The early detection of atherosclerosis in type 1 diabetes: why, how and what to do about it.
atherosclerosis
biomarkers
coronary artery calcification
intima–media thickness
risk factors
type 1 diabetes
vascular function
Journal
Cardiovascular endocrinology & metabolism
ISSN: 2574-0954
Titre abrégé: Cardiovasc Endocrinol Metab
Pays: United States
ID NLM: 101730894
Informations de publication
Date de publication:
Mar 2019
Mar 2019
Historique:
received:
19
12
2018
accepted:
22
02
2019
entrez:
25
10
2019
pubmed:
28
10
2019
medline:
28
10
2019
Statut:
epublish
Résumé
The major cause of morbidity and often premature mortality in people with type I diabetes (T1D) is cardiovascular disease owing to accelerated atherosclerosis. We review publications relating to the rationale behind, and clinical tests for, detecting and treating early atherosclerosis in people with T1D. Currently available tools for atherosclerosis assessment include risk equations using vascular risk factors, arterial intima-media thickness, the ankle-brachial index, coronary artery calcification and angiography, and for more advanced lesions, intravascular ultrasound and optical coherence tomography. Evolving research tools include risk equations incorporating novel clinical, biochemical and molecular tests; vascular MRI and molecular imaging. As yet there is little information available to quantify early atherosclerosis. With better means to control the vascular risk factors, such as hypertension, dyslipidaemia and glycaemic control, and emerging therapies to control novel risk factors, further epidemiologic and clinical trials are merited to facilitate the translation into clinical practice of robust means to detect, monitor and treat early atherosclerosis in those with T1D.
Identifiants
pubmed: 31646294
doi: 10.1097/XCE.0000000000000169
pmc: PMC6739889
doi:
Types de publication
Journal Article
Review
Langues
eng
Pagination
14-27Informations de copyright
Copyright © 2019 Wolters Kluwer Health, Inc. All rights reserved.
Déclaration de conflit d'intérêts
A.J.J. is supported by a NHMRC Practitioner Fellowship and is a Sydney Medical School Foundation Fellow. A.J.J. and D.N.O. were investigators on the REMOVAL Trial, which was funded by JDRF International/Canada and Australia, and for which metformin and matching placebo was provided free by Merck KGaA (Germany), and EndoPAT equipment and non-financial support was received from Itamar Medical (Israel). A.J.J. is also an investigator on the DCCT/EDIC trial. A.J.J. and D.N.O. have received peer-reviewed research grants from Medtronic, Sanofi-Aventis and Glen-Sys and are on advisory boards for Abbott (Diabetes Devices, Australia), Sanofi-Aventis (Diabetes, Australia) and Medtronic Australia. A.J.J. has also received research grants or product from Mylan. For the remaining author, there are no conflicts of interest.
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