Zfp281 orchestrates interconversion of pluripotent states by engaging Ehmt1 and Zic2.


Journal

The EMBO journal
ISSN: 1460-2075
Titre abrégé: EMBO J
Pays: England
ID NLM: 8208664

Informations de publication

Date de publication:
15 01 2020
Historique:
received: 03 06 2019
revised: 21 10 2019
accepted: 24 10 2019
pubmed: 30 11 2019
medline: 14 7 2020
entrez: 30 11 2019
Statut: ppublish

Résumé

Developmental cell fate specification is a unidirectional process that can be reverted in response to injury or experimental reprogramming. Whether differentiation and de-differentiation trajectories intersect mechanistically is unclear. Here, we performed comparative screening in lineage-related mouse naïve embryonic stem cells (ESCs) and primed epiblast stem cells (EpiSCs), and identified the constitutively expressed zinc finger transcription factor (TF) Zfp281 as a bidirectional regulator of cell state interconversion. We showed that subtle chromatin binding changes in differentiated cells translate into activation of the histone H3 lysine 9 (H3K9) methyltransferase Ehmt1 and stabilization of the zinc finger TF Zic2 at enhancers and promoters. Genetic gain-of-function and loss-of-function experiments confirmed a critical role of Ehmt1 and Zic2 downstream of Zfp281 both in driving exit from the ESC state and in restricting reprogramming of EpiSCs. Our study reveals that cell type-invariant chromatin association of Zfp281 provides an interaction platform for remodeling the cis-regulatory network underlying cellular plasticity.

Identifiants

pubmed: 31782544
doi: 10.15252/embj.2019102591
pmc: PMC6960450
mid: EMS85790
doi:

Substances chimiques

Chromatin 0
Transcription Factors 0
Zfp281 protein, mouse 0
Zic2 protein, mouse 0
GLP protein, mouse EC 2.1.1.43
Histone-Lysine N-Methyltransferase EC 2.1.1.43

Banques de données

GEO
['GSE131017']

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

e102591

Subventions

Organisme : Medical Research Council
ID : G1100526
Pays : United Kingdom
Organisme : EU Seventh Framework Programme Integrated Project SyBoSS
Pays : International
Organisme : Novartis Research Foundation
Pays : International

Informations de copyright

© 2019 Friedrich Miescher Institute for Biomedical Research.

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Auteurs

Daniela Mayer (D)

Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
Faculty of Sciences, University of Basel, Basel, Switzerland.

Michael B Stadler (MB)

Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
Swiss Institute of Bioinformatics, Basel, Switzerland.

Melanie Rittirsch (M)

Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.

Daniel Hess (D)

Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.

Ilya Lukonin (I)

Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.
Faculty of Sciences, University of Basel, Basel, Switzerland.

Maria Winzi (M)

Medical Systems Biology, UCC, Medical Faculty Carl Gustav Carus, TU Dresden, Dresden, Germany.

Austin Smith (A)

Wellcome-MRC Cambridge Stem Cell Institute and Department of Biochemistry, University of Cambridge, Cambridge, UK.

Frank Buchholz (F)

Medical Systems Biology, UCC, Medical Faculty Carl Gustav Carus, TU Dresden, Dresden, Germany.

Joerg Betschinger (J)

Friedrich Miescher Institute for Biomedical Research, Basel, Switzerland.

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Classifications MeSH