Apolipoprotein E Deficiency Causes Endothelial Dysfunction in the Mouse Retina.


Journal

Oxidative medicine and cellular longevity
ISSN: 1942-0994
Titre abrégé: Oxid Med Cell Longev
Pays: United States
ID NLM: 101479826

Informations de publication

Date de publication:
2019
Historique:
received: 10 06 2019
revised: 25 09 2019
accepted: 16 10 2019
entrez: 30 11 2019
pubmed: 30 11 2019
medline: 21 4 2020
Statut: epublish

Résumé

Atherogenic lipoproteins may impair vascular reactivity consecutively causing tissue damage in multiple organs via abnormal perfusion and excessive reactive oxygen species generation. We tested the hypothesis that chronic hypercholesterolemia causes endothelial dysfunction and cell loss in the retina. Twelve-month-old apolipoprotein E-deficient (ApoE-/-) mice and age-matched wild-type controls were used in this study ( Responses to the endothelium-dependent vasodilator, acetylcholine, were markedly impaired in retinal arterioles of ApoE-/- mice ( Apolipoprotein E deficiency induces oxidative stress and endothelial dysfunction in retinal arterioles, which may trigger hypoxia in the retinal tissue. Oxidative stress in nonvascular retinal tissue appears to be prevented by the upregulation of antioxidant redox enzymes, resulting in neuron preservation.

Identifiants

pubmed: 31781340
doi: 10.1155/2019/5181429
pmc: PMC6875001
doi:

Substances chimiques

Apolipoproteins E 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

5181429

Informations de copyright

Copyright © 2019 Jenia Kouchek Zadeh et al.

Déclaration de conflit d'intérêts

The authors state that there is no conflict of interests.

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Auteurs

Jenia Kouchek Zadeh (JK)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Mayagozel B Zhutdieva (MB)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Panagiotis Laspas (P)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Can Yuksel (C)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Aytan Musayeva (A)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Norbert Pfeiffer (N)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Christoph Brochhausen (C)

Institute of Pathology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.
Institute of Pathology, University of Regensburg, Franz-Josef-Strauss-Allee 11, 93053 Regensburg, Germany.

Matthias Oelze (M)

Center of Cardiology 1, Molecular Cardiology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Andreas Daiber (A)

Center of Cardiology 1, Molecular Cardiology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

Ning Xia (N)

Department of Pharmacology, University Medical Center, Johannes Gutenberg University Mainz, Obere Zahlbacher Str. 67, 55131 Mainz, Germany.

Huige Li (H)

Department of Pharmacology, University Medical Center, Johannes Gutenberg University Mainz, Obere Zahlbacher Str. 67, 55131 Mainz, Germany.

Adrian Gericke (A)

Department of Ophthalmology, University Medical Center, Johannes Gutenberg University Mainz, Langenbeckstr. 1, 55131 Mainz, Germany.

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