Outcome of cutaneous psoriasis in hepatitis C virus-infected patients treated with Direct-Acting Antiviral therapy.


Journal

Journal of viral hepatitis
ISSN: 1365-2893
Titre abrégé: J Viral Hepat
Pays: England
ID NLM: 9435672

Informations de publication

Date de publication:
03 2020
Historique:
received: 28 06 2019
revised: 06 09 2019
accepted: 06 10 2019
pubmed: 8 11 2019
medline: 26 3 2021
entrez: 8 11 2019
Statut: ppublish

Résumé

Apart from chronic liver disease, hepatitis C virus (HCV) may be responsible for several extra-hepatic manifestations. Its involvement in psoriasis development is still controversial. The aim of this study was to evaluate the possible effect of anti-HCV direct-acting antiviral (DAA) treatment on cutaneous psoriasis. Thirty-seven consecutive HCV patients with cutaneous psoriasis underwent efficacious DAA treatment, and all of them were efficiently cured as shown by HCV RNA negativity 24 weeks after stopping therapy (PT24W). An expert dermatologist evaluated the skin lesions at baseline, end of treatment (EOT) and PT24W using the psoriasis area severity index (PASI) scoring system. The impact on quality of life was measured with the Dermatologic Quality of Life Index (DLQI). Six patients had a stable disease throughout the study period, whereas 31/37 patients (83.8%) showed a significant improvement of the skin lesions at EOT (P < .0001). However, 24 of these 31 patients (77.4%) had a dramatic worsening of the psoriatic lesions at PT24W compared with EOT (P < .001), with lesion severity comparable to baseline. The outcome of psoriasis during and after treatment was independent of baseline PASI score, age, sex, HCV genotype, liver disease stage and of the presence of arterial hypertension, diabetes and autoimmune diseases. In conclusion, DAA-based HCV cure has only a transient effect on skin lesions of patients with concomitant cutaneous psoriasis.

Identifiants

pubmed: 31698529
doi: 10.1111/jvh.13230
doi:

Substances chimiques

Antiviral Agents 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

333-337

Informations de copyright

© 2019 John Wiley & Sons Ltd.

Références

Negro F. Facts and fictions of HCV and comorbidities: steatosis, diabetes mellitus, and cardiovascular diseases. J Hepatol. 2014;61(1 Suppl):S69-78.
Zignego AL, Ramos-Casals M, Ferri C, et al. International therapeutic guidelines for patients with HCV-related extrahepatic disorders. A multidisciplinary expert statement. Autoimmun Rev. 2017;16(5):523-541.
Sayiner M, Golabi P, Farhat F, Younossi ZM. Dermatologic manifestations of chronic hepatitis C infection. Clin Liver Dis. 2017;21(3):555-564.
Imafuku S, Nakayama J. Profile of patients with psoriasis associated with hepatitis C virus infection. J Dermatol. 2013;40(6):428-433.
Afshar M, Martinez AD, Gallo RL, Hata TR. Induction and exacerbation of psoriasis with Interferon-alpha therapy for hepatitis C: A review and analysis of 36 cases. J Eur Acad Dermatol Venereol. 2013;27(6):771-778.
Simpson MJ, Chow C, Morgenstern H, Luger TA, Ellis CN. Comparison of three methods for measuring psoriasis severity in clinical studies (Part 2 of 2): use of quality of life to assess construct validity of the Lattice System Physician's Global Assessment, Psoriasis Area and Severity Index and Static Physician's Global Assessment. J Eur Acad Dermatol Venereol. 2015;29(7):1415-1420.
Sasaki R, Meyer K, Moriyama M, et al. Rapid hepatitis C virus clearance by antivirals correlates with immune status of infected patients. J Med Virol. 2019;91(3):411-418.
Orr C, Aartun J, Masur H, Kottilil S, Meissner EG. Characterization of changes in intrahepatic immune cell populations during HCV treatment with sofosbuvir and ribavirin. J Viral Hepat. 2019;26(3):323-328.
Holmes JA, Carlton-Smith C, Kim AY, et al. Dynamic changes in innate immune responses during direct-acting antiviral therapy for HCV infection. J Viral Hepat. 2019;26(3):362-372.

Auteurs

Irene Cacciola (I)

Division of Clinical and Molecular Hepatology, University of Messina, Messina, Italy.
Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.

Francesco Borgia (F)

Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Division of Dermatology, University of Messina, Messina, Italy.

Roberto Filomia (R)

Division of Clinical and Molecular Hepatology, University of Messina, Messina, Italy.

Concetta Pitrone (C)

Division of Clinical and Molecular Hepatology, University of Messina, Messina, Italy.
Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.

Maria Stella Franzè (MS)

Division of Clinical and Molecular Hepatology, University of Messina, Messina, Italy.
Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.

Angela Alibrandi (A)

Department of Economics, Unit of Statistical and Mathematical Sciences, University of Messina, Messina, Italy.

Giovanni Squadrito (G)

Division of Clinical and Molecular Hepatology, University of Messina, Messina, Italy.
Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.

Claudio Guarneri (C)

Division of Dermatology, University of Messina, Messina, Italy.
Department of Biomedical and Dental Sciences and Morphofunctional Imaging, University of Messina, Messina, Italy.

Valeria Papaianni (V)

Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Division of Dermatology, University of Messina, Messina, Italy.

Serafinella Patrizia Cannavò (SP)

Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.
Division of Dermatology, University of Messina, Messina, Italy.

Giovanni Raimondo (G)

Division of Clinical and Molecular Hepatology, University of Messina, Messina, Italy.
Department of Clinical and Experimental Medicine, University of Messina, Messina, Italy.

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