–42 (2017).
pubmed: 28957666 pmcid: 5657612 doi: 10.1016/j.neuron.2017.07.030
Yanamandra, K. et al. Anti-tau antibodies that block tau aggregate seeding in vitro markedly decrease pathology and improve cognition in vivo. Neuron 80, 402–414 (2013).
pubmed: 24075978 pmcid: 3924573 doi: 10.1016/j.neuron.2013.07.046
Powles, J. et al. Global, regional and national sodium intakes in 1990 and 2010: a systematic analysis of 24 h urinary sodium excretion and dietary surveys worldwide. BMJ Open 3, e003733 (2013).
pubmed: 24366578 pmcid: 3884590 doi: 10.1136/bmjopen-2013-003733
Hochrainer, K. et al. The ubiquitin ligase HERC3 attenuates NF-κB-dependent transcription independently of its enzymatic activity by delivering the RelA subunit for degradation. Nucleic Acids Res. 43, 9889–9904 (2015).
pubmed: 26476452 pmcid: 4787756
Shukla, V. et al. TFP5, a peptide inhibitor of aberrant and hyperactive Cdk5/p25, attenuates pathological phenotypes and restores synaptic function in CK-p25Tg mice. J. Alzheimers Dis. 56, 335–349 (2017).
pubmed: 28085018 doi: 10.3233/JAD-160916
Faraco, G. et al. Circulating endothelin-1 alters critical mechanisms regulating cerebral microcirculation. Hypertension 62, 759–766 (2013).
pubmed: 23959559 doi: 10.1161/HYPERTENSIONAHA.113.01761
Kober, F. et al. High-resolution myocardial perfusion mapping in small animals in vivo by spin-labeling gradient-echo imaging. Magn. Reson. Med. 51, 62–67 (2004).
pubmed: 14705046 doi: 10.1002/mrm.10676
Petry, F. R. et al. Specificity of anti-tau antibodies when analyzing mice models of Alzheimer’s disease: problems and solutions. PLoS One 9, e94251 (2014).
pubmed: 24788298 pmcid: 4008431 doi: 10.1371/journal.pone.0094251
Faraco, G. et al. Hypertension enhances Aβ-induced neurovascular dysfunction, promotes β-secretase activity, and leads to amyloidogenic processing of APP. J. Cereb. Blood Flow Metab. 36, 241–252 (2016).
pubmed: 25920959 pmcid: 4758560
Voit, A. et al. Reducing sarcolipin expression mitigates Duchenne muscular dystrophy and associated cardiomyopathy in mice. Nat. Commun. 8, 1068 (2017).
pubmed: 29051551 pmcid: 5648780 doi: 10.1038/s41467-017-01146-7
Liu, W. et al. Metabolic stress-induced cardiomyopathy is caused by mitochondrial dysfunction due to attenuated Erk5 signaling. Nat. Commun. 8, 494 (2017).
pubmed: 28887535 pmcid: 5591279 doi: 10.1038/s41467-017-00664-8
Forrester, M. T., Foster, M. W., Benhar, M. & Stamler, J. S. Detection of protein S-nitrosylation with the biotin-switch technique. Free Radic. Biol. Med. 46, 119–126 (2009).
pubmed: 18977293 doi: 10.1016/j.freeradbiomed.2008.09.034
Cohen, S. J. & Stackman, R. W. Jr Assessing rodent hippocampal involvement in the novel object recognition task. A review. Behav. Brain Res. 285, 105–117 (2015).
pubmed: 25169255 doi: 10.1016/j.bbr.2014.08.002
Grayson, B. et al. Assessment of disease-related cognitive impairments using the novel object recognition (NOR) task in rodents. Behav. Brain Res. 285, 176–193 (2015).
pubmed: 25447293 doi: 10.1016/j.bbr.2014.10.025
O’Leary, T. P. & Brown, R. E. Optimization of apparatus design and behavioral measures for the assessment of visuo-spatial learning and memory of mice on the Barnes maze. Learn. Mem. 20, 85–96 (2013).
pubmed: 23322557 doi: 10.1101/lm.028076.112

Auteurs

Giuseppe Faraco (G)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA. gif2004@med.cornell.edu.

Karin Hochrainer (K)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.

Steven G Segarra (SG)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.

Samantha Schaeffer (S)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.

Monica M Santisteban (MM)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.

Ajay Menon (A)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.

Hong Jiang (H)

Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University, St. Louis, MO, USA.

David M Holtzman (DM)

Department of Neurology, Hope Center for Neurological Disorders, Knight Alzheimer's Disease Research Center, Washington University, St. Louis, MO, USA.

Josef Anrather (J)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA.

Costantino Iadecola (C)

Feil Family Brain and Mind Research Institute, Weill Cornell Medicine, New York, NY, USA. coi2001@med.cornell.edu.

Articles similaires

[Redispensing of expensive oral anticancer medicines: a practical application].

Lisanne N van Merendonk, Kübra Akgöl, Bastiaan Nuijen
1.00
Humans Antineoplastic Agents Administration, Oral Drug Costs Counterfeit Drugs

Smoking Cessation and Incident Cardiovascular Disease.

Jun Hwan Cho, Seung Yong Shin, Hoseob Kim et al.
1.00
Humans Male Smoking Cessation Cardiovascular Diseases Female
Humans United States Aged Cross-Sectional Studies Medicare Part C
1.00
Humans Yoga Low Back Pain Female Male

Classifications MeSH