Association of Plasma Marker of Oxidized Lipid with Histologic Plaque Instability in Patients with Peripheral Artery Disease.


Journal

Annals of vascular surgery
ISSN: 1615-5947
Titre abrégé: Ann Vasc Surg
Pays: Netherlands
ID NLM: 8703941

Informations de publication

Date de publication:
Jul 2020
Historique:
received: 13 08 2019
revised: 05 10 2019
accepted: 01 11 2019
pubmed: 11 11 2019
medline: 21 10 2020
entrez: 11 11 2019
Statut: ppublish

Résumé

The association between oxidized low-density lipoprotein (OxLDL) and plaque instability in coronary and carotid artery disease is well established. However, the association between OxLDL and the histologic changes of plaque in peripheral artery disease has not been clearly elucidated. This study aims to investigate the association between plasma OxLDL and histologic plaque instability in patients with peripheral artery disease. Prospectively obtained plaques from 48 patients who underwent endovascular atherectomy (n = 20), surgical endarterectomy (n = 9), or bypass surgery (n = 19) for treatment of atherosclerotic femoropopliteal artery disease were evaluated for histologic fibrosis, sclerosis, calcification, necrosis, cholesterol cleft, and foamy macrophages using hematoxylin and eosin, oil red O, and immunohistochemical staining. Unstable plaques were defined as plaques that were positive for foamy macrophages and with lipid content of more than 10% of the total plaque area. Plasma OxLDL levels were measured using an enzyme-linked immunosorbent assay (Mercodia AB, Uppsala, Sweden). Of the 48 patients, 26 (54%) had unstable plaques. The unstable plaque group was younger, had fewer angiographic total occlusions, less calcification, and more CD68-positive and LOX-1-positive cells than the stable plaque group. Plasma OxLDL levels were significantly higher in the unstable plaque group than in the stable plaque group (57.4 ± 13.9 vs. 47.2 ± 13.6 U/L, P = 0.014). Multivariate analysis revealed that plasma OxLDL level, smoking, angiographic nontotal occlusion, and statin nonuse were independent predictors of unstable plaque. Among patients with peripheral artery disease, the histologic instability of femoropopliteal plaque was independently associated with high plasma OxLDL, smoking, nontotal occlusion, and statin nonuse. Further large-scale studies are necessary to evaluate the role of noninvasive OxLDL measurement for predicting plaque instability and future adverse vascular event.

Sections du résumé

BACKGROUND BACKGROUND
The association between oxidized low-density lipoprotein (OxLDL) and plaque instability in coronary and carotid artery disease is well established. However, the association between OxLDL and the histologic changes of plaque in peripheral artery disease has not been clearly elucidated. This study aims to investigate the association between plasma OxLDL and histologic plaque instability in patients with peripheral artery disease.
METHODS METHODS
Prospectively obtained plaques from 48 patients who underwent endovascular atherectomy (n = 20), surgical endarterectomy (n = 9), or bypass surgery (n = 19) for treatment of atherosclerotic femoropopliteal artery disease were evaluated for histologic fibrosis, sclerosis, calcification, necrosis, cholesterol cleft, and foamy macrophages using hematoxylin and eosin, oil red O, and immunohistochemical staining. Unstable plaques were defined as plaques that were positive for foamy macrophages and with lipid content of more than 10% of the total plaque area. Plasma OxLDL levels were measured using an enzyme-linked immunosorbent assay (Mercodia AB, Uppsala, Sweden).
RESULTS RESULTS
Of the 48 patients, 26 (54%) had unstable plaques. The unstable plaque group was younger, had fewer angiographic total occlusions, less calcification, and more CD68-positive and LOX-1-positive cells than the stable plaque group. Plasma OxLDL levels were significantly higher in the unstable plaque group than in the stable plaque group (57.4 ± 13.9 vs. 47.2 ± 13.6 U/L, P = 0.014). Multivariate analysis revealed that plasma OxLDL level, smoking, angiographic nontotal occlusion, and statin nonuse were independent predictors of unstable plaque.
CONCLUSIONS CONCLUSIONS
Among patients with peripheral artery disease, the histologic instability of femoropopliteal plaque was independently associated with high plasma OxLDL, smoking, nontotal occlusion, and statin nonuse. Further large-scale studies are necessary to evaluate the role of noninvasive OxLDL measurement for predicting plaque instability and future adverse vascular event.

Identifiants

pubmed: 31706994
pii: S0890-5096(19)30956-2
doi: 10.1016/j.avsg.2019.11.004
pii:
doi:

Substances chimiques

Biomarkers 0
Lipoproteins, LDL 0
oxidized low density lipoprotein 0

Types de publication

Journal Article Multicenter Study

Langues

eng

Sous-ensembles de citation

IM

Pagination

554-565

Informations de copyright

Copyright © 2019 Elsevier Inc. All rights reserved.

Auteurs

Kichun Kim (K)

The Armed Forces Medical Command, Seongnam, South Korea.

Cheong Lim (C)

Department of Thoracic and Cardiovascular Surgery, Seoul National University Bundang Hospital, Seongnam, South Korea.

Gilhyang Kim (G)

Department of Pathology, Seoul National University Bundang Hospital, Seongnam, South Korea.

Jin-Haeng Chung (JH)

Department of Pathology, Seoul National University Bundang Hospital, Seongnam, South Korea.

Young-Seok Cho (YS)

Department of Internal Medicine, Seoul National University Bundang Hospital, Seongnam, South Korea. Electronic address: flammeus1@gmail.com.

Jun Hwan Cho (JH)

Department of Internal Medicine, Chung-Ang University Hospital, Seoul, South Korea.

Jae-Bin Seo (JB)

Department of Internal Medicine, Seoul National University-Seoul Metropolitan Government Boramae Medical Center, Seoul, South Korea.

Woo-Young Chung (WY)

Department of Internal Medicine, Seoul National University-Seoul Metropolitan Government Boramae Medical Center, Seoul, South Korea.

Se-Jin Oh (SJ)

Department of Cardiothoracic Surgery, Seoul Metropolitan Government-Seoul National University Boramae Medical Center, Seoul, South Korea.

Jae-Sung Choi (JS)

Department of Cardiothoracic Surgery, Seoul Metropolitan Government-Seoul National University Boramae Medical Center, Seoul, South Korea.

Jun-Sung Kim (JS)

Department of Thoracic and Cardiovascular Surgery, Seoul National University Bundang Hospital, Seongnam, South Korea.

Jin Joo Park (JJ)

Department of Internal Medicine, Seoul National University Bundang Hospital, Seongnam, South Korea.

Jung-Won Suh (JW)

Department of Internal Medicine, Seoul National University Bundang Hospital, Seongnam, South Korea.

Tae-Jin Youn (TJ)

Department of Internal Medicine, Seoul National University Bundang Hospital, Seongnam, South Korea.

In-Ho Chae (IH)

Department of Internal Medicine, Seoul National University Bundang Hospital, Seongnam, South Korea.

Dong-Ju Choi (DJ)

Department of Internal Medicine, Seoul National University Bundang Hospital, Seongnam, South Korea.

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Classifications MeSH