Analysis of the Molecular Signaling Signatures of Muscle Protein Wasting Between the Intercostal Muscles and the Gastrocnemius Muscles in
Animals
Diabetes Complications
/ genetics
Diabetes Mellitus, Experimental
/ genetics
Diabetes Mellitus, Type 2
/ genetics
Male
Mechanistic Target of Rapamycin Complex 1
/ genetics
Mice
Mice, Transgenic
Muscle Proteins
/ genetics
Muscle, Skeletal
/ metabolism
Muscular Atrophy
/ genetics
Signal Transduction
Akt
Atrogin-1
FoxO
MuRF1
autophagic flux
gastrocnemius muscle
intercostal muscles
mTOR
muscle atrophy
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
01 Dec 2019
01 Dec 2019
Historique:
received:
18
10
2019
revised:
22
11
2019
accepted:
28
11
2019
entrez:
7
12
2019
pubmed:
7
12
2019
medline:
9
4
2020
Statut:
epublish
Résumé
Type 2 diabetes (T2D) patients suffer from dyspnea, which contributes to disease-related morbidity. Although T2D has been reported to induce a catabolic state in skeletal muscle, whether T2D induces muscle wasting in respiratory muscles has not yet been investigated. In this study, we examine the difference in the molecular signaling signature of muscle wasting between the intercostal and gastrocnemius muscles using
Identifiants
pubmed: 31805681
pii: ijms20236062
doi: 10.3390/ijms20236062
pmc: PMC6929000
pii:
doi:
Substances chimiques
Muscle Proteins
0
Mechanistic Target of Rapamycin Complex 1
EC 2.7.11.1
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Ministry of Science and ICT
ID : 2018R1A2B6004513
Organisme : the ministry of education
ID : 2017R1C1B5016952
Organisme : Korea Health Industry Development Institute
ID : HI17C0426
Pays : Republic of Korea
Organisme : Gil Medical Center, Gachon University
ID : FRD2018-5189
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