Glutathione Deficiency and Alterations in the Sulfur Amino Acid Homeostasis during Early Postnatal Development as Potential Triggering Factors for Schizophrenia-Like Behavior in Adult Rats.
GSH deficiency
global DNA methylation
neurodevelopmental model of schizophrenia
social and cognitive deficits
sulfur amino acids levels
Journal
Molecules (Basel, Switzerland)
ISSN: 1420-3049
Titre abrégé: Molecules
Pays: Switzerland
ID NLM: 100964009
Informations de publication
Date de publication:
22 Nov 2019
22 Nov 2019
Historique:
received:
10
10
2019
revised:
15
11
2019
accepted:
18
11
2019
entrez:
27
11
2019
pubmed:
27
11
2019
medline:
9
4
2020
Statut:
epublish
Résumé
Impaired glutathione (GSH) synthesis and dopaminergic transmission are important factors in the pathophysiology of schizophrenia. Our research aimed to assess the effects of l-buthionine-(
Identifiants
pubmed: 31766654
pii: molecules24234253
doi: 10.3390/molecules24234253
pmc: PMC6930621
pii:
doi:
Substances chimiques
Amino Acids, Sulfur
0
Piperazines
0
Buthionine Sulfoximine
5072-26-4
vanoxerine
90X28IKH43
Glutathione
GAN16C9B8O
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : National Science Centre of Poland
ID : UMO-2016/23/B/NZ7/01280
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