The anti-cancer effects of phenformin in thyroid cancer cell lines and in normal thyrocytes.

CXCL8 phenformin thyroid cancer tumor-microenvironment

Journal

Oncotarget
ISSN: 1949-2553
Titre abrégé: Oncotarget
Pays: United States
ID NLM: 101532965

Informations de publication

Date de publication:
05 Nov 2019
Historique:
received: 12 08 2019
accepted: 24 09 2019
entrez: 20 11 2019
pubmed: 20 11 2019
medline: 20 11 2019
Statut: epublish

Résumé

Phenformin is a biguanide drug which, besides the original anti-diabetic effect, also exerts anti-cancer effects. The aim of this study was to further characterize these latter in terms of both cell-viability and modulation of the secretion of the pro-tumorigenic chemokine CXCL8. Normal human thyrocytes in primary cultures (NHT) and thyroid cancer cell lines, TPC-1 and 8505C (RET/PTC and BRAFV600E mutated, respectively) were treated with increasing concentrations of phenformin at different times. Cell-viability was assessed by WST-1 and further characterized by AnnexinV/PI staining and cell proliferation colony-assay. CXCL8 levels were measured in cell supernatants. Phenformin reduced cell-viability in TPC-1 and 8505C and their ability to form colonies. In NHT cells, phenformin affected cell-viability only at the maximal dose but interestingly it inhibited CXCL8 secretion at all the concentrations not affecting cell-viability. Phenformin had no effect on CXCL8 secretion in thyroid cancer cell lines. Thus, phenformin exerts anti-cancer effects on both cancer cells (cell death induction) and surrounding normal cells (inhibition of CXCL8 secretion). These results highlight that the anti-cancer effects of phenformin are multifaceted and effective on both solid and soluble components of the tumor-microenvironment.

Identifiants

pubmed: 31741708
doi: 10.18632/oncotarget.27266
pii: 27266
pmc: PMC6849649
doi:

Types de publication

Journal Article

Langues

eng

Pagination

6432-6443

Informations de copyright

Copyright: © 2019 Coperchini et al.

Déclaration de conflit d'intérêts

CONFLICTS OF INTEREST The authors have nothing to disclose

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Auteurs

Francesca Coperchini (F)

Istituti Clinici Scientifici Maugeri IRCCS, Unit of Internal Medicine and Endocrinology, Laboratory for Endocrine Disruptors, Pavia, Italy.

Laura Croce (L)

Istituti Clinici Scientifici Maugeri IRCCS, Unit of Internal Medicine and Endocrinology, Laboratory for Endocrine Disruptors, Pavia, Italy.
PHD course in Experimental Medicine, University of Pavia, Pavia, Italy.

Marco Denegri (M)

Molecular Cardiology, ICS-Maugeri, Pavia, Italy.

Oriana Awwad (O)

Department of Biopharmaceutics and Clinical Pharmacy, The University of Jordan, Amman, Jordan.

Samuel Tata Ngnitejeu (ST)

Department of General and Minimally Invasive Surgery, Istituti Clinici Scientifici Maugeri IRCCS, Pavia, Italy.

Flavia Magri (F)

Istituti Clinici Scientifici Maugeri IRCCS, Unit of Internal Medicine and Endocrinology, Laboratory for Endocrine Disruptors, Pavia, Italy.
Department of Internal Medicine and Therapeutics, University of Pavia, Pavia, Italy.

Luca Chiovato (L)

Istituti Clinici Scientifici Maugeri IRCCS, Unit of Internal Medicine and Endocrinology, Laboratory for Endocrine Disruptors, Pavia, Italy.
Department of Internal Medicine and Therapeutics, University of Pavia, Pavia, Italy.

Mario Rotondi (M)

Istituti Clinici Scientifici Maugeri IRCCS, Unit of Internal Medicine and Endocrinology, Laboratory for Endocrine Disruptors, Pavia, Italy.
Department of Internal Medicine and Therapeutics, University of Pavia, Pavia, Italy.

Classifications MeSH