Risedronate Attenuates Podocyte Injury in Phosphate Transporter-Overexpressing Rats.


Journal

International journal of endocrinology
ISSN: 1687-8337
Titre abrégé: Int J Endocrinol
Pays: Egypt
ID NLM: 101516376

Informations de publication

Date de publication:
2019
Historique:
received: 27 06 2019
accepted: 29 08 2019
entrez: 28 11 2019
pubmed: 28 11 2019
medline: 28 11 2019
Statut: epublish

Résumé

Osteoporosis patients with chronic kidney disease (CKD) are becoming common in our superaging society. Renal dysfunction causes phosphorus accumulation in the circulating plasma and leads to the development of CKD-mineral bone disorder (MBD). We have previously reported that type III Pi transporter-overexpressing transgenic (Pit-1 TG) rats manifest phosphate (Pi)-dependent podocyte injury. In the present study, we explored the effect of risedronate on Pi-induced podocyte injury

Identifiants

pubmed: 31772574
doi: 10.1155/2019/4194853
pmc: PMC6854176
doi:

Types de publication

Journal Article

Langues

eng

Pagination

4194853

Informations de copyright

Copyright © 2019 Yohei Asada et al.

Déclaration de conflit d'intérêts

There are no conflicts of interest regarding the publication of this paper.

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Auteurs

Yohei Asada (Y)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Takeshi Takayanagi (T)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Tsukasa Kawakami (T)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Eisuke Tomatsu (E)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Atsushi Masuda (A)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Yasumasa Yoshino (Y)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Sahoko Sekiguchi-Ueda (S)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Megumi Shibata (M)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Tomihiko Ide (T)

Joint Research Support Promotion Facility, Center for Research Promotion and Support, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Hajime Niimi (H)

Department of Anatomy, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Eishin Yaoita (E)

Department of Structural Pathology, Institute of Nephrology, Niigata University Graduate School of Medical and Dental Sciences, Niigata 950-2102, Japan.

Yusuke Seino (Y)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Yoshihisa Sugimura (Y)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Atsushi Suzuki (A)

Department of Endocrinology and Metabolism, Fujita Health University, Toyoake, Aichi 470-1192, Japan.

Classifications MeSH