Astaxanthin Inhibits Mitochondrial Permeability Transition Pore Opening in Rat Heart Mitochondria.
astaxantin (AST) administration
cell signaling
mitochondria
non-specific pore
permeability transition
regulatory proteins of mPTP
Journal
Antioxidants (Basel, Switzerland)
ISSN: 2076-3921
Titre abrégé: Antioxidants (Basel)
Pays: Switzerland
ID NLM: 101668981
Informations de publication
Date de publication:
21 Nov 2019
21 Nov 2019
Historique:
received:
22
10
2019
revised:
19
11
2019
accepted:
20
11
2019
entrez:
27
11
2019
pubmed:
27
11
2019
medline:
27
11
2019
Statut:
epublish
Résumé
The mitochondrion is the main organelle of oxidative stress in cells. Increased permeability of the inner mitochondrial membrane is a key phenomenon in cell death. Changes in membrane permeability result from the opening of the mitochondrial permeability transition pore (mPTP), a large-conductance channel that forms after the overload of mitochondria with Ca
Identifiants
pubmed: 31766490
pii: antiox8120576
doi: 10.3390/antiox8120576
pmc: PMC6943429
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : Российский Фонд Фундаментальных Исследований (РФФИ)
ID : 17-04-00747
Déclaration de conflit d'intérêts
The authors declare no conflict of interest.
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