Modulation of Glycine Receptor-Mediated Pain Signaling
glucose modulation
glycinergic transmission
hotplate assay
nociception
pain-related behavior
patch-clamp recording
spinal α3 glycine receptors
tactile sensitivity
Journal
Frontiers in molecular neuroscience
ISSN: 1662-5099
Titre abrégé: Front Mol Neurosci
Pays: Switzerland
ID NLM: 101477914
Informations de publication
Date de publication:
2019
2019
Historique:
received:
24
07
2019
accepted:
01
11
2019
entrez:
12
12
2019
pubmed:
12
12
2019
medline:
12
12
2019
Statut:
epublish
Résumé
The inhibitory glycine receptor (GlyR) plays an important role in rapid synaptic inhibition in mammalian spinal cord, brainstem, higher brain centers, and is involved in transmission of nociceptive signals. Glucose and related mono- and disaccharides potentiate currents mediated by recombinant α1, α1-β, and α3 GlyRs. Here, we confirmed the specific potentiation of α3 GlyR signaling by glucose through: (i) patch-clamp electrophysiology on recombinant receptors; and (ii) by verifying
Identifiants
pubmed: 31824259
doi: 10.3389/fnmol.2019.00280
pmc: PMC6883931
doi:
Types de publication
Journal Article
Langues
eng
Pagination
280Informations de copyright
Copyright © 2019 Hussein, Ahmed, Breitinger and Breitinger.
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