Reverse transcriptase multiplex ligation-dependent probe amplification in endomyocardial biopsies for the diagnosis of cardiac allograft rejection.


Journal

The Journal of heart and lung transplantation : the official publication of the International Society for Heart Transplantation
ISSN: 1557-3117
Titre abrégé: J Heart Lung Transplant
Pays: United States
ID NLM: 9102703

Informations de publication

Date de publication:
02 2020
Historique:
received: 13 04 2019
revised: 17 11 2019
accepted: 20 11 2019
pubmed: 12 12 2019
medline: 1 4 2021
entrez: 12 12 2019
Statut: ppublish

Résumé

Molecular biology has emerged as a potential companion to histology for the diagnosis of rejection after heart transplantation. Reverse transcriptase multiplex ligation-dependent probe amplification (RT-MLPA) is a technique of targeted gene expression analysis suitable for formalin-fixed paraffin-embedded (FFPE) biopsies. Our aim was to assess RT-MLPA for the diagnosis of allograft rejection in heart transplantation. We performed a cross-sectional, case-control, multicenter study. After the selection of a 14-transcript panel (endothelial burden, Natural killer cells, interferon-γ pathway, effector T-cells, and antigen presentation), RT-MLPA was applied to 183 FFPE endomyocardial biopsies (EMB), randomized into a training (n = 113) and a validation (n = 70) series. A two-step class prediction analysis was developed (Linear prediction score-LPS1: rejection vs non-rejection; LPS2: antibody-mediated rejection [AMR] vs acute cellular rejection [ACR]). A study of the agreement between pathology and RT-MLPA was performed. Overall, 48 ACR, 82 AMR, 5 mixed rejection, and 48 non-rejection EMBs were analyzed. Three molecular clusters were delineated by unsupervised hierarchical analysis (molecular non-rejection, ACR, and AMR). AMR was characterized by the high expression of CCL4, GNLY, FCGR3, CXCL11 and ACR by the high expression of CCL18 and ADAMdec. RT-MLPA and histopathology agreed in the final diagnosis in 82.2%, 67.7%, and 76.8% of the EMB in the test, validation, and overall cohort, respectively. Disagreement cases were more common in the case of histologic low-grade rejection and early post-transplant EMB. RT-MLPA is a suitable technique for targeted gene expression analysis on FFPE EMB with a good overall agreement with the histologic diagnosis of heart allograft rejection.

Sections du résumé

BACKGROUND
Molecular biology has emerged as a potential companion to histology for the diagnosis of rejection after heart transplantation. Reverse transcriptase multiplex ligation-dependent probe amplification (RT-MLPA) is a technique of targeted gene expression analysis suitable for formalin-fixed paraffin-embedded (FFPE) biopsies. Our aim was to assess RT-MLPA for the diagnosis of allograft rejection in heart transplantation.
METHODS
We performed a cross-sectional, case-control, multicenter study. After the selection of a 14-transcript panel (endothelial burden, Natural killer cells, interferon-γ pathway, effector T-cells, and antigen presentation), RT-MLPA was applied to 183 FFPE endomyocardial biopsies (EMB), randomized into a training (n = 113) and a validation (n = 70) series. A two-step class prediction analysis was developed (Linear prediction score-LPS1: rejection vs non-rejection; LPS2: antibody-mediated rejection [AMR] vs acute cellular rejection [ACR]). A study of the agreement between pathology and RT-MLPA was performed.
RESULTS
Overall, 48 ACR, 82 AMR, 5 mixed rejection, and 48 non-rejection EMBs were analyzed. Three molecular clusters were delineated by unsupervised hierarchical analysis (molecular non-rejection, ACR, and AMR). AMR was characterized by the high expression of CCL4, GNLY, FCGR3, CXCL11 and ACR by the high expression of CCL18 and ADAMdec. RT-MLPA and histopathology agreed in the final diagnosis in 82.2%, 67.7%, and 76.8% of the EMB in the test, validation, and overall cohort, respectively. Disagreement cases were more common in the case of histologic low-grade rejection and early post-transplant EMB.
CONCLUSIONS
RT-MLPA is a suitable technique for targeted gene expression analysis on FFPE EMB with a good overall agreement with the histologic diagnosis of heart allograft rejection.

Identifiants

pubmed: 31822443
pii: S1053-2498(19)31762-0
doi: 10.1016/j.healun.2019.11.010
pii:
doi:

Substances chimiques

RNA 63231-63-0

Types de publication

Journal Article Multicenter Study Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

115-124

Informations de copyright

Copyright © 2019. Published by Elsevier Inc.

Auteurs

Nicolas Adam (N)

Paris Translational Research Centre for Organ Transplantation, INSERM, UMR-S970, Paris, France; Pathology department, Hôpital Necker, Assistance Publique - Hôpitaux de Paris and Paris Descartes University, Paris, France.

Guillaume Coutance (G)

Paris Translational Research Centre for Organ Transplantation, INSERM, UMR-S970, Paris, France; Department of cardio-thoracic surgery, La Pitié-Salpêtrière, Assistance Publique des hôpitaux de Paris, Paris, France.

Pierre-Julien Viailly (PJ)

INSERM U1245, Centre Henri Becquerel, Rouen, France.

Fanny Drieux (F)

INSERM U1245, Centre Henri Becquerel, Rouen, France.

Philippe Ruminy (P)

Pathology department, La Pitié Salpétrière, Assistance Publique - Hôpitaux de Paris, Paris, France.

Ahmad Abdel Sater (AA)

INSERM U1245, Centre Henri Becquerel, Rouen, France.

Claire Toquet (C)

Pathology department, Nantes University, Nantes, France.

Philippe Rouvier (P)

Department of cardio-thoracic surgery, La Pitié-Salpêtrière, Assistance Publique des hôpitaux de Paris, Paris, France.

Arnaud François (A)

Pathology department, Rouen, France.

Marie-Pierre Chenard (MP)

Pathology department, Strasbourg, France.

Eric Epailly (E)

Thoracic Surgery departments, Strasbourg, France.

Romain Guillemain (R)

Cardiovascular surgery and heart transplant department Georges Pompidou Hospital, Assistance Publique - Hôpitaux de Paris, Paris, France.

Sabine Pattier (S)

Cardiovascular surgery and heart transplant department, Nantes, France.

Arnaud Gay (A)

Cardiovascular surgery and heart transplant department, Rouen, France.

Shaida Varnous (S)

Department of cardio-thoracic surgery, La Pitié-Salpêtrière, Assistance Publique des hôpitaux de Paris, Paris, France.

Jean-Luc Taupin (JL)

Histocompatibility laboratory, Saint Louis hospital, Paris, France.

Marion Rabant (M)

Paris Translational Research Centre for Organ Transplantation, INSERM, UMR-S970, Paris, France; Pathology department, Hôpital Necker, Assistance Publique - Hôpitaux de Paris and Paris Descartes University, Paris, France.

Alexandre Loupy (A)

Paris Translational Research Centre for Organ Transplantation, INSERM, UMR-S970, Paris, France.

Patrick Bruneval (P)

Paris Translational Research Centre for Organ Transplantation, INSERM, UMR-S970, Paris, France; Pathology, cardiology and heart transplant department Georges Pompidou Hospital, Assistance Publique - Hôpitaux de Paris, Paris, France.

Jean Paul Duong Van Huyen (JP)

Paris Translational Research Centre for Organ Transplantation, INSERM, UMR-S970, Paris, France; Pathology department, Hôpital Necker, Assistance Publique - Hôpitaux de Paris and Paris Descartes University, Paris, France; Pathology department, Nantes University, Nantes, France. Electronic address: jean-paul.duong@aphp.fr.

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