Left ventricular clefts - incidental finding or pathologic sign of Wilson's disease?


Journal

Orphanet journal of rare diseases
ISSN: 1750-1172
Titre abrégé: Orphanet J Rare Dis
Pays: England
ID NLM: 101266602

Informations de publication

Date de publication:
07 11 2019
Historique:
received: 21 08 2019
accepted: 22 10 2019
entrez: 9 11 2019
pubmed: 9 11 2019
medline: 7 7 2020
Statut: epublish

Résumé

Wilson's disease is an inherited autosomal recessive multi-systemic disorder characterized by reduced excretion and consequently excessive accumulation of copper in different organs, such as the heart. In a prospective controlled trial, which is the largest to date, we evaluated 61 patients with Wilson's disease, age- and sex-matched to 61 healthy patients, for cardiac manifestation using cardiac magnetic resonance imaging. Patients were under stable disease and had no signs of heart failure at the time of examination. We detected a left ventricular cleft, an invagination penetrating more than 50% wall thickness of the adjoining compact myocardium in diastole, in 20% of the patients (12 out of 61) compared to 5% among control patients (3 out of 61, p = 0.013). No correlation between the incidence of cleft and a certain genotype of Wilson's disease was found. All described cases were incidental findings and none of the patients showed other signs of cardiac involvement. To conclude, the results of this study suggests that the increased occurrence of left ventricular clefts is due to Wilson's disease. Large studies with a long observation period are needed for further evaluation.

Sections du résumé

BACKGROUND
Wilson's disease is an inherited autosomal recessive multi-systemic disorder characterized by reduced excretion and consequently excessive accumulation of copper in different organs, such as the heart.
RESULTS
In a prospective controlled trial, which is the largest to date, we evaluated 61 patients with Wilson's disease, age- and sex-matched to 61 healthy patients, for cardiac manifestation using cardiac magnetic resonance imaging. Patients were under stable disease and had no signs of heart failure at the time of examination. We detected a left ventricular cleft, an invagination penetrating more than 50% wall thickness of the adjoining compact myocardium in diastole, in 20% of the patients (12 out of 61) compared to 5% among control patients (3 out of 61, p = 0.013). No correlation between the incidence of cleft and a certain genotype of Wilson's disease was found. All described cases were incidental findings and none of the patients showed other signs of cardiac involvement.
CONCLUSIONS
To conclude, the results of this study suggests that the increased occurrence of left ventricular clefts is due to Wilson's disease. Large studies with a long observation period are needed for further evaluation.

Identifiants

pubmed: 31699127
doi: 10.1186/s13023-019-1238-7
pii: 10.1186/s13023-019-1238-7
pmc: PMC6836362
doi:

Types de publication

Controlled Clinical Trial Letter

Langues

eng

Sous-ensembles de citation

IM

Pagination

244

Références

Circ Cardiovasc Imaging. 2014 Mar;7(2):259-64
pubmed: 24508667
Orphanet J Rare Dis. 2019 Jan 28;14(1):22
pubmed: 30691535
J Am Coll Cardiol. 2018 Dec 4;72(22):2808-2809
pubmed: 30497569
J Am Coll Cardiol. 2006 Dec 19;48(12):2518-23
pubmed: 17174192
Circ Cardiovasc Imaging. 2012 Jul;5(4):431-2
pubmed: 22811415

Auteurs

Kun Zhang (K)

Department of Internal Medicine and Cardiology, Charité - Universitätsmedizin Berlin, Campus Virchow-Klinikum, Augustenburger Platz 1, 13353, Berlin, Germany. kun.zhang@charite.de.
Berlin Institute of Health (BIH), Berlin, Germany. kun.zhang@charite.de.
DZHK (German Centre for Cardiovascular Research), partner site Berlin, Berlin, Germany. kun.zhang@charite.de.

Ulrike Reuner (U)

Department of Neurology, Technische Universität Dresden, University Hospital, Dresden, Germany.

Marie Weidauer (M)

Technische Universität Dresden, Heart Center, University Hospital, Clinic of Internal Medicine and Cardiology, Dresden, Germany.

Uwe Speiser (U)

Technische Universität Dresden, Heart Center, University Hospital, Clinic of Internal Medicine and Cardiology, Dresden, Germany.

Karim Ibrahim (K)

Department of Cardiology, Technische Universität Dresden, Klinikum Chemnitz, Chemnitz, Germany.

Marian Christoph (M)

Department of Cardiology, Technische Universität Dresden, Klinikum Chemnitz, Chemnitz, Germany.

Frank R Heinzel (FR)

Department of Internal Medicine and Cardiology, Charité - Universitätsmedizin Berlin, Campus Virchow-Klinikum, Augustenburger Platz 1, 13353, Berlin, Germany.
DZHK (German Centre for Cardiovascular Research), partner site Berlin, Berlin, Germany.

Burkert Pieske (B)

Department of Internal Medicine and Cardiology, Charité - Universitätsmedizin Berlin, Campus Virchow-Klinikum, Augustenburger Platz 1, 13353, Berlin, Germany.
Berlin Institute of Health (BIH), Berlin, Germany.
DZHK (German Centre for Cardiovascular Research), partner site Berlin, Berlin, Germany.

Felix M Heidrich (FM)

Technische Universität Dresden, Heart Center, University Hospital, Clinic of Internal Medicine and Cardiology, Dresden, Germany.

Silvio Quick (S)

Department of Cardiology, Technische Universität Dresden, Klinikum Chemnitz, Chemnitz, Germany.

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